What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Cytokine
Jack A. Elias mainly investigates Immunology, Inflammation, Fibrosis, Interleukin 13 and Lung.
Jack A. Elias regularly ties together related areas like Pulmonary fibrosis in his Immunology studies.
His studies deal with areas such as Signal transduction, Cell biology, Matrix metalloproteinase, Macrophage and Programmed cell death as well as Inflammation.
His Fibrosis research focuses on Cancer research and how it relates to Transforming growth factor, Transforming growth factor beta and Receptor.
Jack A. Elias has researched Interleukin 13 in several fields, including Vascular endothelial growth factor A, Molecular biology and Effector.
His Lung research is multidisciplinary, incorporating perspectives in Endocrinology and Pathology.
His most cited work include:
- Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production (1541 citations)
- IL-17 is increased in asthmatic airways and induces human bronchial fibroblasts to produce cytokines. (806 citations)
- Proceedings of the ATS Workshop on Refractory Asthma Current Understanding, Recommendations, and Unanswered Questions (771 citations)
What are the main themes of his work throughout his whole career to date?
Immunology, Inflammation, Cancer research, Lung and Interleukin 13 are his primary areas of study.
In his research on the topic of Immunology, Macrophage is strongly related with Fibrosis.
In his study, which falls under the umbrella issue of Inflammation, Hyperoxia and Lung injury is strongly linked to Programmed cell death.
His Cancer research research is multidisciplinary, incorporating elements of Transforming growth factor and Metastasis.
His research investigates the link between Lung and topics such as Pathology that cross with problems in Respiratory disease.
As part of the same scientific family, Jack A. Elias usually focuses on Cytokine, concentrating on Fibroblast and intersecting with Endocrinology, Internal medicine and Molecular biology.
He most often published in these fields:
- Immunology (50.53%)
- Inflammation (27.93%)
- Cancer research (17.82%)
What were the highlights of his more recent work (between 2014-2021)?
- Immunology (50.53%)
- CHI3L1 (12.77%)
- Cancer research (17.82%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Immunology, CHI3L1, Cancer research, Lung and Inflammation.
His Immunology study incorporates themes from Fibrosis, COPD and Chitinase.
His CHI3L1 research includes themes of Receptor, Cytotoxic T cell, Antibody and T cell.
His research in Receptor tackles topics such as Interleukin 13 which are related to areas like Asthma.
The study incorporates disciplines such as Retinoic acid receptor beta, Plexin, Immunity and Pathology in addition to Lung.
Jack A. Elias has researched Inflammation in several fields, including M2 Macrophage and Ovalbumin.
Between 2014 and 2021, his most popular works were:
- Chitin recognition via chitotriosidase promotes pathologic type-2 helper T cell responses to cryptococcal infection (131 citations)
- Epithelial Cell Mitochondrial Dysfunction and PINK1 Are Induced by Transforming Growth Factor- Beta1 in Pulmonary Fibrosis (97 citations)
- The regulation of lung fibroblast proliferation by alveolar macrophages in experimental silicosis. (61 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
His main research concerns Immunology, Inflammation, CHI3L1, Lung and Fibrosis.
He has included themes like COPD, Downregulation and upregulation and Chitinase in his Immunology study.
His Inflammation study combines topics from a wide range of disciplines, such as T cell, Innate immune system, Endocrinology and White adipose tissue.
His CHI3L1 study combines topics in areas such as Receptor and Myofibroblast.
Jack A. Elias interconnects Cancer research and Pathology in the investigation of issues within Lung.
His studies in Pulmonary fibrosis integrate themes in fields like In vivo and Cell biology.
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