What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Enzyme
The scientist’s investigation covers issues in Immunology, Asthma, Interleukin 13, Inflammation and Pathogenesis.
His study in Pulmonary fibrosis extends to Immunology with its themes.
His Asthma research incorporates elements of Respiratory tract infections, Mice transgenic, Disease and Atopic dermatitis.
The study incorporates disciplines such as Fibrosis and Chemokine in addition to Interleukin 13.
His study on Inflammation also encompasses disciplines like
- Lung which is related to area like Pathology,
- Matrix metalloproteinase that intertwine with fields like Macrophage.
His work deals with themes such as NFKB1, Interleukin 6, In vitro and Receptor, which intersect with Pathogenesis.
His most cited work include:
- A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17 (3489 citations)
- Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production (1541 citations)
- Direct effects of interleukin-13 on epithelial cells cause airway hyperreactivity and mucus overproduction in asthma (754 citations)
What are the main themes of his work throughout his whole career to date?
Immunology, Inflammation, Interleukin 13, Asthma and Immune system are his primary areas of study.
As part of his studies on Immunology, Zhou Zhu often connects relevant subjects like Lung.
His Inflammation study combines topics in areas such as Receptor, Ovalbumin and Signal transduction, Cell biology.
His Interleukin 13 research incorporates themes from Fibrosis and Eotaxin.
His research integrates issues of COPD, Disease, Pathogenesis and Allergy in his study of Asthma.
His studies deal with areas such as Lipopolysaccharide, Internal medicine, Endocrinology and Allergen as well as Immune system.
He most often published in these fields:
- Immunology (66.44%)
- Inflammation (31.51%)
- Interleukin 13 (23.29%)
What were the highlights of his more recent work (between 2012-2019)?
- Immunology (66.44%)
- Atopic dermatitis (13.01%)
- Cell biology (16.44%)
In recent papers he was focusing on the following fields of study:
Zhou Zhu mainly focuses on Immunology, Atopic dermatitis, Cell biology, Immune system and Inflammation.
His research on Immunology often connects related topics like Signal transduction.
His study in Cell biology is interdisciplinary in nature, drawing from both Mast cell, Intrinsic apoptosis, Programmed cell death and SIGLEC.
His study in the fields of Allergen immunotherapy under the domain of Immune system overlaps with other disciplines such as Dietary Reference Intake.
His Inflammation research incorporates elements of Eosinophil, Bronchoalveolar lavage, Antibody and Immunotherapy.
His Interleukin 13 research is multidisciplinary, incorporating elements of Allergic sensitization and NADPH oxidase.
Between 2012 and 2019, his most popular works were:
- The Atopic March: Progression from Atopic Dermatitis to Allergic Rhinitis and Asthma. (173 citations)
- TRPA1-dependent pruritus in IL-13-induced chronic atopic dermatitis. (103 citations)
- TRPA1-dependent pruritus in IL-13-induced chronic atopic dermatitis. (103 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Enzyme
Zhou Zhu focuses on Immunology, Signal transduction, Atopic dermatitis, Immune system and Neuropeptide.
His research is interdisciplinary, bridging the disciplines of Sialyltransferase and Immunology.
His Signal transduction research is multidisciplinary, incorporating perspectives in Thymic stromal lymphopoietin, Downregulation and upregulation, Interleukin 22 and Keratinocyte.
His research on Atopic dermatitis often connects related areas such as Asthma.
Zhou Zhu interconnects Autoimmune disease, Cytokine and Arthritis in the investigation of issues within Immune system.
His Neuropeptide study combines topics from a wide range of disciplines, such as Interleukin 13, Transient receptor potential channel and Epidermis.
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