2016 - Fellow of the American Association for the Advancement of Science (AAAS)
2015 - Member of the National Academy of Sciences
Immunology, Experimental autoimmune encephalomyelitis, Multiple sclerosis, Encephalomyelitis and Myelin basic protein are his primary areas of study. His is doing research in Autoimmune disease, Immune system, T cell, Autoimmunity and Inflammation, both of which are found in Immunology. His study in Experimental autoimmune encephalomyelitis is interdisciplinary in nature, drawing from both Interleukin 17, Pharmacology and Virology.
His biological study spans a wide range of topics, including Central nervous system, Disease and Pathogenesis. His research in Encephalomyelitis focuses on subjects like Myelin, which are connected to Antigen and Biochemistry. His Myelin basic protein research is multidisciplinary, relying on both Paralysis, Molecular biology, T-cell receptor, Epitope and Peptide.
Lawrence Steinman spends much of his time researching Immunology, Multiple sclerosis, Experimental autoimmune encephalomyelitis, Immune system and Autoimmune disease. His study in Immunology focuses on Antigen, Encephalomyelitis, Antibody, Autoimmunity and T cell. His research integrates issues of Cytotoxic T cell and T lymphocyte in his study of T cell.
Within one scientific family, he focuses on topics pertaining to Myelin basic protein under Multiple sclerosis, and may sometimes address concerns connected to Major histocompatibility complex and Peptide. His work carried out in the field of Experimental autoimmune encephalomyelitis brings together such families of science as Neuroinflammation, Pharmacology and Cell biology. Lawrence Steinman has included themes like Autoantibody and Immunotherapy in his Autoimmune disease study.
His primary areas of study are Immunology, Multiple sclerosis, Experimental autoimmune encephalomyelitis, Immune system and Antibody. His study on Immunology is mostly dedicated to connecting different topics, such as Disease. His Multiple sclerosis research is multidisciplinary, relying on both Interferon, Internal medicine, Pathogenesis and Neuroscience.
While the research belongs to areas of Experimental autoimmune encephalomyelitis, Lawrence Steinman spends his time largely on the problem of Neuroinflammation, intersecting his research to questions surrounding Microglia. His Immune system study combines topics from a wide range of disciplines, such as Demyelinating disease and Myelin. His biological study spans a wide range of topics, including Virus, Virology, Cancer research and Cancer.
His primary areas of investigation include Immunology, Multiple sclerosis, Experimental autoimmune encephalomyelitis, Immune system and Internal medicine. His Immunology study frequently draws connections to other fields, such as Disease. His Multiple sclerosis research incorporates elements of Tolerability, Central nervous system, Neuroscience and Cytokine.
His research integrates issues of Neuroinflammation, Adoptive cell transfer, Cell biology, Encephalomyelitis and Autoimmunity in his study of Experimental autoimmune encephalomyelitis. The various areas that Lawrence Steinman examines in his Immune system study include Lymphopoiesis, Sphingosine-1-phosphate, B cell, APOM and Bone marrow. The Internal medicine study combines topics in areas such as Gastroenterology and Endocrinology.
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Prevention of experimental autoimmune encephalomyelitis by antibodies against alpha 4 beta 1 integrin.
Ted A. Yednock;Catherine Cannon;Lawrence C. Fritz;Francisco Sanchez-Madrid.
A brief history of T(H)17, the first major revision in the T(H)1/T(H)2 hypothesis of T cell-mediated tissue damage.
Nature Medicine (2007)
The design, synthesis, and evaluation of molecules that enable or enhance cellular uptake: Peptoid molecular transporters
Paul A. Wender;Dennis J. Mitchell;Kanaka Pattabiraman;Erin T. Pelkey.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis
Christopher Lock;Guy Hermans;Rosetta Pedotti;Andrea Brendolan.
Nature Medicine (2002)
The HMG-CoA reductase inhibitor, atorvastatin, promotes a Th2 bias and reverses paralysis in central nervous system autoimmune disease
Sawsan Youssef;Olaf Stüve;Juan C. Patarroyo;Pedro J. Ruiz;Pedro J. Ruiz.
The T Lymphocyte in Experimental Allergic Encephalomyelitis
Scott S. Zamvil;Lawrence Steinman.
Annual Review of Immunology (1990)
Polyarginine enters cells more efficiently than other polycationic homopolymers.
D.J. Mitchell;L. Steinman;D.T. Kim;C.G. Fathman.
Journal of Peptide Research (2000)
Mice with a disrupted IFN-gamma gene are susceptible to the induction of experimental autoimmune encephalomyelitis (EAE).
I A Ferber;S Brocke;C Taylor-Edwards;W Ridgway.
Journal of Immunology (1996)
Limited heterogeneity of T cell receptors from lymphocytes mediating autoimmune encephalomyelitis allows specific immune intervention
Hans Acha-Orbea;Dennis J. Mitchell;Luika Timmermann;David C. Wraith.
The influence of the proinflammatory cytokine, osteopontin, on autoimmune demyelinating disease.
Dorothée Chabas;Sergio E. Baranzini;Dennis Mitchell;Claude C. A. Bernard.
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