D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 122 Citations 47,042 348 World Ranking 1434 National Ranking 874

Research.com Recognitions

Awards & Achievements

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Cancer

His scientific interests lie mostly in Lung, Pulmonary hypertension, Pathology, Immunology and Respiratory disease. The concepts of his Lung study are interwoven with issues in Inflammation, Pharmacology and Pathogenesis. His Pulmonary hypertension study is concerned with the larger field of Internal medicine.

His research in Pathology intersects with topics in Endothelial stem cell, Neovascularization, Vascular endothelial growth factor, Vascular disease and Prostacyclin. Rubin M. Tuder has researched Immunology in several fields, including Telomere, Oxidative stress, Bronchoalveolar lavage and Telomerase. His Oxidative stress research is multidisciplinary, incorporating perspectives in Apoptosis and COPD.

His most cited work include:

  • Inhibition of VEGF receptors causes lung cell apoptosis and emphysema (980 citations)
  • Genetic ablation of Nrf2 enhances susceptibility to cigarette smoke–induced emphysema in mice (791 citations)
  • Prostacyclin Synthase Expression Is Decreased in Lungs from Patients with Severe Pulmonary Hypertension (664 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Pathology, Pulmonary hypertension, Lung, Immunology and Internal medicine. His Pathology study also includes fields such as

  • Intensive care most often made with reference to Respiratory disease,
  • Endothelial stem cell which intersects with area such as Vascular endothelial growth factor A. His Pulmonary hypertension research integrates issues from Pulmonary pathology, Hypoxia, Ventricle, Pulmonary artery and Endothelium.

His work deals with themes such as Cancer research, Disease, Pathogenesis and COPD, which intersect with Lung. His Immunology research includes themes of Oxidative stress, Apoptosis and Alveolar cells. His research in Internal medicine intersects with topics in Gastroenterology, Endocrinology and Cardiology.

He most often published in these fields:

  • Pathology (40.09%)
  • Pulmonary hypertension (39.40%)
  • Lung (40.09%)

What were the highlights of his more recent work (between 2015-2021)?

  • Pulmonary hypertension (39.40%)
  • Pathology (40.09%)
  • Lung (40.09%)

In recent papers he was focusing on the following fields of study:

Rubin M. Tuder mostly deals with Pulmonary hypertension, Pathology, Lung, Internal medicine and Inflammation. His work carried out in the field of Pulmonary hypertension brings together such families of science as TGF beta signaling pathway, Transforming growth factor, Hypoxia and Pulmonary artery. His studies in Pathology integrate themes in fields like Lung injury, Crosstalk, Hypoxia-inducible factors and Period.

His Lung study combines topics in areas such as Cancer research, Disease, Pathogenesis and Intensive care medicine. Rubin M. Tuder has researched Internal medicine in several fields, including Histopathology, Endocrinology, Human genetics and Cardiology. His Inflammation study results in a more complete grasp of Immunology.

Between 2015 and 2021, his most popular works were:

  • Assessment of Right Ventricular Function in the Research Setting: Knowledge Gaps and Pathways Forward. An Official American Thoracic Society Research Statement (79 citations)
  • Pulmonary vascular remodeling in pulmonary hypertension. (70 citations)
  • HIPPO–Integrin-linked Kinase Cross-Talk Controls Self-Sustaining Proliferation and Survival in Pulmonary Hypertension (57 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Cancer

His primary scientific interests are in Pulmonary hypertension, Pathology, Lung, Internal medicine and Lung injury. The study incorporates disciplines such as Pulmonary embolism, Ventricular function and Hypoxia in addition to Pulmonary hypertension. Rubin M. Tuder combines subjects such as Transdifferentiation, Cell Transdifferentiation, Green fluorescent protein, Alveolar type and In vivo with his study of Pathology.

His biological study spans a wide range of topics, including Syndecan 1, Glycocalyx, Intensive care medicine, Disease and Transforming growth factor. His Internal medicine research includes elements of Endocrinology, Surgery and Cardiology. His Lung injury study incorporates themes from Heparan sulfate, Barrier function, Alveolar Epithelium and Vascular permeability.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Inhibition of VEGF receptors causes lung cell apoptosis and emphysema

Yasunori Kasahara;Rubin M. Tuder;Laimute Taraseviciene-Stewart;Timothy D. Le Cras.
Journal of Clinical Investigation (2000)

1268 Citations

Genetic ablation of Nrf2 enhances susceptibility to cigarette smoke–induced emphysema in mice

Tirumalai Rangasamy;Chung Y. Cho;Rajesh K. Thimmulappa;Lijie Zhen.
Journal of Clinical Investigation (2004)

1030 Citations

Prostacyclin Synthase Expression Is Decreased in Lungs from Patients with Severe Pulmonary Hypertension

Rubin M. Tuder;Carlyne D. Cool;Mark W. Geraci;Jun Wang.
American Journal of Respiratory and Critical Care Medicine (1999)

972 Citations

Exuberant endothelial cell growth and elements of inflammation are present in plexiform lesions of pulmonary hypertension

R. M. Tuder;B. Groves;D. B. Badesch;N. F. Voelkel.
American Journal of Pathology (1994)

925 Citations

Endothelial Dysfunction in Pulmonary Hypertension

Rohit Budhiraja;Rubin M. Tuder;Paul M. Hassoun.
Circulation (2004)

913 Citations

Inhibition of the VEGF receptor 2 combined with chronic hypoxia causes cell death-dependent pulmonary endothelial cell proliferation and severe pulmonary hypertension

Laimute Taraseviciene-Stewart;Yasunori Kasahara;Lori Alger;Peter Hirth.
The FASEB Journal (2001)

813 Citations

Pathologic assessment of vasculopathies in pulmonary hypertension.

Giuseppe G. Pietra;Frederique Capron;Susan Stewart;Ornella Leone.
Journal of the American College of Cardiology (2004)

798 Citations

Endothelial cell death and decreased expression of vascular endothelial growth factor and vascular endothelial growth factor receptor 2 in emphysema.

Yasunori Kasahara;Rubin M. Tuder;Carlyne D. Cool;David A. Lynch.
American Journal of Respiratory and Critical Care Medicine (2001)

793 Citations

Increased gene expression for VEGF and the VEGF receptors KDR/Flk and Flt in lungs exposed to acute or to chronic hypoxia. Modulation of gene expression by nitric oxide.

R M Tuder;B E Flook;N F Voelkel.
Journal of Clinical Investigation (1995)

709 Citations

Short telomeres are a risk factor for idiopathic pulmonary fibrosis

Jonathan K. Alder;Julian J.L. Chen;Lisa Lancaster;Sonye Danoff.
Proceedings of the National Academy of Sciences of the United States of America (2008)

703 Citations

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