D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 70 Citations 21,407 130 World Ranking 1494 National Ranking 104

Research.com Recognitions

Awards & Achievements

1998 - Member of Academia Europaea

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

Martin Eilers mainly investigates Cancer research, Molecular biology, Transcription factor, Cyclin A and Cyclin D. His Cancer research research incorporates elements of Carcinogenesis, Oncogene, Neuroblastoma, N-Myc and Ubiquitin ligase. He has included themes like Regulation of gene expression and Transactivation in his Molecular biology study.

His study in Transcription factor is interdisciplinary in nature, drawing from both Promoter, Transcription and Cell growth. His studies examine the connections between Cyclin A and genetics, as well as such issues in Cyclin A2, with regards to Cyclin E. His Gene research includes themes of Cell culture, Binding site and Cell biology.

His most cited work include:

  • The beta-catenin/TCF-4 complex imposes a crypt progenitor phenotype on colorectal cancer cells. (1819 citations)
  • Transcriptional regulation and transformation by Myc proteins (919 citations)
  • Myc’s broad reach (741 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Cancer research, Cell biology, Transcription factor, Molecular biology and Gene. His Cancer research research integrates issues from Gene expression, Proto-Oncogene Proteins c-myc, Carcinogenesis, Oncogene and Neuroblastoma. His Cell biology research focuses on Cell growth and how it relates to Cellular differentiation.

Martin Eilers has researched Transcription factor in several fields, including Psychological repression, Transcription, Apoptosis and Regulation of gene expression. His Molecular biology study incorporates themes from Cyclin A2, Transactivation, Promoter, Cyclin A and Cyclin D. His Gene research is under the purview of Genetics.

He most often published in these fields:

  • Cancer research (44.54%)
  • Cell biology (34.93%)
  • Transcription factor (25.33%)

What were the highlights of his more recent work (between 2013-2021)?

  • Cancer research (44.54%)
  • Cell biology (34.93%)
  • Transcription factor (25.33%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Cancer research, Cell biology, Transcription factor, Transcription and Gene. His research in Cancer research intersects with topics in Cancer, Psychological repression, Gene expression, Apoptosis and In vivo. Martin Eilers combines subjects such as RNA polymerase II, Protein subunit and Ubiquitin, Ubiquitin ligase with his study of Cell biology.

His studies deal with areas such as Cell growth and Transactivation as well as Ubiquitin. His studies in Transcription factor integrate themes in fields like Carcinogenesis, Medulloblastoma, Phenotype, Endogeny and Neuroblastoma. His research in N-Myc tackles topics such as Molecular biology which are related to areas like Cellular differentiation.

Between 2013 and 2021, his most popular works were:

  • MYC regulates the antitumor immune response through CD47 and PD-L1 (516 citations)
  • Activation and repression by oncogenic MYC shape tumour-specific gene expression profiles (286 citations)
  • Genomic analysis identifies new drivers and progression pathways in skin basal cell carcinoma (202 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

Martin Eilers mainly focuses on Cancer research, Transcription factor, Carcinogenesis, Cell biology and Regulation of gene expression. Martin Eilers is interested in Liver cancer, which is a branch of Cancer research. His work carried out in the field of Transcription factor brings together such families of science as Phenotype and Oncogene.

His Cell biology research is multidisciplinary, incorporating perspectives in Protein subunit and Ubiquitin, Ubiquitin ligase. The concepts of his Ubiquitin study are interwoven with issues in Molecular biology and Transactivation. His Regulation of gene expression research is multidisciplinary, relying on both Cell culture, Transcriptome, Gene expression, Medulloblastoma and Chromatin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The beta-catenin/TCF-4 complex imposes a crypt progenitor phenotype on colorectal cancer cells.

Marc van de Wetering;Elena Sancho;Cornelis Verweij;Wim de Lau.
Cell (2002)

2321 Citations

Transcriptional regulation and transformation by Myc proteins

Sovana Adhikary;Martin Eilers.
Nature Reviews Molecular Cell Biology (2005)

1283 Citations

Myc’s broad reach

Martin Eilers;Robert N. Eisenman.
Genes & Development (2008)

997 Citations

Binding of a specific ligand inhibits import of a purified precursor protein into mitochondria

Martin Eilers;Gottfried Schatz.
Nature (1986)

764 Citations

The MYC protein activates transcription of the alpha‐prothymosin gene.

M. Eilers;S. Schirm;J. M. Bishop.
The EMBO Journal (1991)

711 Citations

Repression of p15INK4b expression by Myc through association with Miz-1.

Peter Staller;Karen Peukert;Astrid Kiermaier;Joan Seoane.
Nature Cell Biology (2001)

677 Citations

MYC regulates the antitumor immune response through CD47 and PD-L1

Stephanie C. Casey;Ling Tong;Yulin Li;Rachel Do.
Science (2016)

627 Citations

Direct induction of cyclin D2 by Myc contributes to cell cycle progression and sequestration of p27.

Caroline Bouchard;Katja Thieke;Antje Maier;Rainer Saffrich.
The EMBO Journal (1999)

614 Citations

TGFbeta Influences Myc, Miz-1 and Smad to Control the CDK Inhibitor p15INK4b

Joan Seoane;Celio Pouponnot;Peter Staller;Manuela Schader.
Nature Cell Biology (2001)

595 Citations

Chimaeras of Myc oncoprotein and steroid receptors cause hormone-dependent transformation of cells

Eilers M;Picard D;Yamamoto Kr;Bishop Jm.
Nature (1989)

590 Citations

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