D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 90 Citations 34,027 305 World Ranking 7574 National Ranking 4086
Biology and Biochemistry D-index 91 Citations 34,283 308 World Ranking 1479 National Ranking 858

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Cancer

Myofibroblast, Fibrosis, Cell biology, Idiopathic pulmonary fibrosis and Lung are his primary areas of study. Victor J. Thannickal combines subjects such as Lung injury and Cellular differentiation with his study of Myofibroblast. His Fibrosis research includes themes of Fibrocyte, Cancer research, CCR2 and Collagen, type I, alpha 1.

His Cell biology research incorporates themes from Cell adhesion and Cytokine. His Idiopathic pulmonary fibrosis research incorporates elements of Oxidative stress, Pulmonary fibrosis and Immunology, Pathogenesis. His Lung study incorporates themes from Chemokine receptor and Bone marrow, Pathology.

His most cited work include:

  • Reactive oxygen species in cell signaling (2164 citations)
  • Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines. (1551 citations)
  • The Myofibroblast: One Function, Multiple Origins (1485 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Idiopathic pulmonary fibrosis, Cell biology, Lung, Fibrosis and Pathology. His Idiopathic pulmonary fibrosis research is multidisciplinary, incorporating elements of Pulmonary fibrosis, Lung injury and Immunology, Pathogenesis. His Cell biology research is multidisciplinary, incorporating perspectives in Myofibroblast and Gene knockdown.

The study incorporates disciplines such as Wound healing, Fibroblast, Cellular differentiation and NOX4 in addition to Myofibroblast. His research in Fibrosis intersects with topics in Oxidative stress, Cancer research and Senescence. His studies deal with areas such as Extracellular matrix and Usual interstitial pneumonia as well as Pathology.

He most often published in these fields:

  • Idiopathic pulmonary fibrosis (35.44%)
  • Cell biology (26.90%)
  • Lung (25.00%)

What were the highlights of his more recent work (between 2018-2021)?

  • Idiopathic pulmonary fibrosis (35.44%)
  • Cancer research (24.05%)
  • Fibrosis (26.27%)

In recent papers he was focusing on the following fields of study:

Victor J. Thannickal mainly focuses on Idiopathic pulmonary fibrosis, Cancer research, Fibrosis, Lung and Cell biology. The concepts of his Idiopathic pulmonary fibrosis study are interwoven with issues in Lung injury, Pathogenesis, Bioinformatics and Cellular senescence. His Cancer research research is multidisciplinary, incorporating elements of Cell, Ex vivo, Myofibroblast, Senescence and Fibroblast.

His Pulmonary fibrosis study in the realm of Fibrosis connects with subjects such as Bleomycin. His Lung research incorporates themes from Genetics, Epigenetics and Immunology. As a member of one scientific family, Victor J. Thannickal mostly works in the field of Cell biology, focusing on Gene silencing and, on occasion, MALAT1, Gene knockdown and Stromal cell.

Between 2018 and 2021, his most popular works were:

  • Developmental pathways in the pathogenesis of lung fibrosis. (57 citations)
  • SIRT3 diminishes inflammation and mitigates endotoxin-induced acute lung injury. (37 citations)
  • Long noncoding RNA Malat1 regulates differential activation of macrophages and response to lung injury. (32 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Enzyme

His primary areas of study are Idiopathic pulmonary fibrosis, Fibrosis, Cell biology, Lung injury and Pulmonary fibrosis. The study incorporates disciplines such as Cancer research, Proteostasis and Pathology in addition to Idiopathic pulmonary fibrosis. His work investigates the relationship between Cancer research and topics such as Senescence that intersect with problems in MyoD, Myofibroblast and Proinflammatory cytokine.

His study in Fibrosis is interdisciplinary in nature, drawing from both Process, Computational biology and Organ function. His work is dedicated to discovering how Cell biology, Macrophage are connected with Chemokine, Secretion, Cytokine and Cell and other disciplines. His Pulmonary fibrosis research includes themes of Pathogenesis, Bioinformatics, Disease, Pulmonary function testing and Mitochondrial biogenesis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Reactive oxygen species in cell signaling

Victor J. Thannickal;Barry L. Fanburg.
American Journal of Physiology-lung Cellular and Molecular Physiology (2000)

3648 Citations

The Myofibroblast: One Function, Multiple Origins

Boris Hinz;Sem H. Phan;Victor J. Thannickal;Andrea Galli.
American Journal of Pathology (2007)

2298 Citations

Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines.

R De Caterina;Peter Libby;H B Peng;V J Thannickal.
Journal of Clinical Investigation (1995)

2224 Citations

Recent developments in myofibroblast biology: paradigms for connective tissue remodeling.

Boris Hinz;Sem H. Phan;Victor J. Thannickal;Marco Prunotto.
American Journal of Pathology (2012)

1204 Citations

miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis

Gang Liu;Arnaud Friggeri;Yanping Yang;Jadranka Milosevic.
Journal of Experimental Medicine (2010)

961 Citations

NADPH oxidase-4 mediates myofibroblast activation and fibrogenic responses to lung injury.

Louise Hecker;Ragini Vittal;Tamara Jones;Rajesh Jagirdar.
Nature Medicine (2009)

793 Citations

Mechanisms of pulmonary fibrosis.

Victor J. Thannickal;Galen B. Toews;Eric S. White;Joseph P. Lynch.
Annual Review of Medicine (2004)

760 Citations

Myofibroblast Differentiation by Transforming Growth Factor-β1 Is Dependent on Cell Adhesion and Integrin Signaling via Focal Adhesion Kinase

Victor J. Thannickal;Daniel Y. Lee;Eric S. White;Zongbin Cui.
Journal of Biological Chemistry (2003)

655 Citations

Activation of an H2O2-generating NADH Oxidase in Human Lung Fibroblasts by Transforming Growth Factor β1

Victor J. Thannickal;Barry L. Fanburg.
Journal of Biological Chemistry (1995)

531 Citations

Host Responses in Tissue Repair and Fibrosis

Jeremy S. Duffield;Mark L Lupher;Victor J. Thannickal;Thomas A. Wynn.
Annual Review of Pathology-mechanisms of Disease (2013)

527 Citations

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