D-Index & Metrics Best Publications

Naftali Kaminski

Yale University
United States

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Medicine D-index 98 Citations 40,318 533 World Ranking 5313 National Ranking 2940

Overview

What is he best known for?

The fields of study Naftali Kaminski is best known for:

Gene
Cancer
Enzyme

His Cell biology study frequently involves adjacent topics like Transforming growth factor and Signal transduction. His research combines Cell biology and Signal transduction. His research on Gene expression profiling and Microarray is centered around Gene expression. Naftali Kaminski performs multidisciplinary studies into Gene expression profiling and Gene expression in his work. His Internal medicine study frequently links to related topics such as Receptor. Much of his study explores Receptor relationship to Internal medicine. Naftali Kaminski combines Lung and Idiopathic pulmonary fibrosis in his studies. In his works, he undertakes multidisciplinary study on Idiopathic pulmonary fibrosis and Lung. He performs multidisciplinary study on Genetics and Cell in his works.

His most cited work include:

A Mechanism for Regulating Pulmonary Inflammation and Fibrosis: The Integrin αvβ6 Binds and Activates Latent TGF β1 (1819 citations)
SARS-CoV-2 Receptor ACE2 Is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Detected in Specific Cell Subsets across Tissues (1731 citations)
Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis (1524 citations)

What are the main themes of his work throughout his whole career to date

His work on Chemotherapy as part of general Bleomycin study is frequently linked to Cancer research, bridging the gap between disciplines. In his articles, Naftali Kaminski combines various disciplines, including Chemotherapy and Bleomycin. Cancer research and Genetics are two areas of study in which he engages in interdisciplinary work. His Genetics study frequently involves adjacent topics like Gene expression. His Disease research extends to the thematically linked field of Internal medicine. In his research, Naftali Kaminski undertakes multidisciplinary study on Disease and Pathology. As part of his studies on Pathology, he often connects relevant subjects like Fibrosis. He carries out multidisciplinary research, doing studies in Fibrosis and Pulmonary fibrosis. Pulmonary fibrosis and Lung are two areas of study in which he engages in interdisciplinary research.

Naftali Kaminski most often published in these fields:

Internal medicine (66.67%)
Gene (55.43%)
Lung (54.26%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis.

John S Munger;Xiaozhu Huang;Hisaaki Kawakatsu;Mark J.D Griffiths.
Cell (1999)

2791 Citations

Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis

Christopher Lock;Guy Hermans;Rosetta Pedotti;Andrea Brendolan.
Nature Medicine (2002)

1952 Citations

Mesenchymal stem cell engraftment in lung is enhanced in response to bleomycin exposure and ameliorates its fibrotic effects.

Luis A. Ortiz;Frederica Gambelli;Christine McBride;Dina Gaupp.
Proceedings of the National Academy of Sciences of the United States of America (2003)

1780 Citations

SARS-CoV-2 Receptor ACE2 Is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Detected in Specific Cell Subsets across Tissues.

Carly G.K. Ziegler;Samuel J. Allon;Sarah K. Nyquist;Ian M. Mbano.
Cell (2020)

1760 Citations

Acute Exacerbations of Idiopathic Pulmonary Fibrosis

Harold R. Collard;Bethany B. Moore;Kevin R. Flaherty;Kevin K. Brown.
American Journal of Respiratory and Critical Care Medicine (2007)

1220 Citations

miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis

Gang Liu;Arnaud Friggeri;Yanping Yang;Jadranka Milosevic.
Journal of Experimental Medicine (2010)

961 Citations

Acute Exacerbation of Idiopathic Pulmonary Fibrosis. An International Working Group Report

Collard Hr;Ryerson Cj;Corte Tj;Jenkins G.
American Journal of Respiratory and Critical Care Medicine (2016)

903 Citations

Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans

Fengrong Zuo;Naftali Kaminski;Elsie Eugui;John Allard.
Proceedings of the National Academy of Sciences of the United States of America (2002)

686 Citations

Genome-wide association study identifies multiple susceptibility loci for pulmonary fibrosis.

Tasha E. Fingerlin;Elissa Murphy;Weiming Zhang;Anna L. Peljto.
Nature Genetics (2013)

658 Citations

Loss of integrin alpha(v)beta6-mediated TGF-beta activation causes Mmp12-dependent emphysema.

David G. Morris;Xiaozhu Huang;Naftali Kaminski;Yanli Wang.
Nature (2003)

652 Citations

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Frequent Co-Authors

External Links

Personal Website for Naftali Kaminski