Zvonimir S. Katusic mostly deals with Internal medicine, Endocrinology, Endothelium, Nitric oxide synthase and Nitric oxide. His work in Internal medicine addresses subjects such as Endothelial stem cell, which are connected to disciplines such as Transplantation. His Endocrinology study typically links adjacent topics like Sickle cell anemia.
His research in Endothelium intersects with topics in Blood vessel, Oxidative stress, Immunology, Circulatory system and Superoxide. His Superoxide research includes elements of Cyclooxygenase, Endothelin receptor, Superoxide dismutase and Paracrine signalling. He has included themes like Cell biology and Endothelial dysfunction in his Nitric oxide study.
His primary areas of investigation include Internal medicine, Endocrinology, Endothelium, Nitric oxide and Nitric oxide synthase. Internal medicine is closely attributed to Superoxide in his research. His study in Endocrinology is interdisciplinary in nature, drawing from both Basilar artery, Endothelial stem cell and Bradykinin.
His Endothelium study integrates concerns from other disciplines, such as Blood vessel, Anatomy, Amyloid precursor protein, Cell biology and Pharmacology. His Nitric oxide research is multidisciplinary, incorporating perspectives in Biochemistry and Calcium. His biological study spans a wide range of topics, including Molecular biology, Genetic transfer and In vivo.
His primary areas of study are Internal medicine, Endocrinology, Amyloid precursor protein, Endothelium and Nitric oxide. The various areas that he examines in his Internal medicine study include Bioavailability and Cortex. His Endocrinology study combines topics from a wide range of disciplines, such as Hyperoxia and Superoxide.
His research integrates issues of Deficient mouse, Immunology, Endothelial dysfunction and Cell biology in his study of Amyloid precursor protein. His Endothelium research is multidisciplinary, incorporating elements of Angiotensin II and Vasodilation. His work is connected to Nitric Oxide Synthase Type III and Nitric oxide synthase, as a part of Nitric oxide.
His primary scientific interests are in Internal medicine, Endocrinology, Cell biology, Enos and Amyloid precursor protein. His Internal medicine research incorporates elements of Genetic therapy and Cardiology. His Endocrinology study focuses mostly on Tetrahydrobiopterin, Nitric oxide and Endothelial dysfunction.
His Nitric oxide research focuses on Nitric oxide synthase in particular. His Enos course of study focuses on Superoxide and Superoxide dismutase. His Amyloid precursor protein study incorporates themes from Endothelium, Immunology and Cognitive decline.
High Glucose Increases Nitric Oxide Synthase Expression and Superoxide Anion Generation in Human Aortic Endothelial Cells
Francesco Cosentino;Keiichi Hishikawa;Zvonimir S. Katusic;Thomas F. Lüscher.
Oxidation of Tetrahydrobiopterin by Peroxynitrite: Implications for Vascular Endothelial Function
Sheldon Milstien;Zvonimir Katusic.
Biochemical and Biophysical Research Communications (1999)
Superoxide anion is an endothelium-derived contracting factor
Z. S. Katusic;P. M. Vanhoutte.
American Journal of Physiology-heart and Circulatory Physiology (1989)
Tetrahydrobiopterin and Dysfunction of Endothelial Nitric Oxide Synthase in Coronary Arteries
Francesco Cosentino;Zvonimir S. Katusic.
Vascular endothelial dysfunction: does tetrahydrobiopterin play a role?
Zvonimir S. Katusic.
American Journal of Physiology-heart and Circulatory Physiology (2001)
Vascular dysfunction-The disregarded partner of Alzheimer's disease
Melanie D. Sweeney;Axel Montagne;Abhay P. Sagare;Daniel A. Nation.
Alzheimers & Dementia (2019)
Long-Term Vitamin C Treatment Increases Vascular Tetrahydrobiopterin Levels and Nitric Oxide Synthase Activity
Livius V. d’Uscio;Sheldon Milstien;Darcy Richardson;Leslie Smith.
Circulation Research (2003)
Vasopressin causes endothelium-dependent relaxations of the canine basilar artery.
Z S Katusic;J T Shepherd;P M Vanhoutte.
Circulation Research (1984)
Human Endothelial Progenitor Cells Tolerate Oxidative Stress Due to Intrinsically High Expression of Manganese Superoxide Dismutase
Tongrong He;Timothy E. Peterson;Ekhson L. Holmuhamedov;Andre Terzic.
Arteriosclerosis, Thrombosis, and Vascular Biology (2004)
Thromboxane A2 receptor antagonists inhibit endothelium-dependent contractions.
Wolfgang Auch-Schwelk;Zvonimir S. Katusic;Paul M. Vanhoutte.
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