D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 81 Citations 15,102 290 World Ranking 985 National Ranking 530
Medicine D-index 85 Citations 17,111 348 World Ranking 9765 National Ranking 5164

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

His primary areas of study are Immunology, Lung, Chemokine, Pulmonary fibrosis and Fibrosis. His Immunology research focuses on Inflammation, Sepsis, Interleukin 13, Innate immune system and Aspergillus fumigatus. His studies deal with areas such as Exacerbation, Allergic sensitization and Respiratory system as well as Lung.

His Chemokine research incorporates themes from Acetaminophen and Monocyte. He has included themes like Prostaglandin E2, Idiopathic pulmonary fibrosis, Usual interstitial pneumonia and Zebularine in his Pulmonary fibrosis study. In his research on the topic of Fibrosis, Fibroblast, Macrophage colony-stimulating factor, Cellular differentiation, Chromatin and Histone is strongly related with Molecular biology.

His most cited work include:

  • Serum amyloid P therapeutically attenuates murine bleomycin-induced pulmonary fibrosis via its effects on macrophages. (152 citations)
  • Protection from Fluorescein Isothiocyanate-Induced Fibrosis in IL-13-Deficient, but Not IL-4-Deficient, Mice Results from Impaired Collagen Synthesis by Fibroblasts (152 citations)
  • Future directions in idiopathic pulmonary fibrosis research. An NHLBI workshop report. (147 citations)

What are the main themes of his work throughout his whole career to date?

Cory M. Hogaboam mostly deals with Immunology, Lung, Chemokine, Idiopathic pulmonary fibrosis and Immune system. His research on Immunology frequently connects to adjacent areas such as Receptor. His study in Lung is interdisciplinary in nature, drawing from both Fibrosis and Pathology.

His research integrates issues of Bronchoalveolar lavage and Sepsis in his study of Chemokine. The various areas that Cory M. Hogaboam examines in his Idiopathic pulmonary fibrosis study include Pulmonary fibrosis and Cancer research. The various areas that Cory M. Hogaboam examines in his Immune system study include Aspergillus fumigatus and Microbiology.

He most often published in these fields:

  • Immunology (56.98%)
  • Lung (24.58%)
  • Chemokine (23.18%)

What were the highlights of his more recent work (between 2016-2021)?

  • Idiopathic pulmonary fibrosis (18.72%)
  • Cancer research (17.32%)
  • Lung (24.58%)

In recent papers he was focusing on the following fields of study:

His main research concerns Idiopathic pulmonary fibrosis, Cancer research, Lung, Pulmonary fibrosis and Fibrosis. The concepts of his Cancer research study are interwoven with issues in Myofibroblast, Progenitor cell, Tumor microenvironment, Fibroblast and Mesenchymal stem cell. His work carried out in the field of Lung brings together such families of science as Immune checkpoint, Immunology, Immune system and Pathology.

His research in Immunology is mostly focused on Innate immune system. His research integrates issues of Humanized mouse, Extracellular matrix, Protein kinase B and Interleukin 8 in his study of Pulmonary fibrosis. His Fibrosis study combines topics in areas such as Inflammation, Epithelial Damage, Epithelial–mesenchymal transition and Gastroenterology.

Between 2016 and 2021, his most popular works were:

  • Microbes Are Associated with Host Innate Immune Response in Idiopathic Pulmonary Fibrosis (70 citations)
  • Quercetin Enhances Ligand-induced Apoptosis in Senescent Idiopathic Pulmonary Fibrosis Fibroblasts and Reduces Lung Fibrosis In Vivo. (39 citations)
  • Targeting of TAM Receptors Ameliorates Fibrotic Mechanisms in Idiopathic Pulmonary Fibrosis. (37 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

Cory M. Hogaboam spends much of his time researching Idiopathic pulmonary fibrosis, Lung, Cancer research, Pulmonary fibrosis and Fibrosis. His Idiopathic pulmonary fibrosis research incorporates themes from Immune checkpoint, Epithelium, Pathology, Signal transduction and Fibroblast. His Lung research includes themes of Respiratory tract and Immune system, Immunology, Immunity, Innate immune system.

His work deals with themes such as Epithelial–mesenchymal transition, COPD and Hyperplasia, which intersect with Immunology. His research in Pulmonary fibrosis intersects with topics in Apoptosis, Protein kinase B, Lung injury and Extracellular matrix. His Fibrosis study incorporates themes from Inflammation, Endothelium, Downregulation and upregulation and PD-L1.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Protection from Fluorescein Isothiocyanate-Induced Fibrosis in IL-13-Deficient, but Not IL-4-Deficient, Mice Results from Impaired Collagen Synthesis by Fibroblasts

Jill E. Kolodsick;Galen B. Toews;Claudia Jakubzick;Cory Hogaboam.
Journal of Immunology (2004)

242 Citations

Respiratory syncytial virus predisposes mice to augmented allergic airway responses via IL-13-mediated mechanisms.

Nicholas W. Lukacs;Kim K. Tekkanat;Aaron Berlin;Cory M. Hogaboam.
Journal of Immunology (2001)

217 Citations

Epigenetic regulation of dendritic cell–derived interleukin-12 facilitates immunosuppression after a severe innate immune response

Haitao Wen;Yali Dou;Cory M. Hogaboam;Steven L. Kunkel.
Blood (2008)

216 Citations

Future directions in idiopathic pulmonary fibrosis research. An NHLBI workshop report.

Timothy S. Blackwell;Andrew M. Tager;Zea Borok;Bethany B. Moore.
American Journal of Respiratory and Critical Care Medicine (2014)

204 Citations

The Chemokine RANTES Is a Crucial Mediator of the Progression from Acute to Chronic Colitis in the Rat

Maureen N. Ajuebor;Cory M. Hogaboam;Steven L. Kunkel;Amanda E. I. Proudfoot.
Journal of Immunology (2001)

200 Citations

MIP‐1α[CCL3] acting on the CCR1 receptor mediates neutrophil migration in immune inflammation via sequential release of TNF‐α and LTB4

Cleber D. L. Ramos;Claudio Canetti;Claudio Canetti;Janeusa T. Souto;Janeusa T. Souto;João S. Silva.
Journal of Leukocyte Biology (2005)

196 Citations

Differential monocyte chemoattractant protein-1 and chemokine receptor 2 expression by murine lung fibroblasts derived from Th1- and Th2-type pulmonary granuloma models.

Cory M. Hogaboam;Cynthia L. Bone-Larson;Scott Lipinski;Nicholas W. Lukacs.
Journal of Immunology (1999)

192 Citations

Serum amyloid P therapeutically attenuates murine bleomycin-induced pulmonary fibrosis via its effects on macrophages.

Lynne A Murray;Rogério S. Rosada;Ana Paula Duarte Moreira;Amrita D Joshi.
PLOS ONE (2010)

190 Citations

Novel Role of Transmembrane SCF for Mast Cell Activation and Eotaxin Production in Mast Cell-Fibroblast Interactions

Cory Hogaboam;Steven L. Kunkel;Robert M. Strieter;Dennis D. Taub.
Journal of Immunology (1998)

189 Citations

Therapeutic attenuation of pulmonary fibrosis via targeting of IL-4- and IL-13-responsive cells.

Claudia Jakubzick;Esther S. Choi;Bharat H. Joshi;Michael P. Keane.
Journal of Immunology (2003)

186 Citations

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