Immunology, COPD, Bronchoalveolar lavage, Asthma and Alveolar macrophage are her primary areas of study. Her Immunology research is multidisciplinary, incorporating elements of Efferocytosis, Macrophage and Cytotoxic T cell. She has researched Bronchoalveolar lavage in several fields, including Apoptosis and Pathology.
The Asthma study combines topics in areas such as Microbiome, Pediatrics, Eosinophilic and Microbiology. Her work investigates the relationship between Alveolar macrophage and topics such as CD36 that intersect with problems in Transferrin receptor and Ex vivo. Her study explores the link between Inflammation and topics such as Phagocytosis that cross with problems in Necrosis and Azithromycin.
Sandra Hodge focuses on Immunology, Inflammation, Cytotoxic T cell, Tumor necrosis factor alpha and Apoptosis. Sandra Hodge combines topics linked to COPD with her work on Immunology. Her research integrates issues of Pathogenesis, Immune system, Cell biology, Bronchoalveolar lavage and Azithromycin in her study of Inflammation.
Her Tumor necrosis factor alpha study incorporates themes from CD28, Flow cytometry and Lymphocyte. Her studies deal with areas such as Efferocytosis, Cancer, Necrosis and Phagocytosis as well as Apoptosis. The concepts of her Phagocytosis study are interwoven with issues in Macrophage and Alveolar macrophage.
Her primary areas of investigation include Immunology, Inflammation, Asthma, Azithromycin and Tumor necrosis factor alpha. Her Immunology research is multidisciplinary, incorporating perspectives in Bronchoalveolar lavage, Lung and Bronchiolitis obliterans. Her work deals with themes such as Macrophage, Innate immune system, Flow cytometry and Lymphocyte, which intersect with Inflammation.
Her Asthma research is multidisciplinary, relying on both Exacerbation and Prospective cohort study. Her study focuses on the intersection of Azithromycin and fields such as Haemophilus influenzae with connections in the field of Xenophagy. Her Tumor necrosis factor alpha study combines topics from a wide range of disciplines, such as Proinflammatory cytokine and CD8.
Sandra Hodge spends much of her time researching Immunology, Inflammation, Asthma, Eosinophil and Azithromycin. Sandra Hodge has included themes like Bronchiolitis obliterans and Bronchiectasis in her Immunology study. Her Inflammation research includes themes of Acquired immune system, Natural killer T cell, Natural killer cell, Histone deacetylase 2 and Bronchoalveolar lavage.
She combines subjects such as Streptococcus, Gemella, Quality of life and Eosinophilic with her study of Asthma. The study incorporates disciplines such as Biomarker, Exacerbation and Microbiome in addition to Eosinophil. Her Azithromycin study integrates concerns from other disciplines, such as Moraxella catarrhalis, Streptococcus pneumoniae, Haemophilus influenzae and Antibiotic resistance.
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Alveolar macrophages from subjects with chronic obstructive pulmonary disease are deficient in their ability to phagocytose apoptotic airway epithelial cells
Sandra Hodge;Sandra Hodge;Greg Hodge;Greg Hodge;Raffaele Scicchitano;Raffaele Scicchitano;Paul N Reynolds;Paul N Reynolds.
Immunology and Cell Biology (2003)
Smoking alters alveolar macrophage recognition and phagocytic ability: implications in chronic obstructive pulmonary disease.
Sandra Hodge;Greg Hodge;Jessica Ahern;Hubertus Jersmann.
American Journal of Respiratory Cell and Molecular Biology (2007)
Effect of azithromycin on asthma exacerbations and quality of life in adults with persistent uncontrolled asthma (AMAZES): a randomised, double-blind, placebo-controlled trial
Peter G Gibson;Peter G Gibson;Ian A Yang;John W Upham;John W Upham;Paul N Reynolds;Paul N Reynolds.
The Lancet (2017)
Azithromycin Improves Macrophage Phagocytic Function and Expression of Mannose Receptor in Chronic Obstructive Pulmonary Disease
Sandra Hodge;Greg Hodge;Hubertus Jersmann;Geoffrey Matthews.
American Journal of Respiratory and Critical Care Medicine (2008)
Increased airway epithelial and T-cell apoptosis in COPD remains despite smoking cessation
S Hodge;G Hodge;M Holmes;P N Reynolds.
European Respiratory Journal (2005)
Azithromycin increases phagocytosis of apoptotic bronchial epithelial cells by alveolar macrophages
S Hodge;G Hodge;S Brozyna;H Jersmann.
European Respiratory Journal (2006)
Full blood count parameters for the detection of asthma inflammatory phenotypes
X.Y. Zhang;J.L. Simpson;H. Powell;I.A. Yang.
Clinical & Experimental Allergy (2014)
Increased intracellular T helper 1 proinflammatory cytokine production in peripheral blood, bronchoalveolar lavage and intraepithelial T cells of COPD subjects
G Hodge;J Nairn;M Holmes;P N Reynolds.
Clinical and Experimental Immunology (2007)
Cigarette Smoke-Induced Changes to Alveolar Macrophage Phenotype and Function Are Improved by Treatment with Procysteine
Sandra Hodge;Geoffrey Matthews;Violet Mukaro;Jessica Ahern.
American Journal of Respiratory Cell and Molecular Biology (2011)
Increased production of TGF-β and apoptosis of T lymphocytes isolated from peripheral blood in COPD
S. J. Hodge;G. L. Hodge;P. N. Reynolds;R. Scicchitano.
American Journal of Physiology-lung Cellular and Molecular Physiology (2003)
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