Michael R. Edwards focuses on Immunology, Rhinovirus, Interferon, Asthma and Cytokine. His work in Immune system, Chemokine, Virus, Viral replication and NF-κB are all subfields of Immunology research. The various areas that he examines in his Rhinovirus study include Inflammation, Exacerbation, TLR3, Bronchoalveolar lavage and Interleukin 13.
The Interferon study combines topics in areas such as Peripheral blood mononuclear cell, Real-time polymerase chain reaction, Respiratory epithelium and Pattern recognition receptor. His Asthma research focuses on subjects like Pathogenesis, which are linked to Epidemiology, Respiratory pathogens and Respiratory system. His study in Cytokine is interdisciplinary in nature, drawing from both Innate lymphoid cell and Azithromycin.
Michael R. Edwards mainly focuses on Immunology, Rhinovirus, Asthma, Interferon and Biochemistry. Michael R. Edwards combines topics linked to Respiratory system with his work on Immunology. Michael R. Edwards usually deals with Rhinovirus and limits it to topics linked to COPD and Pneumonia.
His Asthma research incorporates elements of Disease, Pathogenesis and Allergy. His Interferon study incorporates themes from TLR3, Cystic fibrosis and Immunity. His Biochemistry study deals with Molecular biology intersecting with Gene.
His primary areas of study are Immunology, Rhinovirus, Asthma, Interferon and Respiratory system. His studies link Pulmonary disease with Immunology. As a part of the same scientific study, Michael R. Edwards usually deals with the Rhinovirus, concentrating on COPD and frequently concerns with Streptococcus pneumoniae and Bacteria.
His biological study spans a wide range of topics, including Exacerbation, Lung and Allergy. His Interferon study integrates concerns from other disciplines, such as Translation, Downregulation and upregulation, Mediator and Frameshift mutation. Michael R. Edwards works mostly in the field of Respiratory system, limiting it down to topics relating to Innate immune system and, in certain cases, Viral load, as a part of the same area of interest.
Michael R. Edwards mainly focuses on Immunology, Rhinovirus, Asthma, Allergy and Interferon. His work in Immunology addresses issues such as COPD, which are connected to fields such as Pneumonia and Immunity. His work in Rhinovirus tackles topics such as Innate immune system which are related to areas like Phagocytosis, Bronchoalveolar lavage and Viral load.
His Asthma study combines topics from a wide range of disciplines, such as Microbiome and Respiratory system. His work deals with themes such as Exacerbation, Randomized controlled trial, Asthma exacerbations and Allergic bronchopulmonary aspergillosis, which intersect with Allergy. Michael R. Edwards combines subjects such as Chemokine, CXCL10 and Immune system with his study of Interferon.
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Role of deficient type III interferon-λ production in asthma exacerbations
Marco Contoli;Simon D Message;Vasile Laza-Stanca;Michael R Edwards.
Nature Medicine (2006)
The Airway Epithelium: Soldier in the Fight against Respiratory Viruses
Marjolaine Vareille;Elisabeth Kieninger;Michael R. Edwards;Michael R. Edwards;Nicolas Regamey.
Clinical Microbiology Reviews (2011)
IL-33–Dependent Type 2 Inflammation during Rhinovirus-induced Asthma Exacerbations In Vivo
David J. Jackson;David J. Jackson;Heidi Makrinioti;Batika M. J. Rana;Betty W. H. Shamji.
American Journal of Respiratory and Critical Care Medicine (2014)
Targeting the NF-kappaB pathway in asthma and chronic obstructive pulmonary disease.
Michael R. Edwards;Nathan W. Bartlett;Deborah Clarke;Mark Birrell.
Pharmacology & Therapeutics (2009)
Mouse models of rhinovirus-induced disease and exacerbation of allergic airway inflammation
Nathan W Bartlett;Ross P Walton;Michael R Edwards;Juliya Aniscenko.
Nature Medicine (2008)
Experimental Rhinovirus Infection as a Human Model of Chronic Obstructive Pulmonary Disease Exacerbation
Patrick Mallia;Simon D. Message;Vera Gielen;Marco Contoli.
American Journal of Respiratory and Critical Care Medicine (2011)
Loss of ACTN3 gene function alters mouse muscle metabolism and shows evidence of positive selection in humans.
Daniel MacArthur;Jane T Seto;Jane T Seto;Joanna M Raftery;Kate G Quinlan;Kate G Quinlan.
Nature Genetics (2007)
Azithromycin induces anti-viral responses in bronchial epithelial cells
V. Gielen;Sebastian L. Johnston;Michael R. Edwards.
European Respiratory Journal (2010)
Toll-Like Receptor 3 Is Induced by and Mediates Antiviral Activity against Rhinovirus Infection of Human Bronchial Epithelial Cells
Christopher A. Hewson;Alice Jardine;Michael R. Edwards;Vasile Laza-Stanca.
Journal of Virology (2005)
Co-ordinated Role of TLR3, RIG-I and MDA5 in the Innate Response to Rhinovirus in Bronchial Epithelium
Louise Slater;Nathan Bartlett;Jennifer J. Haas;Jie Zhu.
PLOS Pathogens (2010)
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