D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 83 Citations 28,971 144 World Ranking 561 National Ranking 336
Medicine D-index 92 Citations 34,087 216 World Ranking 5453 National Ranking 3053

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Internal medicine

Robert J. Homer mainly focuses on Immunology, Inflammation, Interleukin 13, Lung and Cytokine. The study incorporates disciplines such as Fibrosis and Pulmonary fibrosis in addition to Immunology. His Pulmonary fibrosis study combines topics from a wide range of disciplines, such as CXCL10 and Eotaxin.

His Inflammation research includes elements of Eosinophil, Transgene, Eosinophilia, Matrix metalloproteinase and Macrophage. His Interleukin 13 research is multidisciplinary, incorporating perspectives in Effector and Chitinase. Robert J. Homer works mostly in the field of Lung, limiting it down to topics relating to Pathology and, in certain cases, CD8, Antibody and Pulmonary edema, as a part of the same area of interest.

His most cited work include:

  • Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production (1541 citations)
  • Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production (1541 citations)
  • Regulation of lung injury and repair by Toll-like receptors and hyaluronan. (1125 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Immunology, Inflammation, Pathology, Lung and Cancer research. The Immunology study which covers Fibrosis that intersects with Transforming growth factor beta. His biological study spans a wide range of topics, including Receptor, Matrix metalloproteinase, Transgene and Cell biology.

His research in Pathology intersects with topics in Respiratory disease and Interstitial lung disease. His Lung study deals with Airway intersecting with Asthma. In his study, Adenocarcinoma is inextricably linked to Lung cancer, which falls within the broad field of Cancer research.

He most often published in these fields:

  • Immunology (59.70%)
  • Inflammation (36.19%)
  • Pathology (30.97%)

What were the highlights of his more recent work (between 2015-2021)?

  • Pathology (30.97%)
  • Cancer research (19.78%)
  • Lung (27.24%)

In recent papers he was focusing on the following fields of study:

Robert J. Homer focuses on Pathology, Cancer research, Lung, Lung cancer and Idiopathic pulmonary fibrosis. His work deals with themes such as Internal medicine and Antibody, which intersect with Pathology. The concepts of his Cancer research study are interwoven with issues in Epidermal growth factor receptor, Adrenergic, Osimertinib and Transcription factor.

His Lung research incorporates themes from Inflammation, Immunology, Anatomy & histology and Parenchyma. His Acetylcholine receptor research extends to Immunology, which is thematically connected. His studies in Idiopathic pulmonary fibrosis integrate themes in fields like Fibrosis, Pulmonary fibrosis, Myofibroblast and Fibroblast.

Between 2015 and 2021, his most popular works were:

  • A prospective, multi-institutional, pathologist-based assessment of 4 immunohistochemistry assays for PD-L1 expression in non–small cell lung cancer (415 citations)
  • Mx1 reveals innate pathways to antiviral resistance and lethal influenza disease (128 citations)
  • Thyroid hormone inhibits lung fibrosis in mice by improving epithelial mitochondrial function (112 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Internal medicine

Idiopathic pulmonary fibrosis, Pathology, Lung, Cancer research and Virus are his primary areas of study. The Idiopathic pulmonary fibrosis study combines topics in areas such as Pulmonary fibrosis and Interstitial lung disease. While the research belongs to areas of Pulmonary fibrosis, he spends his time largely on the problem of In vivo, intersecting his research to questions surrounding Matrix metalloproteinase, Inflammation, CD68 and Interleukin 13.

Many of his research projects under Pathology are closely connected to Intraclass correlation with Intraclass correlation, tying the diverse disciplines of science together. The various areas that Robert J. Homer examines in his Cancer research study include Cancer, Plexin, DNA methylation, Transcription factor and Fibrosis. His Mucociliary clearance study frequently draws connections to adjacent fields such as Immunology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production

Zhou Zhu;Robert J. Homer;Robert J. Homer;Zhonde Wang;Qingsheng Chen.
Journal of Clinical Investigation (1999)

2482 Citations

Regulation of lung injury and repair by Toll-like receptors and hyaluronan.

Dianhua Jiang;Jiurong Liang;Juan Fan;Shuang Yu.
Nature Medicine (2005)

1360 Citations

Interleukin-13 Induces Tissue Fibrosis by Selectively Stimulating and Activating Transforming Growth Factor β1

Chun Geun Lee;Robert J. Homer;Zhou Zhu;Sophie Lanone.
Journal of Experimental Medicine (2001)

1001 Citations

Acidic Mammalian Chitinase in Asthmatic Th2 Inflammation and IL-13 Pathway Activation

Zhou Zhu;Tao Zheng;Robert J. Homer;Yoon Keun Kim.
Science (2004)

890 Citations

Inducible targeting of IL-13 to the adult lung causes matrix metalloproteinase– and cathepsin-dependent emphysema

Tao Zheng;Zhou Zhu;Zhongde Wang;Robert J. Homer;Robert J. Homer.
Journal of Clinical Investigation (2000)

882 Citations

Airway remodeling in asthma.

Jack A. Elias;Zhou Zhu;Geoffrey Chupp;Robert J. Homer.
Journal of Clinical Investigation (1999)

829 Citations

Vascular endothelial growth factor (VEGF) induces remodeling and enhances TH2-mediated sensitization and inflammation in the lung.

Chun Geun Lee;Holger Link;Peter Baluk;Robert J Homer.
Nature Medicine (2004)

747 Citations

A SNP in a let-7 microRNA Complementary Site in the KRAS 3′ Untranslated Region Increases Non–Small Cell Lung Cancer Risk

Lena J. Chin;Elena Ratner;Shuguang Leng;Rihong Zhai.
Cancer Research (2008)

718 Citations

Induction of Airway Mucus Production By T Helper 2 (Th2) Cells: A Critical Role For Interleukin 4 In Cell Recruitment But Not Mucus Production

Lauren Cohn;Robert J. Homer;Anthony Marinov;John Rankin.
Journal of Experimental Medicine (1997)

674 Citations

New insights into the pathogenesis of asthma

Jack A. Elias;Chun Geun Lee;Tao Zheng;Bing Ma.
Journal of Clinical Investigation (2003)

585 Citations

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Profile was last updated on December 6th, 2021.
Research.com Ranking is based on data retrieved from the Microsoft Academic Graph (MAG).
The ranking d-index is inferred from publications deemed to belong to the considered discipline.

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