His scientific interests lie mostly in Internal medicine, Pulmonary hypertension, Sickle cell anemia, Hemolysis and Hemoglobin. His research in Internal medicine intersects with topics in Endocrinology and Cardiology. His study in Endocrinology is interdisciplinary in nature, drawing from both Pathology, Intercellular Adhesion Molecule-1, Cell adhesion molecule and Lactate dehydrogenase.
Gregory J. Kato combines subjects such as Surgery, Pulmonary artery, Anemia, Hemolytic anemia and Risk factor with his study of Sickle cell anemia. His Hemolysis research is within the category of Immunology. His Hemoglobin research incorporates themes from Nitric oxide and Pharmacology.
The scientist’s investigation covers issues in Internal medicine, Sickle cell anemia, Pulmonary hypertension, Immunology and Cell. His Internal medicine research incorporates elements of Gastroenterology, Endocrinology, Surgery and Cardiology. His Endocrinology study combines topics in areas such as Arginine and Lactate dehydrogenase.
The concepts of his Sickle cell anemia study are interwoven with issues in Hemolytic anemia, Thalassemia and Risk factor. Within one scientific family, Gregory J. Kato focuses on topics pertaining to Nitric oxide under Pulmonary hypertension, and may sometimes address concerns connected to Pharmacology. His study looks at the intersection of Hemolysis and topics like Hemoglobin with Haptoglobin and In vivo.
His primary areas of study are Internal medicine, Cell, Disease, Sickle cell anemia and Anemia. His study in Pulmonary hypertension and Hemoglobin is carried out as part of his Internal medicine studies. His work in Hemoglobin tackles topics such as Hemolysis which are related to areas like Methemoglobin, Lung injury and Nitric oxide.
Gregory J. Kato has included themes like Cancer research, Hypoxia, Pathology, Ischemia and Cell biology in his Cell study. His studies in Sickle cell anemia integrate themes in fields like Thalassemia, Inflammation, Blood transfusion, Immunology and Creatinine. His Anemia research integrates issues from Hydroxycarbamide and MEDLINE.
Gregory J. Kato mainly investigates Cell, Disease, Internal medicine, Immunology and Acute chest syndrome. His research integrates issues of Sleep in non-human animals and Cardiology in his study of Internal medicine. His work on Pulmonary hypertension and Sudden death as part of general Cardiology research is often related to Absolute neutrophil count, thus linking different fields of science.
His Immunology research is multidisciplinary, relying on both Hemostasis, Thrombosis, Sickle cell anemia and Anemia. His work deals with themes such as Hemoglobin, Thrombospondin 1 and Pulmonary artery, which intersect with Sickle cell anemia. As a part of the same scientific family, Gregory J. Kato mostly works in the field of Acute chest syndrome, focusing on Platelet and, on occasion, Intravital microscopy, Lung and Vaso-occlusive crisis.
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Deconstructing sickle cell disease: Reappraisal of the role of hemolysis in the development of clinical subphenotypes
Gregory J. Kato;Mark T. Gladwin;Martin H. Steinberg.
Blood Reviews (2007)
Dysregulated arginine metabolism, hemolysis-associated pulmonary hypertension, and mortality in sickle cell disease.
Claudia R. Morris;Gregory J. Kato;Mirjana Poljakovic;Xunde Wang.
Lactate dehydrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease
Gregory J Kato;Gregory J Kato;Gregory J Kato;Vicki R McGowan;Vicki R McGowan;Vicki R McGowan;Roberto F Machado;Roberto F Machado;Roberto F Machado;Jane A Little;Jane A Little;Jane A Little.
Sickle cell disease
Gregory J Kato;Frédéric B Piel;Clarice D Reid;Marilyn H Gaston.
Nature Reviews Disease Primers (2018)
An amino-terminal c-myc domain required for neoplastic transformation activates transcription.
G J Kato;J Barrett;M Villa-Garcia;C V Dang.
Molecular and Cellular Biology (1990)
Intravascular hemolysis and the pathophysiology of sickle cell disease
Gregory J. Kato;Martin H. Steinberg;Mark T. Gladwin.
Journal of Clinical Investigation (2017)
Platelet activation in patients with sickle disease, hemolysis-associated pulmonary hypertension, and nitric oxide scavenging by cell-free hemoglobin
José Villagra;Sruti Shiva;Lori A. Hunter;Roberto F. Machado.
Vasculopathy in sickle cell disease: Biology, pathophysiology, genetics, translational medicine, and new research directions.
Gregory J. Kato;Robert P. Hebbel;Martin H. Steinberg;Mark T. Gladwin.
American Journal of Hematology (2009)
Diastolic dysfunction is an independent risk factor for death in patients with sickle cell disease.
Vandana Sachdev;Roberto F. Machado;Yukitaka Shizukuda;Yesoda N. Rao.
Journal of the American College of Cardiology (2007)
Sildenafil therapy in patients with sickle cell disease and pulmonary hypertension
Roberto F. Machado;Sabrina Martyr;Gregory J. Kato;Robyn J. Barst.
British Journal of Haematology (2005)
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