What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Immune system
The scientist’s investigation covers issues in Immunology, Asthma, Rhinovirus, Exacerbation and Respiratory disease.
Immunology is often connected to COPD in his work.
The Asthma study combines topics in areas such as Viral disease, Atypical bacteria, Disease and Allergy.
His Rhinovirus research integrates issues from Eosinophil, Common cold, Interferon and Interleukin 8.
His Exacerbation study also includes
- Etiology most often made with reference to Biomarker,
- Lung microbiome together with Case-control study.
His biological study spans a wide range of topics, including Wheeze, Respiratory tract, Respiratory system and Pediatrics.
His most cited work include:
- Allergic rhinitis and its impact on asthma (ARIA) 2008 update (in collaboration with the World Health Organization, GA(2)LEN and AllerGen) (3245 citations)
- Community study of role of viral infections in exacerbations of asthma in 9-11 year old children. (1668 citations)
- Asthmatic bronchial epithelial cells have a deficient innate immune response to infection with rhinovirus (954 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Immunology, Rhinovirus, Asthma, COPD and Respiratory system.
His research in Immunology tackles topics such as Respiratory disease which are related to areas like Viral disease.
His Rhinovirus study deals with Interferon intersecting with Innate immune system.
In his study, Pediatrics is inextricably linked to Respiratory tract infections, which falls within the broad field of Asthma.
His research in COPD intersects with topics in Pulmonary disease, Respiratory virus, Lung and Airway.
His Respiratory system research is mostly focused on the topic Respiratory tract.
He most often published in these fields:
- Immunology (76.23%)
- Rhinovirus (46.45%)
- Asthma (42.44%)
What were the highlights of his more recent work (between 2016-2021)?
- Immunology (76.23%)
- Asthma (42.44%)
- Rhinovirus (46.45%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Immunology, Asthma, Rhinovirus, Exacerbation and COPD.
His Immunology research includes themes of Lung and Respiratory system.
His work carried out in the field of Asthma brings together such families of science as Microbiome, Allergic sensitization, Allergy and Intensive care medicine.
In his study, which falls under the umbrella issue of Rhinovirus, Viral load is strongly linked to Bronchoalveolar lavage.
Sebastian L. Johnston has researched Exacerbation in several fields, including Interleukin 13 and Pathogenesis.
His studies deal with areas such as Pulmonary disease, Eosinophil, Gastroenterology and Etiology as well as COPD.
Between 2016 and 2021, his most popular works were:
- Host DNA released by NETosis promotes rhinovirus-induced type-2 allergic asthma exacerbation (128 citations)
- Viral infections in allergy and immunology: How allergic inflammation influences viral infections and illness (76 citations)
- Corticosteroid suppression of antiviral immunity increases bacterial loads and mucus production in COPD exacerbations. (72 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Immune system
Sebastian L. Johnston mostly deals with Immunology, Asthma, Rhinovirus, Allergy and COPD.
His Immunology research integrates issues from Lung and Respiratory system.
His Asthma research includes elements of Microbiome, Respiratory tract infections, Exacerbation, Randomized controlled trial and Allergic sensitization.
His Exacerbation research is multidisciplinary, incorporating elements of Proinflammatory cytokine, Interleukin 13 and Plasmacytoid dendritic cell.
The various areas that he examines in his Rhinovirus study include Interferon, Platelet-activating factor receptor, Immune system and Microbiology.
As part of one scientific family, Sebastian L. Johnston deals mainly with the area of Allergy, narrowing it down to issues related to the Toll-like receptor, and often Type I interferon production, Polyinosinic:polycytidylic acid, Pattern recognition receptor and Allergic inflammation.
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