D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 83 Citations 31,956 306 World Ranking 8205 National Ranking 4466

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Cancer

Idiopathic pulmonary fibrosis, Pulmonary fibrosis, Immunology, Pathology and Lung are his primary areas of study. His research on Idiopathic pulmonary fibrosis concerns the broader Internal medicine. His Pulmonary fibrosis study combines topics from a wide range of disciplines, such as Cancer research, microRNA, Intensive care and In situ hybridization.

His Immunology research incorporates themes from Gene expression and Disease. Naftali Kaminski has included themes like Gene expression profiling, RAGE, Glycation, Hypersensitivity pneumonitis and In vivo in his Pathology study. His Bronchoalveolar lavage study, which is part of a larger body of work in Lung, is frequently linked to Bleomycin, bridging the gap between disciplines.

His most cited work include:

  • The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis. (1645 citations)
  • Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis (1457 citations)
  • Mesenchymal stem cell engraftment in lung is enhanced in response to bleomycin exposure and ameliorates its fibrotic effects. (1227 citations)

What are the main themes of his work throughout his whole career to date?

Naftali Kaminski focuses on Idiopathic pulmonary fibrosis, Pathology, Lung, Immunology and Pulmonary fibrosis. His study looks at the intersection of Idiopathic pulmonary fibrosis and topics like Cancer research with microRNA. His studies link Microarray analysis techniques with Pathology.

Naftali Kaminski interconnects Extracellular matrix and COPD in the investigation of issues within Lung. His research on Immunology also deals with topics like

  • Gene expression which intersects with area such as Cell biology,
  • Disease and related Bioinformatics. He conducts interdisciplinary study in the fields of Pulmonary fibrosis and Bleomycin through his research.

He most often published in these fields:

  • Idiopathic pulmonary fibrosis (31.68%)
  • Pathology (23.76%)
  • Lung (21.92%)

What were the highlights of his more recent work (between 2017-2021)?

  • Idiopathic pulmonary fibrosis (31.68%)
  • Lung (21.92%)
  • Cell biology (10.87%)

In recent papers he was focusing on the following fields of study:

His main research concerns Idiopathic pulmonary fibrosis, Lung, Cell biology, Immunology and Internal medicine. His Idiopathic pulmonary fibrosis research is multidisciplinary, incorporating elements of Gene expression, Fibrosis, Pulmonary fibrosis, Pathology and Interstitial lung disease. While working on this project, Naftali Kaminski studies both Pulmonary fibrosis and Bleomycin.

His Lung study also includes fields such as

  • Cancer research which is related to area like Fibroblast and microRNA,
  • Cell and related RNA. As a part of the same scientific family, Naftali Kaminski mostly works in the field of Immunology, focusing on Bronchoalveolar lavage and, on occasion, Sarcoidosis. His Internal medicine research includes themes of Gastroenterology, Endocrinology and Cardiology.

Between 2017 and 2021, his most popular works were:

  • SARS-CoV-2 Receptor ACE2 Is an Interferon-Stimulated Gene in Human Airway Epithelial Cells and Is Detected in Specific Cell Subsets across Tissues. (901 citations)
  • Thyroid hormone inhibits lung fibrosis in mice by improving epithelial mitochondrial function (112 citations)
  • Single-cell RNA-seq reveals ectopic and aberrant lung-resident cell populations in idiopathic pulmonary fibrosis. (96 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Cancer

His primary scientific interests are in Idiopathic pulmonary fibrosis, Lung, Fibrosis, Pathology and Cell biology. His studies in Idiopathic pulmonary fibrosis integrate themes in fields like Retrospective cohort study, microRNA, Interstitial lung disease, Pulmonary fibrosis and Lung disease. Naftali Kaminski has researched Pulmonary fibrosis in several fields, including Disease and Mitochondrial biogenesis.

His studies deal with areas such as COPD, Cystic fibrosis, Immune system and Transplantation as well as Lung. His Fibrosis study incorporates themes from Cancer research, Complete blood count and Gene regulatory network. While the research belongs to areas of Pathology, Naftali Kaminski spends his time largely on the problem of Telomere, intersecting his research to questions surrounding Elastin, Collagen, type I, alpha 1 and Extracellular matrix.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis.

John S Munger;Xiaozhu Huang;Hisaaki Kawakatsu;Mark J.D Griffiths.
Cell (1999)

2692 Citations

Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis

Christopher Lock;Guy Hermans;Rosetta Pedotti;Andrea Brendolan.
Nature Medicine (2002)

1852 Citations

Mesenchymal stem cell engraftment in lung is enhanced in response to bleomycin exposure and ameliorates its fibrotic effects.

Luis A. Ortiz;Frederica Gambelli;Christine McBride;Dina Gaupp.
Proceedings of the National Academy of Sciences of the United States of America (2003)

1708 Citations

Acute Exacerbations of Idiopathic Pulmonary Fibrosis

Harold R. Collard;Bethany B. Moore;Kevin R. Flaherty;Kevin K. Brown.
American Journal of Respiratory and Critical Care Medicine (2007)

1131 Citations

miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis

Gang Liu;Arnaud Friggeri;Yanping Yang;Jadranka Milosevic.
Journal of Experimental Medicine (2010)

891 Citations

Acute Exacerbation of Idiopathic Pulmonary Fibrosis. An International Working Group Report

Collard Hr;Ryerson Cj;Corte Tj;Jenkins G.
American Journal of Respiratory and Critical Care Medicine (2016)

671 Citations

Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans

Fengrong Zuo;Naftali Kaminski;Elsie Eugui;John Allard.
Proceedings of the National Academy of Sciences of the United States of America (2002)

656 Citations

Loss of integrin αvβ6-mediated TGF-β activation causes Mmp12-dependent emphysema

David G. Morris;Xiaozhu Huang;Naftali Kaminski;Yanli Wang.
Nature (2003)

564 Citations

Genome-wide association study identifies multiple susceptibility loci for pulmonary fibrosis.

Tasha E. Fingerlin;Elissa Murphy;Weiming Zhang;Anna L. Peljto.
Nature Genetics (2013)

546 Citations

TGF-β is a critical mediator of acute lung injury

Jean-Francois Pittet;Mark J.D. Griffiths;Tom Geiser;Naftali Kaminski.
Journal of Clinical Investigation (2001)

535 Citations

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