His scientific interests lie mostly in Internal medicine, Endocrinology, Kidney, Pathology and Renal function. His Internal medicine course of study focuses on Gastroenterology and Creatinine and Biopsy. His studies in Endocrinology integrate themes in fields like Inflammation and Calcitriol receptor, Receptor.
The study incorporates disciplines such as Hemeprotein, Heme oxygenase and Heme in addition to Kidney. His work deals with themes such as Cell culture, Nephrology and Transplantation, which intersect with Pathology. His research integrates issues of Fibrosis and Surgery in his study of Renal function.
Joseph P. Grande mainly investigates Internal medicine, Endocrinology, Kidney, Pathology and Immunology. His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology and Cardiology. In his study, which falls under the umbrella issue of Endocrinology, Immunohistochemistry is strongly linked to Calcitriol receptor.
His Kidney study incorporates themes from Fibrosis, Inflammation and Urology. His Pathology research integrates issues from Glomerulonephritis and Transplantation, Kidney transplantation. His Immunology research includes elements of Systemic lupus erythematosus and Lupus nephritis.
The scientist’s investigation covers issues in Internal medicine, Kidney, Pathology, Biopsy and Endocrinology. His studies deal with areas such as Preeclampsia and Cardiology as well as Internal medicine. His biological study spans a wide range of topics, including Atrophy and Urology.
He interconnects Immunofluorescence, Glomerulonephritis and Fibrillary Glomerulonephritis in the investigation of issues within Pathology. The Biopsy study combines topics in areas such as Gastroenterology, Proteinuria, Transplantation and Creatinine. His study in Endocrinology is interdisciplinary in nature, drawing from both Blot and Ligand.
Joseph P. Grande mainly focuses on Kidney, Pathology, Internal medicine, Inflammation and Endocrinology. His Kidney study combines topics in areas such as Biopsy, Cohort study and Urology. His Pathology research is multidisciplinary, incorporating elements of Fibrillary Glomerulonephritis, Heme oxygenase, Downregulation and upregulation and Kidney disease.
His Internal medicine study frequently draws connections to adjacent fields such as Cardiology. His Inflammation research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha and Adipose tissue. Joseph P. Grande is interested in Podocyte, which is a branch of Endocrinology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The Banff 97 working classification of renal allograft pathology
Lorraine C. Racusen;Kim Solez;Robert B. Colvin;Stephen M. Bonsib.
Kidney International (1999)
Predicting renal outcome in IgA nephropathy.
M G Radford;J V Donadio;E J Bergstralh;J P Grande.
Journal of The American Society of Nephrology (1997)
Evidence for antibody-mediated injury as a major determinant of late kidney allograft failure.
Robert S. Gaston;J. Michael Cecka;Bert L. Kasiske;Ann M Fieberg.
Transplantation (2010)
Predicting subsequent decline in kidney allograft function from early surveillance biopsies.
Fernando G. Cosio;Joseph P. Grande;Hani Wadei;Timothy S. Larson.
American Journal of Transplantation (2005)
Immunohistochemical localization of the 1,25(OH)2D3 receptor and calbindin D28k in human and rat pancreas
J. A. Johnson;Joseph Peter Grande;P. C. Roche;Rajiv Kumar.
American Journal of Physiology-endocrinology and Metabolism (1994)
Role of Transforming Growth Factor-β in Tissue Injury and Repair
Joseph P. Grande.
Experimental Biology and Medicine (1997)
The indispensability of heme oxygenase-1 in protecting against acute heme protein-induced toxicity in vivo.
Karl A. Nath;Jill J. Haggard;Anthony J. Croatt;Joseph P. Grande.
American Journal of Pathology (2000)
Distinct Renal Injury in Early Atherosclerosis and Renovascular Disease
Alejandro R. Chade;Martin Rodriguez-Porcel;Joseph P. Grande;James D. Krier.
Circulation (2002)
Heme protein-induced chronic renal inflammation: suppressive effect of induced heme oxygenase-1.
Karl A. Nath;Karl A. Nath;Gregory M. Vercellotti;Gregory M. Vercellotti;Joseph P. Grande;Joseph P. Grande;Hiroko Miyoshi;Hiroko Miyoshi;Hiroko Miyoshi.
Kidney International (2001)
Targeting senescent cells alleviates obesity‐induced metabolic dysfunction
Allyson K. Palmer;Ming Xu;Ming Xu;Yi Zhu;Tamar Pirtskhalava.
Aging Cell (2019)
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