2018 - Fellow of the American Mathematical Society For contributions to analytic number theory, cryptography, and theoretical physics.
2004 - Fellow of the American Association for the Advancement of Science (AAAS)
Stephen D. Miller focuses on Immunology, Experimental autoimmune encephalomyelitis, Immune system, T cell and Myelin. His Immunology research focuses on Demyelinating disease, Autoimmune disease, Epitope, Antigen and Encephalomyelitis. His research on Experimental autoimmune encephalomyelitis also deals with topics like
His work carried out in the field of Immune system brings together such families of science as Neuroinflammation and Microglia. His T cell study combines topics in areas such as Receptor, Tolerance induction, Molecular biology and Interleukin 10. The concepts of his Myelin study are interwoven with issues in Priming and Cellular differentiation.
His primary areas of study are Immunology, T cell, Immune system, Experimental autoimmune encephalomyelitis and Multiple sclerosis. Antigen, Autoimmune disease, Demyelinating disease, Autoimmunity and Immune tolerance are the primary areas of interest in his Immunology study. Stephen D. Miller interconnects Spleen, Molecular biology, Major histocompatibility complex and Cell biology in the investigation of issues within T cell.
His Immune system study combines topics from a wide range of disciplines, such as Cell, Microglia and Suppressor. His biological study deals with issues like Encephalomyelitis, which deal with fields such as Adoptive cell transfer. His Multiple sclerosis research incorporates themes from Disease, Pathogenesis, Pathology and Central nervous system, Neuroscience.
Stephen D. Miller mostly deals with Immunology, Immune system, T cell, Immune tolerance and Experimental autoimmune encephalomyelitis. His study in Antigen, Autoimmune disease, Autoimmunity, Multiple sclerosis and Inflammation falls within the category of Immunology. His Immune system research integrates issues from Proinflammatory cytokine, Cytokine and Monocyte.
His T cell research incorporates elements of Acquired immune system, Cancer research and CD8. His Immune tolerance research focuses on Transplantation and how it relates to Splenocyte. His Experimental autoimmune encephalomyelitis research includes themes of Spleen and Neuroinflammation.
His scientific interests lie mostly in Immunology, Experimental autoimmune encephalomyelitis, Immune tolerance, Multiple sclerosis and Immune system. In most of his Immunology studies, his work intersects topics such as Myelin. Stephen D. Miller combines subjects such as Spleen and Cellular differentiation with his study of Experimental autoimmune encephalomyelitis.
His Immune tolerance study integrates concerns from other disciplines, such as Effector, Disease and Immunotherapy. Stephen D. Miller has researched Multiple sclerosis in several fields, including Knockout mouse and Central nervous system. His studies deal with areas such as Virus, Bystander effect and Etiology as well as Immune system.
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Microglia Initiate Central Nervous System Innate and Adaptive Immune Responses through Multiple TLRs
Julie K. Olson;Stephen D. Miller.
Journal of Immunology (2004)
Cutting edge: CD4+CD25+ regulatory T cells suppress antigen-specific autoreactive immune responses and central nervous system inflammation during active experimental autoimmune encephalomyelitis.
Adam P. Kohm;Pamela A. Carpentier;Holly A. Anger;Stephen D. Miller.
Journal of Immunology (2002)
Epitope spreading in immune-mediated diseases: implications for immunotherapy
Carol L. Vanderlugt;Stephen D. Miller.
Nature Reviews Immunology (2002)
Epitope spreading initiates in the CNS in two mouse models of multiple sclerosis
Eileen J McMahon;Eileen J McMahon;Samantha L Bailey;Carol Vanderlugt Castenada;Hanspeter Waldner.
Nature Medicine (2005)
Persistent infection with Theiler's virus leads to CNS autoimmunity via epitope spreading
Stephen D. Miller;Carol L. Vanderlugt;Wendy Smith Begolka;Winnie Pao.
Nature Medicine (1997)
Functional evidence for epitope spreading in the relapsing pathology of experimental autoimmune encephalomyelitis.
B L McRae;C L Vanderlugt;M C Dal Canto;S D Miller.
Journal of Experimental Medicine (1995)
Guidelines for the use of flow cytometry and cell sorting in immunological studies (second edition)
Andrea Cossarizza;Hyun Dong Chang;Andreas Radbruch;Andreas Acs.
European Journal of Immunology (2019)
Human bone marrow-derived mesenchymal stem cells induce Th2-polarized immune response and promote endogenous repair in animal models of multiple sclerosis.
Lianhua Bai;Donald P. Lennon;Valerie Eaton;Kari Maier.
CNS myeloid DCs presenting endogenous myelin peptides 'preferentially' polarize CD4+ TH-17 cells in relapsing EAE
Samantha L Bailey;Bettina Schreiner;Eileen J McMahon;Stephen D Miller.
Nature Immunology (2007)
An important role for the chemokine macrophage inflammatory protein-1 alpha in the pathogenesis of the T cell-mediated autoimmune disease, experimental autoimmune encephalomyelitis.
W J Karpus;N W Lukacs;B L McRae;R M Strieter.
Journal of Immunology (1995)
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