Member of the Association of American Physicians
His scientific interests lie mostly in Lung, Pulmonary hypertension, Pathology, Immunology and Respiratory disease. The concepts of his Lung study are interwoven with issues in Inflammation, Pharmacology and Pathogenesis. His Pulmonary hypertension study is concerned with the larger field of Internal medicine.
His research in Pathology intersects with topics in Endothelial stem cell, Neovascularization, Vascular endothelial growth factor, Vascular disease and Prostacyclin. Rubin M. Tuder has researched Immunology in several fields, including Telomere, Oxidative stress, Bronchoalveolar lavage and Telomerase. His Oxidative stress research is multidisciplinary, incorporating perspectives in Apoptosis and COPD.
The scientist’s investigation covers issues in Pathology, Pulmonary hypertension, Lung, Immunology and Internal medicine. His Pathology study also includes fields such as
His work deals with themes such as Cancer research, Disease, Pathogenesis and COPD, which intersect with Lung. His Immunology research includes themes of Oxidative stress, Apoptosis and Alveolar cells. His research in Internal medicine intersects with topics in Gastroenterology, Endocrinology and Cardiology.
Rubin M. Tuder mostly deals with Pulmonary hypertension, Pathology, Lung, Internal medicine and Inflammation. His work carried out in the field of Pulmonary hypertension brings together such families of science as TGF beta signaling pathway, Transforming growth factor, Hypoxia and Pulmonary artery. His studies in Pathology integrate themes in fields like Lung injury, Crosstalk, Hypoxia-inducible factors and Period.
His Lung study combines topics in areas such as Cancer research, Disease, Pathogenesis and Intensive care medicine. Rubin M. Tuder has researched Internal medicine in several fields, including Histopathology, Endocrinology, Human genetics and Cardiology. His Inflammation study results in a more complete grasp of Immunology.
His primary scientific interests are in Pulmonary hypertension, Pathology, Lung, Internal medicine and Lung injury. The study incorporates disciplines such as Pulmonary embolism, Ventricular function and Hypoxia in addition to Pulmonary hypertension. Rubin M. Tuder combines subjects such as Transdifferentiation, Cell Transdifferentiation, Green fluorescent protein, Alveolar type and In vivo with his study of Pathology.
His biological study spans a wide range of topics, including Syndecan 1, Glycocalyx, Intensive care medicine, Disease and Transforming growth factor. His Internal medicine research includes elements of Endocrinology, Surgery and Cardiology. His Lung injury study incorporates themes from Heparan sulfate, Barrier function, Alveolar Epithelium and Vascular permeability.
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Inhibition of VEGF receptors causes lung cell apoptosis and emphysema
Yasunori Kasahara;Rubin M. Tuder;Laimute Taraseviciene-Stewart;Timothy D. Le Cras.
Journal of Clinical Investigation (2000)
Genetic ablation of Nrf2 enhances susceptibility to cigarette smoke–induced emphysema in mice
Tirumalai Rangasamy;Chung Y. Cho;Rajesh K. Thimmulappa;Lijie Zhen.
Journal of Clinical Investigation (2004)
Prostacyclin Synthase Expression Is Decreased in Lungs from Patients with Severe Pulmonary Hypertension
Rubin M. Tuder;Carlyne D. Cool;Mark W. Geraci;Jun Wang.
American Journal of Respiratory and Critical Care Medicine (1999)
Exuberant endothelial cell growth and elements of inflammation are present in plexiform lesions of pulmonary hypertension
R. M. Tuder;B. Groves;D. B. Badesch;N. F. Voelkel.
American Journal of Pathology (1994)
Endothelial Dysfunction in Pulmonary Hypertension
Rohit Budhiraja;Rubin M. Tuder;Paul M. Hassoun.
Inhibition of the VEGF receptor 2 combined with chronic hypoxia causes cell death-dependent pulmonary endothelial cell proliferation and severe pulmonary hypertension
Laimute Taraseviciene-Stewart;Yasunori Kasahara;Lori Alger;Peter Hirth.
The FASEB Journal (2001)
Pathologic assessment of vasculopathies in pulmonary hypertension.
Giuseppe G. Pietra;Frederique Capron;Susan Stewart;Ornella Leone.
Journal of the American College of Cardiology (2004)
Endothelial cell death and decreased expression of vascular endothelial growth factor and vascular endothelial growth factor receptor 2 in emphysema.
Yasunori Kasahara;Rubin M. Tuder;Carlyne D. Cool;David A. Lynch.
American Journal of Respiratory and Critical Care Medicine (2001)
Increased gene expression for VEGF and the VEGF receptors KDR/Flk and Flt in lungs exposed to acute or to chronic hypoxia. Modulation of gene expression by nitric oxide.
R M Tuder;B E Flook;N F Voelkel.
Journal of Clinical Investigation (1995)
Short telomeres are a risk factor for idiopathic pulmonary fibrosis
Jonathan K. Alder;Julian J.L. Chen;Lisa Lancaster;Sonye Danoff.
Proceedings of the National Academy of Sciences of the United States of America (2008)
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