What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His primary areas of study are Cell biology, Heart failure, Internal medicine, Endocrinology and Autophagy.
The study incorporates disciplines such as HEK 293 cells, Apoptosis and ATG5 in addition to Cell biology.
His research integrates issues of Myocyte, Myocardial infarction and Ischemia in his study of Heart failure.
His Autophagy research incorporates themes from Basal, Hemodynamic stress, Cardiomyopathy, Programmed cell death and Organelle.
His Cardiomyopathy study combines topics in areas such as Myocarditis, Inflammation, Proinflammatory cytokine, Immunology and Dilated cardiomyopathy.
Kinya Otsu works mostly in the field of Physiology, limiting it down to topics relating to Computational biology and, in certain cases, Autophagosome, MAP1LC3B, Chaperone-mediated autophagy and BECN1, as a part of the same area of interest.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses (2867 citations)
What are the main themes of his work throughout his whole career to date?
Kinya Otsu mostly deals with Cell biology, Internal medicine, Endocrinology, Heart failure and Autophagy.
The concepts of his Cell biology study are interwoven with issues in Inflammation and Apoptosis.
His Internal medicine research is multidisciplinary, incorporating elements of ASK1 and Cardiology.
His Endocrinology study integrates concerns from other disciplines, such as Cardiac function curve and Cardiac fibrosis.
The concepts of his Heart failure study are interwoven with issues in Downregulation and upregulation, Knockout mouse and Pathogenesis.
The study of Autophagy is intertwined with the study of Organelle in a number of ways.
He most often published in these fields:
- Cell biology (41.10%)
- Internal medicine (36.53%)
- Endocrinology (26.48%)
What were the highlights of his more recent work (between 2014-2021)?
- Cell biology (41.10%)
- Autophagy (17.81%)
- Internal medicine (36.53%)
In recent papers he was focusing on the following fields of study:
Kinya Otsu mainly focuses on Cell biology, Autophagy, Internal medicine, Heart failure and Inflammation.
His research integrates issues of Fibrosis and Immune system in his study of Cell biology.
His Autophagy research is multidisciplinary, incorporating elements of Signal transduction, Computational biology and Programmed cell death.
His Computational biology research includes elements of Chaperone-mediated autophagy and Autolysosome.
His Internal medicine study combines topics from a wide range of disciplines, such as Endocrinology and Cardiology.
His study in the field of Pressure overload also crosses realms of Cholinergic anti-inflammatory pathway.
Between 2014 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Bcl-2-like protein 13 is a mammalian Atg32 homologue that mediates mitophagy and mitochondrial fragmentation (220 citations)
- Mitochondrial Function, Biology, and Role in Disease A Scientific Statement From the American Heart Association (160 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
His main research concerns Cell biology, Mitochondrion, Autophagy, Mitophagy and Programmed cell death.
The Cell biology study combines topics in areas such as Immunology and Cell growth.
He combines subjects such as Inflammation, Adipose tissue, PI3K/AKT/mTOR pathway and Cardiac fibrosis with his study of Mitochondrion.
His is doing research in Cellular homeostasis, Autophagosome and Sequestosome 1, both of which are found in Autophagy.
His work carried out in the field of Autophagosome brings together such families of science as MAP1LC3B and BECN1.
His Programmed cell death research is multidisciplinary, relying on both Chaperone-mediated autophagy, Computational biology and Autolysosome.
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