Edward V. Prochownik mostly deals with Molecular biology, Cancer research, Carcinogenesis, Cell biology and Gene. His study in Molecular biology is interdisciplinary in nature, drawing from both Cellular differentiation, Cell cycle, Oncogene, Complementary DNA and Regulation of gene expression. Edward V. Prochownik integrates many fields in his works, including Cancer research and MYC Gene Amplification.
His Carcinogenesis research integrates issues from Aneuploidy, Cell growth, Phenotype, Genome instability and Gene duplication. His Cell biology research incorporates themes from Transformation and Myogenic regulatory factors. He has included themes like Clinical investigation, Primary tumor, Pathogenesis and Neoplastic disease in his Gene study.
Edward V. Prochownik mostly deals with Cancer research, Molecular biology, Cell biology, Transcription factor and Cell culture. His Cancer research research also works with subjects such as
The various areas that Edward V. Prochownik examines in his Gene study include DNA and DNA damage. His studies deal with areas such as Phenotype, Transformation, Genome instability and Cancer cell as well as Cell biology. His Transcription factor research incorporates elements of Gene expression and Transcription.
His primary areas of study are Cancer research, Carcinogenesis, Biochemistry, Cancer and Glycolysis. His research integrates issues of Hepatoblastoma, Transcription factor, Mutant, Downregulation and upregulation and Cell cycle in his study of Cancer research. His Transcription factor research includes themes of Liver disease and Liver function.
His biological study spans a wide range of topics, including Mutation and Inflammation. His Glycolysis study incorporates themes from Oxidative phosphorylation and Citric acid cycle. His Surgical oncology study combines topics in areas such as Computational biology and Gene.
Edward V. Prochownik focuses on Cancer research, Ribosomopathy, Ribosomal protein, Cell biology and Carcinogenesis. In his work, he performs multidisciplinary research in Cancer research and Inflammatory bowel disease. His Ribosomopathy study integrates concerns from other disciplines, such as Translation, Surgical oncology, Organelle and Mutation.
Edward V. Prochownik combines subjects such as Phenotype, Point mutation, Gene mutation, Mdm2 and Messenger RNA with his study of Ribosomal protein. His Cell biology research is multidisciplinary, incorporating elements of Cancer cell, Warburg effect, Anabolism, Metabolic pathway and Tumor progression. Edward V. Prochownik has researched Carcinogenesis in several fields, including Immunology, STAT3, Signal transduction, Gene silencing and Colorectal cancer.
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MYC oncogenes and human neoplastic disease
Chadd E Nesbit;Jean M Tersak;Edward V Prochownik;Edward V Prochownik.
Oncogene (1999)
Myc-induced T cell leukemia in transgenic zebrafish.
David M. Langenau;David Traver;Adolfo A. Ferrando;Jeffery L. Kutok.
Science (2003)
Tumor necrosis factor-alpha induction of novel gene products in human endothelial cells including a macrophage-specific chemotaxin.
V M Dixit;S Green;V Sarma;L B Holzman.
Journal of Biological Chemistry (1990)
Low molecular weight inhibitors of Myc-Max interaction and function.
Xiaoying Yin;Christine Giap;John S Lazo;Edward V Prochownik;Edward V Prochownik.
Oncogene (2003)
Constitutive expression of a c-myb cDNA blocks Friend murine erythroleukemia cell differentiation.
M F Clarke;J F Kukowska-Latallo;E Westin;M Smith.
Molecular and Cellular Biology (1988)
Deregulated expression of c-myc by murine erythroleukaemia cells prevents differentiation.
E. V. Prochownik;J. Kukowska.
Nature (1986)
c-myc and bcl-2 modulate p53 function by altering p53 subcellular trafficking during the cell cycle
James J. Ryan;Edward Prochownik;Christopher A. Gottlieb;Ingrid J. Apel.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Differential Interactions of Id Proteins with Basic-Helix-Loop-Helix Transcription Factors
Kenneth Langlands;Xiaoying Yin;Geetha Anand;Edward V. Prochownik;Edward V. Prochownik.
Journal of Biological Chemistry (1997)
Mutation of the MXI1 gene in prostate cancer
Linette R. Eagle;Xiaoying Yin;Arthur R. Brothman;Briana J. Williams.
Nature Genetics (1995)
c-myc antisense transcripts accelerate differentiation and inhibit G1 progression in murine erythroleukemia cells.
E V Prochownik;J Kukowska;C Rodgers.
Molecular and Cellular Biology (1988)
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