His primary areas of investigation include Immunology, Asthma, Immune system, Allergy and Antigen. Dendritic cell, T cell, Eosinophil, Allergen and Antigen-presenting cell are the subjects of his Immunology studies. His Asthma research is multidisciplinary, incorporating perspectives in Microbiome, Clinical trial, Eosinophilic and Microbiology.
His studies in Immune system integrate themes in fields like Ex vivo, Peripheral blood mononuclear cell and Cytokine. He focuses mostly in the field of Peripheral blood mononuclear cell, narrowing it down to matters related to T lymphocyte and, in some cases, Immunoglobulin E. His work focuses on many connections between Antigen and other disciplines, such as Immunity, that overlap with his field of interest in Respiratory tract, Homeostasis and Respiratory system.
His primary areas of study are Immunology, Asthma, Immune system, Internal medicine and Inflammation. As part of his studies on Immunology, John W. Upham frequently links adjacent subjects like Peripheral blood mononuclear cell. His work deals with themes such as Exacerbation, Azithromycin and Intensive care medicine, which intersect with Asthma.
His study ties his expertise on Antigen together with the subject of Immune system. His Internal medicine study frequently links to related topics such as Endocrinology. As a part of the same scientific study, John W. Upham usually deals with the Inflammation, concentrating on Viral load and frequently concerns with Interferon.
The scientist’s investigation covers issues in Asthma, Immunology, Severe asthma, Internal medicine and Inflammation. John W. Upham has researched Asthma in several fields, including Exacerbation, Severity of illness and Azithromycin. Immunology is closely attributed to Peripheral blood mononuclear cell in his work.
His Severe asthma study integrates concerns from other disciplines, such as Quality of life, Pediatrics, Emergency medicine and Intensive care medicine. His Inflammation study also includes
Asthma, Immunology, Internal medicine, Severity of illness and Inflammation are his primary areas of study. John W. Upham combines subjects such as Exacerbation, Quality of life and Intensive care medicine with his study of Asthma. His Immunology research includes elements of Moraxella catarrhalis, Peripheral blood mononuclear cell and Antibiotic resistance.
John W. Upham interconnects Retrospective cohort study, Influenza A virus, Pediatrics and Vaccination in the investigation of issues within Severity of illness. The Inflammation study combines topics in areas such as Bronchiolitis, Bronchoalveolar lavage, Viral load and Pathogenesis. His biological study spans a wide range of topics, including Interleukin 33, IL1RL1, Rhinovirus and Cytokine.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Effect of azithromycin on asthma exacerbations and quality of life in adults with persistent uncontrolled asthma (AMAZES): a randomised, double-blind, placebo-controlled trial
Peter G Gibson;Peter G Gibson;Ian A Yang;John W Upham;John W Upham;Paul N Reynolds;Paul N Reynolds.
The Lancet (2017)
Rapid dendritic cell recruitment to the bronchial mucosa of patients with atopic asthma in response to local allergen challenge
F.L. Jahnsen;E.D. Moloney;T. Hogan;J.W. Upham.
Contemporaneous maturation of immunologic and respiratory functions during early childhood: implications for development of asthma prevention strategies.
Patrick G. Holt;John W. Upham;Peter D. Sly.
The Journal of Allergy and Clinical Immunology (2005)
Development of interleukin-12-producing capacity throughout childhood.
John W. Upham;Peter T. Lee;Barbara J. Holt;Tricia Heaton.
Infection and Immunity (2002)
Functional Maturation of CD4+CD25+CTLA4+CD45RA+ T Regulatory Cells in Human Neonatal T Cell Responses to Environmental Antigens/Allergens
Catherine A. Thornton;John W. Upham;Matthew E. Wikström;Barbara J. Holt.
Journal of Immunology (2004)
TLR4 polymorphisms mediate impaired responses to respiratory syncytial virus and lipopolysaccharide.
Meri K. Tulic;Robert J. Hurrelbrink;Cecilia M. Prêle;Ingrid A. Laing.
Journal of Immunology (2007)
Full blood count parameters for the detection of asthma inflammatory phenotypes
X.Y. Zhang;J.L. Simpson;H. Powell;I.A. Yang.
Clinical & Experimental Allergy (2014)
Postnatal development of monocyte cytokine responses to bacterial lipopolysaccharide.
Stephanie T Yerkovich;Matthew E Wikström;Devinda Suriyaarachchi;Susan L Prescott.
Pediatric Research (2007)
Inhalant allergen‐specific T‐cell reactivity is detectable in close to 100% of atopic and normal individuals: covert responses are unmasked by serum‐free medium
J. W. Upham;J. W. Upham;B. J. Holt;M. J. Baron-Hay;A. Yabuhara.
Clinical & Experimental Allergy (1995)
T‐cell “priming” against environmental allergens in human neonates: sequential deletion of food antigen reactivity during infancy with concomitant expansion of responses to ubiquitous inhalant allergens
P.G. Holt;P. O'Keeffe;B.J. Holt;J.W. Upham.
Pediatric Allergy and Immunology (1995)
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