Nicholas W. Lukacs mainly focuses on Immunology, Chemokine, Lung, Cytokine and Monocyte. His research related to Inflammation, Immune system, Eosinophil, Macrophage inflammatory protein and Eotaxin might be considered part of Immunology. His research in Chemokine intersects with topics in Peripheral blood mononuclear cell, Interleukin 8 and Endocrinology.
Nicholas W. Lukacs interconnects Infiltration, Respiratory system, Fibrosis, Interleukin 13 and Airway in the investigation of issues within Lung. His study in Cytokine is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Necrosis, Endogeny and Cell biology. His Monocyte research incorporates themes from CCR2, Autoimmune disease, T helper cell, Interleukin 4 and Granuloma.
His main research concerns Immunology, Chemokine, Immune system, Inflammation and Cytokine. His Immunology research integrates issues from Lung and Respiratory system. His studies examine the connections between Chemokine and genetics, as well as such issues in Monocyte, with regards to Chemotaxis.
His work carried out in the field of Immune system brings together such families of science as Receptor and Antigen. His Inflammation study combines topics from a wide range of disciplines, such as Eosinophilia, Immunopathology and Pathogenesis. His Cytokine research is multidisciplinary, relying on both Pathology, Dendritic cell and Cell biology.
Nicholas W. Lukacs spends much of his time researching Immunology, Immune system, Inflammation, Asthma and Cell biology. His study on Immunology is mostly dedicated to connecting different topics, such as Lung. His study in Immune system is interdisciplinary in nature, drawing from both Regulation of gene expression and Antigen.
The Inflammation study combines topics in areas such as Acquired immune system, Fibrosis and Microbiology. As part of the same scientific family, Nicholas W. Lukacs usually focuses on Cell biology, concentrating on Epigenetics and intersecting with FOXP3. He has included themes like Autophagy and Unfolded protein response in his Cytokine study.
Immunology, Immune system, Cytokine, Cell biology and Immunity are his primary areas of study. The concepts of his Immunology study are interwoven with issues in Microbiome and Hematopoietic stem cell transplantation. His Immune system study integrates concerns from other disciplines, such as Virus, Disease and Microbiology.
In Cytokine, Nicholas W. Lukacs works on issues like Autophagy, which are connected to Physiology. His Immunity research is multidisciplinary, incorporating elements of Inflammation and Interleukin. His Inflammation study incorporates themes from Interferon and Methyltransferase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Autophagy (2012)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Abnormalities in Monocyte Recruitment and Cytokine Expression in Monocyte Chemoattractant Protein 1–deficient Mice
Bao Lu;Barbara J. Rutledge;Long Gu;Joseph Fiorillo.
Journal of Experimental Medicine (1998)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Parasites & Vectors (2016)
Neonatal gut microbiota associates with childhood multisensitized atopy and T cell differentiation
Kei E Fujimura;Alexandra R Sitarik;Suzanne Havstad;Din L Lin.
Nature Medicine (2016)
TLR3 is an endogenous sensor of tissue necrosis during acute inflammatory events
Karen A. Cavassani;Makoto Ishii;Haitao Wen;Matthew A. Schaller.
Journal of Experimental Medicine (2008)
Differential CC chemokine-induced enhancement of T helper cell cytokine production.
W J Karpus;N W Lukacs;K J Kennedy;W S Smith.
Journal of Immunology (1997)
An important role for the chemokine macrophage inflammatory protein-1 alpha in the pathogenesis of the T cell-mediated autoimmune disease, experimental autoimmune encephalomyelitis.
W J Karpus;N W Lukacs;B L McRae;R M Strieter.
Journal of Immunology (1995)
Balance of inflammatory cytokines related to severity and mortality of murine sepsis.
Keith R. Walley;Nicholas W. Lukacs;Theodore J. Standiford;Robert M. Strieter.
Infection and Immunity (1996)
Epigenetic regulation of the alternatively activated macrophage phenotype
Makoto Ishii;Haitao Wen;Callie A.S. Corsa;Tianju Liu.
Blood (2009)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Michigan–Ann Arbor
Cedars-Sinai Medical Center
University of Virginia
University of Michigan–Ann Arbor
University of Michigan–Ann Arbor
University of California, San Francisco
Augusta University
Henry Ford Health System
University of Virginia
University of California, San Francisco
Facebook (United States)
Universidade de São Paulo
Algorand Foundation
Vrije Universiteit Brussel
Toyota Motor Corporation (Japan)
Tokyo Institute of Technology
University of Maryland, Baltimore
Nippon Medical School
University of East Anglia
National Institutes of Health
Complutense University of Madrid
Ulsan National Institute of Science and Technology
University of Poitiers
University of Michigan–Ann Arbor
University of British Columbia
University College London