D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 72 Citations 22,336 148 World Ranking 981 National Ranking 529
Medicine D-index 74 Citations 23,648 173 World Ranking 12886 National Ranking 6802

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Internal medicine
  • Immune system

Her primary areas of study are Immunology, Cytokine, Interleukin 13, Immune system and Asthma. Her study in Immunology concentrates on Interleukin, Antigen, Allergy, Allergic response and Immunoglobulin E. Her Cytokine study integrates concerns from other disciplines, such as Peripheral blood mononuclear cell, Regulation of gene expression, Case-control study and Incubation.

Marsha Wills-Karp has included themes like Interleukin 23, Interleukin 17, Complement C5a and Complement receptor in her Interleukin 13 study. Her work on Inactivated vaccine as part of her general Immune system study is frequently connected to Viral pneumonia, thereby bridging the divide between different branches of science. Her study in Asthma is interdisciplinary in nature, drawing from both Mediator and Tralokinumab.

Her most cited work include:

  • Interleukin-13: Central Mediator of Allergic Asthma (2434 citations)
  • IMMUNOLOGIC BASIS OF ANTIGEN-INDUCED AIRWAY HYPERRESPONSIVENESS (963 citations)
  • The germless theory of allergic disease: revisiting the hygiene hypothesis (723 citations)

What are the main themes of her work throughout her whole career to date?

Marsha Wills-Karp focuses on Immunology, Asthma, Immune system, Cytokine and Interleukin 13. Her Immunology study frequently intersects with other fields, such as Lung. The various areas that Marsha Wills-Karp examines in her Asthma study include Respiratory disease, Disease and Genetics, Gene.

Marsha Wills-Karp has researched Interleukin 13 in several fields, including STAT6, House dust mite, Receptor and Signal transduction, Cell biology. Her Inflammation research is multidisciplinary, incorporating elements of Eosinophilia and Pathogenesis. Her biological study spans a wide range of topics, including Allergic response and Interleukin.

She most often published in these fields:

  • Immunology (72.58%)
  • Asthma (25.00%)
  • Immune system (17.74%)

What were the highlights of her more recent work (between 2012-2021)?

  • Immunology (72.58%)
  • Inflammation (16.94%)
  • Immune system (17.74%)

In recent papers she was focusing on the following fields of study:

Marsha Wills-Karp mostly deals with Immunology, Inflammation, Immune system, Asthma and Interleukin 13. Her Immunology study combines topics from a wide range of disciplines, such as Signal transduction, Disease and Lung. Marsha Wills-Karp interconnects Endocrinology, Cytokine and Lymphocyte in the investigation of issues within Inflammation.

Her Immune system research is multidisciplinary, relying on both Microbiome, Expression quantitative trait loci, Tropomyosin and Pharmacology. Her Asthma research integrates issues from Leukocytosis and Bioinformatics. Her Interleukin 13 research includes themes of STAT6, Blockade and Cell biology.

Between 2012 and 2021, her most popular works were:

  • IL-4 and IL-13 signaling in allergic airway disease. (197 citations)
  • Bone Marrow Dendritic Cells from Mice with an Altered Microbiota Provide Interleukin 17A-Dependent Protection against Entamoeba histolytica Colitis (75 citations)
  • Intrauterine Inflammation and Maternal Exposure to Ambient PM2.5 during Preconception and Specific Periods of Pregnancy: The Boston Birth Cohort. (63 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Immune system

The scientist’s investigation covers issues in Immunology, Interleukin 13, Signal transduction, Immune system and Inflammation. Her Immunology study integrates concerns from other disciplines, such as Blockade and Interleukin 12. Her work in Interleukin 12 covers topics such as Cytokine which are related to areas like Asthma.

While the research belongs to areas of Interleukin 13, she spends her time largely on the problem of STAT6, intersecting her research to questions surrounding Gene expression, Cell biology, Cell culture and Interleukin 17. Her work deals with themes such as Interleukin, Regulation of gene expression, Cancer research and Keratin, which intersect with Signal transduction. Her study in Immune system is interdisciplinary in nature, drawing from both Microbiome, Human microbiome, Gut flora and Entamoeba histolytica.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Interleukin-13: Central Mediator of Allergic Asthma

M. Wills-Karp;J. Luyimbazi;X. Xu;B. Schofield.
Science (1998)

3118 Citations

IMMUNOLOGIC BASIS OF ANTIGEN-INDUCED AIRWAY HYPERRESPONSIVENESS

Marsha Wills-Karp.
Annual Review of Immunology (1999)

1268 Citations

The germless theory of allergic disease: revisiting the hygiene hypothesis

Marsha Wills-Karp;Joanna Santeliz;Christopher L. Karp.
Nature Reviews Immunology (2001)

1018 Citations

Interleukin-13 in asthma pathogenesis

Marsha Wills-Karp.
Immunological Reviews (2004)

872 Citations

Allergenicity resulting from functional mimicry of a Toll-like receptor complex protein

Aurelien Trompette;Senad Divanovic;Alberto Visintin;Carine Blanchard.
Nature (2009)

758 Citations

Interleukin 12 inhibits antigen-induced airway hyperresponsiveness, inflammation, and Th2 cytokine expression in mice.

Stephen H. Gavett;Daniel J. O’Hearn;Xiumin Li;Shau Ku Huang.
Journal of Experimental Medicine (1995)

712 Citations

Depletion of murine CD4+ T lymphocytes prevents antigen-induced airway hyperreactivity and pulmonary eosinophilia.

S. H. Gavett;Xiaoling Chen;F. Finkelman;M. Wills-Karp.
American Journal of Respiratory Cell and Molecular Biology (1994)

586 Citations

Signal Transducer and Activator of Transcription Factor 6 (Stat6)-deficient Mice Are Protected from Antigen-induced Airway Hyperresponsiveness and Mucus Production

Douglas Kuperman;Brian Schofield;Marsha Wills-Karp;Michael J. Grusby.
Journal of Experimental Medicine (1998)

531 Citations

CD4+CD25+ T cells protect against experimentally induced asthma and alter pulmonary dendritic cell phenotype and function

Ian P. Lewkowich;Nancy S. Herman;Kathleen W. Schleifer;Matthew P. Dance.
Journal of Experimental Medicine (2005)

484 Citations

Identification of complement factor 5 as a susceptibility locus for experimental allergic asthma.

Christopher L. Karp;Andrew Grupe;Eric Schadt;Susan L. Ewart.
Nature Immunology (2000)

455 Citations

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