What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Endocrinology
- Gene
His scientific interests lie mostly in Internal medicine, Endocrinology, Endothelium, Vascular smooth muscle and Nitric oxide.
His studies in Internal medicine integrate themes in fields like Endothelial stem cell, Cyclooxygenase and Anatomy.
He usually deals with Endocrinology and limits it to topics linked to Thromboxane A2 and Muscle relaxation.
His Endothelium research is multidisciplinary, incorporating perspectives in Hyperpolarization, Acetylcholine, Bradykinin and Vasodilation.
In Bradykinin, he works on issues like Coronary arteries, which are connected to Platelet.
Paul M. Vanhoutte focuses mostly in the field of Vascular smooth muscle, narrowing it down to matters related to Vasoconstriction and, in some cases, Endothelins.
His most cited work include:
- Endothelium-derived relaxing and contracting factors. (1775 citations)
- Flow-induced release of endothelium-derived relaxing factor (1153 citations)
- Superoxide anions and hyperoxia inactivate endothelium-derived relaxing factor. (1082 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Internal medicine, Endocrinology, Endothelium, Vascular smooth muscle and Nitric oxide.
His research links Cardiology with Internal medicine.
His research combines Bradykinin and Endocrinology.
The concepts of his Endothelium study are interwoven with issues in Vasoconstriction, Anesthesia, Anatomy, Coronary arteries and Pharmacology.
His work in Vascular smooth muscle covers topics such as Hyperpolarization which are related to areas like Potassium channel.
His Nitric oxide research integrates issues from Biochemistry and Cell biology.
He most often published in these fields:
- Internal medicine (73.35%)
- Endocrinology (65.35%)
- Endothelium (42.35%)
What were the highlights of his more recent work (between 2012-2021)?
- Internal medicine (73.35%)
- Endocrinology (65.35%)
- Endothelium (42.35%)
In recent papers he was focusing on the following fields of study:
Paul M. Vanhoutte focuses on Internal medicine, Endocrinology, Endothelium, Nitric oxide and Endothelial dysfunction.
All of his Internal medicine and Vascular smooth muscle, Vasodilation, Mesenteric arteries, Oxidative stress and Endothelin 1 investigations are sub-components of the entire Internal medicine study.
His study looks at the relationship between Endocrinology and fields such as Receptor, as well as how they intersect with chemical problems.
His work carried out in the field of Endothelium brings together such families of science as Hypoxia, Biochemistry, Apocynin, Phenylephrine and Anatomy.
His Nitric oxide research includes themes of Protein kinase B and Pharmacology.
His study in Endothelial dysfunction is interdisciplinary in nature, drawing from both Adenosine, Protein kinase A, Renin–angiotensin system, Immunology and Electrical impedance myography.
Between 2012 and 2021, his most popular works were:
- Endothelial dysfunction and vascular disease – a 30th anniversary update (328 citations)
- Vascular nitric oxide: Beyond eNOS (312 citations)
- Akkermansia Muciniphila Protects Against Atherosclerosis by Preventing Metabolic Endotoxemia-Induced Inflammation in Apoe−/− Mice (244 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Enzyme
His primary scientific interests are in Internal medicine, Endocrinology, Nitric oxide, Endothelium and Endothelial dysfunction.
He regularly ties together related areas like Receptor in his Endocrinology studies.
Paul M. Vanhoutte has researched Nitric oxide in several fields, including Cardiac function curve, Protein kinase B and Knockout mouse.
His Endothelium research is multidisciplinary, incorporating elements of Blood vessel remodeling, Neuroscience and Gene knockdown.
His Endothelial dysfunction research integrates issues from AMPK, Protein kinase A, Stent, Prostaglandin and microRNA.
His Vasodilation study which covers Endothelial stem cell that intersects with Autacoid.
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