Member of the Association of American Physicians
Myofibroblast, Fibrosis, Cell biology, Idiopathic pulmonary fibrosis and Lung are his primary areas of study. Victor J. Thannickal combines subjects such as Lung injury and Cellular differentiation with his study of Myofibroblast. His Fibrosis research includes themes of Fibrocyte, Cancer research, CCR2 and Collagen, type I, alpha 1.
His Cell biology research incorporates themes from Cell adhesion and Cytokine. His Idiopathic pulmonary fibrosis research incorporates elements of Oxidative stress, Pulmonary fibrosis and Immunology, Pathogenesis. His Lung study incorporates themes from Chemokine receptor and Bone marrow, Pathology.
His main research concerns Idiopathic pulmonary fibrosis, Cell biology, Lung, Fibrosis and Pathology. His Idiopathic pulmonary fibrosis research is multidisciplinary, incorporating elements of Pulmonary fibrosis, Lung injury and Immunology, Pathogenesis. His Cell biology research is multidisciplinary, incorporating perspectives in Myofibroblast and Gene knockdown.
The study incorporates disciplines such as Wound healing, Fibroblast, Cellular differentiation and NOX4 in addition to Myofibroblast. His research in Fibrosis intersects with topics in Oxidative stress, Cancer research and Senescence. His studies deal with areas such as Extracellular matrix and Usual interstitial pneumonia as well as Pathology.
Victor J. Thannickal mainly focuses on Idiopathic pulmonary fibrosis, Cancer research, Fibrosis, Lung and Cell biology. The concepts of his Idiopathic pulmonary fibrosis study are interwoven with issues in Lung injury, Pathogenesis, Bioinformatics and Cellular senescence. His Cancer research research is multidisciplinary, incorporating elements of Cell, Ex vivo, Myofibroblast, Senescence and Fibroblast.
His Pulmonary fibrosis study in the realm of Fibrosis connects with subjects such as Bleomycin. His Lung research incorporates themes from Genetics, Epigenetics and Immunology. As a member of one scientific family, Victor J. Thannickal mostly works in the field of Cell biology, focusing on Gene silencing and, on occasion, MALAT1, Gene knockdown and Stromal cell.
His primary areas of study are Idiopathic pulmonary fibrosis, Fibrosis, Cell biology, Lung injury and Pulmonary fibrosis. The study incorporates disciplines such as Cancer research, Proteostasis and Pathology in addition to Idiopathic pulmonary fibrosis. His work investigates the relationship between Cancer research and topics such as Senescence that intersect with problems in MyoD, Myofibroblast and Proinflammatory cytokine.
His study in Fibrosis is interdisciplinary in nature, drawing from both Process, Computational biology and Organ function. His work is dedicated to discovering how Cell biology, Macrophage are connected with Chemokine, Secretion, Cytokine and Cell and other disciplines. His Pulmonary fibrosis research includes themes of Pathogenesis, Bioinformatics, Disease, Pulmonary function testing and Mitochondrial biogenesis.
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Reactive oxygen species in cell signaling
Victor J. Thannickal;Barry L. Fanburg.
American Journal of Physiology-lung Cellular and Molecular Physiology (2000)
The Myofibroblast: One Function, Multiple Origins
Boris Hinz;Sem H. Phan;Victor J. Thannickal;Andrea Galli.
American Journal of Pathology (2007)
Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines.
R De Caterina;Peter Libby;H B Peng;V J Thannickal.
Journal of Clinical Investigation (1995)
Recent developments in myofibroblast biology: paradigms for connective tissue remodeling.
Boris Hinz;Sem H. Phan;Victor J. Thannickal;Marco Prunotto.
American Journal of Pathology (2012)
miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis
Gang Liu;Arnaud Friggeri;Yanping Yang;Jadranka Milosevic.
Journal of Experimental Medicine (2010)
NADPH oxidase-4 mediates myofibroblast activation and fibrogenic responses to lung injury.
Louise Hecker;Ragini Vittal;Tamara Jones;Rajesh Jagirdar.
Nature Medicine (2009)
Mechanisms of pulmonary fibrosis.
Victor J. Thannickal;Galen B. Toews;Eric S. White;Joseph P. Lynch.
Annual Review of Medicine (2004)
Myofibroblast Differentiation by Transforming Growth Factor-β1 Is Dependent on Cell Adhesion and Integrin Signaling via Focal Adhesion Kinase
Victor J. Thannickal;Daniel Y. Lee;Eric S. White;Zongbin Cui.
Journal of Biological Chemistry (2003)
Activation of an H2O2-generating NADH Oxidase in Human Lung Fibroblasts by Transforming Growth Factor β1
Victor J. Thannickal;Barry L. Fanburg.
Journal of Biological Chemistry (1995)
Host Responses in Tissue Repair and Fibrosis
Jeremy S. Duffield;Mark L Lupher;Victor J. Thannickal;Thomas A. Wynn.
Annual Review of Pathology-mechanisms of Disease (2013)
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