Ralf P. Brandes mainly investigates Endocrinology, Internal medicine, NADPH oxidase, Biochemistry and Endothelium. His Internal medicine research incorporates elements of Progenitor cell and Endothelium-derived hyperpolarizing factor. His work carried out in the field of NADPH oxidase brings together such families of science as Oxidase test and Superoxide.
The Superoxide study combines topics in areas such as Xanthine oxidase and Renovascular hypertension. His work deals with themes such as Endothelial stem cell, Nitric oxide synthase, Pathology, Endothelial dysfunction and Umbilical vein, which intersect with Endothelium. His NOX4 research is multidisciplinary, relying on both NOX1 and NADPH Oxidase 1.
His primary scientific interests are in Internal medicine, Endocrinology, Cell biology, NADPH oxidase and Biochemistry. His Internal medicine study typically links adjacent topics like Cardiology. Many of his studies on Endocrinology apply to Inflammation as well.
His biological study spans a wide range of topics, including Endothelial stem cell, Angiogenesis and Vascular smooth muscle. His NADPH oxidase study integrates concerns from other disciplines, such as Superoxide and Pharmacology. His Superoxide study combines topics from a wide range of disciplines, such as Oxidase test, Xanthine oxidase and Superoxide dismutase.
Ralf P. Brandes focuses on Cell biology, NADPH oxidase, NOX4, Internal medicine and Biochemistry. His Cell biology research is multidisciplinary, incorporating elements of Endothelial stem cell, Transcription factor and Long non-coding RNA. His NADPH oxidase research entails a greater understanding of Reactive oxygen species.
He interconnects Peripheral nerve injury, Knockout mouse, Nerve injury and Neuronal action potential in the investigation of issues within NOX4. The study incorporates disciplines such as Endocrinology and Cardiology in addition to Internal medicine. In the field of Endocrinology, his study on Renal fibrosis and Pathophysiology of hypertension overlaps with subjects such as Geldanamycin and hERG.
Cell biology, NADPH oxidase, NOX4, Biochemistry and NOX1 are his primary areas of study. His Cell biology research includes themes of Transcription factor and TRIF. Ralf P. Brandes has included themes like Hypoxia, Pulmonary hypertension, Molecular biology, Insulin resistance and Pharmacology in his NADPH oxidase study.
His NOX4 research is multidisciplinary, relying on both Beta oxidation, Knockout mouse, Insulin and Mitochondrion. His research in the fields of P22phox, Proximity ligation assay, Chaperone and Protein–protein interaction overlaps with other disciplines such as Calnexin. Reactive oxygen species covers Ralf P. Brandes research in NOX1.
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Transdifferentiation of Blood-Derived Human Adult Endothelial Progenitor Cells Into Functionally Active Cardiomyocytes
Cornel Badorff;Ralf P. Brandes;Rüdiger Popp;Stefan Rupp.
Apocynin Is Not an Inhibitor of Vascular NADPH Oxidases but an Antioxidant
Sabine Heumüller;Sven Wind;Eduardo Barbosa-Sicard;Harald H.H.W. Schmidt.
A gp91phox Containing NADPH Oxidase Selectively Expressed in Endothelial Cells Is a Major Source of Oxygen Radical Generation in the Arterial Wall
A. Görlach;R. P. Brandes;K. Nguyen;M. Amidi.
Circulation Research (2000)
Nox family NADPH oxidases: Molecular mechanisms of activation
Ralf P. Brandes;Norbert Weissmann;Katrin Schröder.
Free Radical Biology and Medicine (2014)
Nox4 Is a Protective Reactive Oxygen Species Generating Vascular NADPH Oxidase
Katrin Schröder;Min Zhang;Sebastian Benkhoff;Anja Mieth.
Circulation Research (2012)
Direct Interaction of the Novel Nox Proteins with p22phox Is Required for the Formation of a Functionally Active NADPH Oxidase
Rashmi K. Ambasta;Pravir Kumar;Kathy K. Griendling;Harald H.H.W. Schmidt.
Journal of Biological Chemistry (2004)
Endothelium-Derived Hyperpolarizing Factor Synthase (Cytochrome P450 2C9) Is a Functionally Significant Source of Reactive Oxygen Species in Coronary Arteries
Ingrid Fleming;U. Ruth Michaelis;Daniel Bredenkötter;Beate Fisslthaler.
Circulation Research (2001)
Thrombin activates the hypoxia-inducible factor-1 signaling pathway in vascular smooth muscle cells: Role of the p22(phox)-containing NADPH oxidase.
A. Görlach;I. Diebold;V. B. Schini-Kerth;U. Berchner-Pfannschmidt.
Circulation Research (2001)
Vascular NADPH oxidases: molecular mechanisms of activation
Ralf P. Brandes;Jörg Kreuzer.
Cardiovascular Research (2005)
NADPH oxidase-4 mediates protection against chronic load-induced stress in mouse hearts by enhancing angiogenesis
Min Zhang;Alison Brewer;Katrin Schröder;Celio Santos Xavier Da Costa Dos Santos.
Proceedings of the National Academy of Sciences of the United States of America (2010)
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