D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 102 Citations 28,693 297 World Ranking 387 National Ranking 235
Medicine D-index 105 Citations 30,686 348 World Ranking 4061 National Ranking 2306

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Internal medicine

His primary areas of investigation include Immunology, Cytokine, Lung, Chemokine and Tumor necrosis factor alpha. The Immunology study combines topics in areas such as Bronchoalveolar lavage and In vivo. Theodore J. Standiford combines subjects such as Alveolar macrophage, Legionella pneumophila and Monocyte with his study of Cytokine.

His studies deal with areas such as Fibrosis, Pathology, Fibroblast and Immunosuppression as well as Lung. Theodore J. Standiford has researched Chemokine in several fields, including Aspergillus fumigatus, Innate immune system and Human disease. His Tumor necrosis factor alpha study integrates concerns from other disciplines, such as Lipopolysaccharide and Macrophage.

His most cited work include:

  • Interleukin-8 gene expression by a pulmonary epithelial cell line. A model for cytokine networks in the lung. (598 citations)
  • Interleukin-8 (IL-8): The Major Neutrophil Chemotactic Factor in the Lung (482 citations)
  • Balance of inflammatory cytokines related to severity and mortality of murine sepsis. (327 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Immunology, Cytokine, Lung, Inflammation and Chemokine. His works in Immune system, Tumor necrosis factor alpha, Innate immune system, Proinflammatory cytokine and Sepsis are all subjects of inquiry into Immunology. His Tumor necrosis factor alpha research integrates issues from Molecular biology, Cell biology and Alveolar macrophage.

His Cytokine study incorporates themes from Lipopolysaccharide and Macrophage. His Lung study combines topics from a wide range of disciplines, such as Pathology, In vivo and Pneumonia. Theodore J. Standiford works mostly in the field of Inflammation, limiting it down to topics relating to Monocyte and, in certain cases, Chemotaxis, as a part of the same area of interest.

He most often published in these fields:

  • Immunology (63.29%)
  • Cytokine (26.01%)
  • Lung (21.97%)

What were the highlights of his more recent work (between 2014-2021)?

  • Immunology (63.29%)
  • Sepsis (13.29%)
  • Inflammation (20.81%)

In recent papers he was focusing on the following fields of study:

Immunology, Sepsis, Inflammation, Microbiology and Lung injury are his primary areas of study. His biological study spans a wide range of topics, including Lung and Pneumonia. His study in Lung is interdisciplinary in nature, drawing from both Fibrosis and Epithelium.

His Sepsis research incorporates themes from Bioinformatics, Transcriptome, Immune system, Neuroinflammation and Human brain. His studies in Microbiology integrate themes in fields like Legionella pneumophila, Interferon, Matricellular protein, Pseudomonas aeruginosa and Proinflammatory cytokine. His Lung injury research includes elements of Psychological intervention, Cancer research, In vitro, Interleukin and Complement receptor.

Between 2014 and 2021, his most popular works were:

  • Enrichment of the lung microbiome with gut bacteria in sepsis and the acute respiratory distress syndrome. (176 citations)
  • Therapeutic targeting of acute lung injury and acute respiratory distress syndrome (94 citations)
  • Neutrophil transfer of miR-223 to lung epithelial cells dampens acute lung injury in mice (82 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

Theodore J. Standiford mostly deals with Immunology, Sepsis, Lung, Immune system and Inflammation. His Cytokine, Chemokine and Virus study in the realm of Immunology connects with subjects such as Trichostatin A. His work carried out in the field of Sepsis brings together such families of science as Precision medicine, Systems biology, Bioinformatics and Proteomics.

As a part of the same scientific study, Theodore J. Standiford usually deals with the Lung, concentrating on Pneumonia and frequently concerns with Respiratory disease, Respiratory system and Interleukin. The concepts of his Immune system study are interwoven with issues in Lung cancer and Primary tumor. The various areas that Theodore J. Standiford examines in his Inflammation study include mir-223, Epithelium, In vitro and Gene expression.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Interleukin-8 gene expression by a pulmonary epithelial cell line. A model for cytokine networks in the lung.

T J Standiford;S L Kunkel;M A Basha;S W Chensue.
Journal of Clinical Investigation (1990)

810 Citations

Interleukin-8 (IL-8): The Major Neutrophil Chemotactic Factor in the Lung

Steven L. Kunkel;Theodore Standiford;Keita Kasahara;Robert M. Strieter.
Experimental Lung Research (1991)

678 Citations

Balance of inflammatory cytokines related to severity and mortality of murine sepsis.

Keith R. Walley;Nicholas W. Lukacs;Theodore J. Standiford;Robert M. Strieter.
Infection and Immunity (1996)

493 Citations

Pre-B-cell colony-enhancing factor as a potential novel biomarker in acute lung injury.

Shui Q. Ye;Brett A. Simon;James P. Maloney;April Zambelli-Weiner.
American Journal of Respiratory and Critical Care Medicine (2005)

480 Citations

The Pseudomonas aeruginosa autoinducer N-3-oxododecanoyl homoserine lactone accelerates apoptosis in macrophages and neutrophils.

Kazuhiro Tateda;Yoshikazu Ishii;Manabu Horikawa;Tetsuya Matsumoto.
Infection and Immunity (2003)

477 Citations

Alveolar macrophage-derived cytokines induce monocyte chemoattractant protein-1 expression from human pulmonary type II-like epithelial cells.

T J Standiford;S L Kunkel;S H Phan;B J Rollins.
Journal of Biological Chemistry (1991)

473 Citations

Neutralization of IL-10 increases survival in a murine model of Klebsiella pneumonia

M. J. Greenberger;R. M. Strieter;S. L. Kunkel;J. M. Danforth.
Journal of Immunology (1995)

464 Citations

MCP-1 protects mice in lethal endotoxemia.

David A. Zisman;Steven L. Kunkel;Robert M. Strieter;Wan C. Tsai.
Journal of Clinical Investigation (1997)

388 Citations

Neutralization of IL-10 increases lethality in endotoxemia. Cooperative effects of macrophage inflammatory protein-2 and tumor necrosis factor.

T J Standiford;R M Strieter;N W Lukacs;S L Kunkel.
Journal of Immunology (1995)

367 Citations

Neutralization of Macrophage Inflammatory Protein-2 Attenuates Neutrophil Recruitment and Bacterial Clearance in Murine Klebsiella Pneumonia

Marc J. Greenberger;Robert M. Strieter;Steven L. Kunkel;Jean M. Danforth.
The Journal of Infectious Diseases (1996)

362 Citations

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