2006 - American Association of Immunologists Lifetime Achievement Award
2006 - AAI Lifetime Achievement Award, American Association of Immunologists
1998 - Jessie Stevenson Kovalenko Medal, US National Academy of Sciences For his landmark discovery and identification of genes that control immune responsiveness, and for his subsequent elucidation of mechanisms of antigen recognition and induction of the immune response.
1994 - Fellow of the Royal Society, United Kingdom
1987 - Fellow of the American Association for the Advancement of Science (AAAS)
1987 - Paul Ehrlich and Ludwig Darmstaedter Prize
1984 - Fellow of the American Academy of Arts and Sciences
1984 - Member of the National Academy of Medicine (NAM)
1977 - Member of the National Academy of Sciences
Member of the Association of American Physicians
His primary areas of investigation include Immunology, Antigen, Molecular biology, T cell and Major histocompatibility complex. His Immunology research includes elements of Endocrinology and Internal medicine. Hugh O. McDevitt has researched Antigen in several fields, including Antibody, Gene and Immune system.
His Molecular biology study combines topics from a wide range of disciplines, such as Apoptosis, Regulation of gene expression, Kidney and Allergy. His work in T cell tackles topics such as Interleukin 2 which are related to areas like Transplant rejection, Cancer research and Interferon gamma. His study on Major histocompatibility complex is covered under Genetics.
His primary areas of study are Immunology, Antigen, Molecular biology, Major histocompatibility complex and Genetics. The study incorporates disciplines such as Internal medicine and Nod in addition to Immunology. His research on Antigen also deals with topics like
His Molecular biology research is multidisciplinary, incorporating perspectives in Antiserum, Biochemistry, Complementary DNA, Epitope and Cytotoxic T cell. His work deals with themes such as Experimental autoimmune encephalomyelitis and Peptide sequence, which intersect with Major histocompatibility complex. His NOD mice study integrates concerns from other disciplines, such as Adoptive cell transfer, IL-2 receptor and Insulitis.
His primary areas of investigation include Immunology, NOD mice, Nod, T cell and Antigen. His Immunology study incorporates themes from Internal medicine and Type 1 diabetes. His NOD mice research is multidisciplinary, relying on both Adoptive cell transfer, Alpha interferon, Immune tolerance and Immunotherapy.
His work carried out in the field of T cell brings together such families of science as Interleukin 2, Cytokine and Molecular biology. In his research, Human leukocyte antigen, Allele, MHC class II and Major histocompatibility complex is intimately related to Epitope, which falls under the overarching field of Molecular biology. Antigen is a primary field of his research addressed under Genetics.
His scientific interests lie mostly in Immunology, NOD mice, T cell, Nod and Tumor necrosis factor alpha. His is involved in several facets of Immunology study, as is seen by his studies on Autoimmunity and Autoimmune disease. His NOD mice research incorporates elements of Adoptive cell transfer, IL-2 receptor and Immune tolerance.
The various areas that Hugh O. McDevitt examines in his T cell study include Interleukin 2, Epitope, Antigen and Islet. Hugh O. McDevitt interconnects Internal medicine and T-cell receptor in the investigation of issues within Nod. As part of one scientific family, Hugh O. McDevitt deals mainly with the area of Allele, narrowing it down to issues related to the Reporter gene, and often Molecular biology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Effects of a polymorphism in the human tumor necrosis factor α promoter on transcriptional activation
Anthony G. Wilson;Julian A. Symons;Tarra L. McDowell;Hugh O. McDevitt.
Proceedings of the National Academy of Sciences of the United States of America (1997)
HLA-DQ beta gene contributes to susceptibility and resistance to insulin-dependent diabetes mellitus.
John A. Todd;John I. Bell;John I. Bell;Hugh O. McDevitt.
Nature (1987)
Insulin-Dependent Diabetes Mellitus
Roland Michael Tisch;Hugh McDevitt.
Cell (1996)
Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseases
Roland S. Liblau;Steven M. Singer;Hugh O. McDevitt.
Immunology Today (1995)
In Vitro–expanded Antigen-specific Regulatory T Cells Suppress Autoimmune Diabetes
Qizhi Tang;Kammi J. Henriksen;Mingying Bi;Erik B. Finger.
Journal of Experimental Medicine (2004)
Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice
Roland Tisch;Xiao Dong Yang;Steven M. Singer;Roland S. Liblau.
Nature (1993)
Limited heterogeneity of T cell receptors from lymphocytes mediating autoimmune encephalomyelitis allows specific immune intervention
Hans Acha-Orbea;Dennis J. Mitchell;Luika Timmermann;David C. Wraith.
Cell (1988)
Heritable major histocompatibility complex class II-associated differences in production of tumor necrosis factor alpha: relevance to genetic predisposition to systemic lupus erythematosus.
Chaim O. Jacob;Zdenka Fronek;Gail D. Lewis;May Koo.
Proceedings of the National Academy of Sciences of the United States of America (1990)
Genetic control of specific immune responses.
Hugh O. Mcdevitt;Baruj Benacerraf.
Advances in Immunology (1969)
Genetic control of specific immune responses.
McDevitt Ho;Benacerraf B.
Advances in Immunology (1969)
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