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Rosemarie H. DeKruyff

Rosemarie H. DeKruyff

D-Index & Metrics

Immunology

D-Index
84
Citations
28311
World Ranking
1357
National Ranking
698

Medicine

D-Index
84
Citations
28301
World Ranking
15013
National Ranking
7592

Overview

Rosemarie H. DeKruyff is affiliated with Stanford University in the United States and has contributed extensively to the field of Medicine, with a focus on Immunology and Allergy. Their research includes multiple subfields such as Physiology, Epidemiology, and Dermatology. The main topics covered in their work consist of Food Allergy and Anaphylaxis Research, Asthma and respiratory diseases, Allergic Rhinitis and Sensitization, Respiratory viral infections research, IL-33, ST2, and ILC Pathways, Immune Cell Function and Interaction, as well as Contact Dermatitis and Allergies.

Among their recent publications are:

  • Summary of the Keystone Symposium "Origins of allergic disease: Microbial, epithelial and immune interactions," March 24-27, Tahoe City, California, 2020, Journal of Allergy and Clinical Immunology
  • Epigenetics and the Environment in Airway Disease: Asthma and Allergic Rhinitis, 2020, Advances in experimental medicine and biology
  • Immune changes beyond Th2 pathways during rapid multifood immunotherapy enabled with omalizumab, 2021, Allergy
  • CD8+ T cell differentiation status correlates with the feasibility of sustained unresponsiveness following oral immunotherapy, 2022, Nature Communications
  • Impact of air pollution exposure on cytokines and histone modification profiles at single-cell levels during pregnancy, 2024, Science Advances

Frequent co-authors working with DeKruyff include Kari C. Nadeau, R. Sharon Chinthrajah, Monali Manohar, Stephen J. Galli, and Diane Dunham.

Their publications are often found in prominent journals such as:

  • Journal of Allergy and Clinical Immunology
  • Allergy
  • Frontiers in Immunology
  • Advances in experimental medicine and biology
  • Nature Communications

Best Publications

  • Pulmonary dendritic cells producing IL-10 mediate tolerance induced by respiratory exposure to antigen.

    Omid Akbari;Rosemarie H. DeKruyff;Dale T. Umetsu

  • Antigen-specific regulatory T cells develop via the ICOS–ICOS-ligand pathway and inhibit allergen-induced airway hyperreactivity

    Omid Akbari;Gordon J. Freeman;Everett H. Meyer;Edward A. Greenfield

  • Innate lymphoid cells mediate influenza-induced airway hyper-reactivity independently of adaptive immunity

    Ya Jen Chang;Hye Young Kim;Lee A. Albacker;Nicole Baumgarth

  • Essential role of NKT cells producing IL-4 and IL-13 in the development of allergen-induced airway hyperreactivity

    Omid Akbari;Philippe Stock;Everett Meyer;Mitchell Kronenberg

  • Asthma: an epidemic of dysregulated immunity.

    Dale T. Umetsu;Jennifer J. McIntire;Omid Akbari;Claudia Macaubas

  • Allergen-specific Th1 cells fail to counterbalance Th2 cell–induced airway hyperreactivity but cause severe airway inflammation

    Gesine Hansen;Gerald Berry;Rosemarie H. DeKruyff;Dale T. Umetsu

  • TIM-1 and TIM-4 glycoproteins bind phosphatidylserine and mediate uptake of apoptotic cells.

    Norimoto Kobayashi;Piia Karisola;Victor Peña-Cruz;David M. Dorfman

  • TIM genes: a family of cell surface phosphatidylserine receptors that regulate innate and adaptive immunity

    Gordon J. Freeman;Jose M. Casasnovas;Dale T. Umetsu;Rosemarie H. DeKruyff

  • The many paths to asthma: phenotype shaped by innate and adaptive immunity

    Hye Young Kim;Rosemarie H DeKruyff;Dale T Umetsu

  • Identification of Tapr (an airway hyperreactivity regulatory locus) and the linked Tim gene family

    Jennifer J. McIntire;Sarah E. Umetsu;Omid Akbari;Michael Potter

  • Interleukin-17–producing innate lymphoid cells and the NLRP3 inflammasome facilitate obesity-associated airway hyperreactivity

    Hye Young Kim;Hyun Jun Lee;Ya Jen Chang;Muriel Pichavant

  • CD4+ invariant T-cell-receptor+ natural killer T cells in bronchial asthma.

    Omid Akbari;John L. Faul;Elisabeth G. Hoyte;Gerald J. Berry

  • The TIM gene family: emerging roles in immunity and disease

    Vijay K. Kuchroo;Dale T. Umetsu;Rosemarie H. DeKruyff;Gordon J. Freeman

  • Critical role for IL-13 in the development of allergen-induced airway hyperreactivity.

    David M. Walter;Jennifer J. McIntire;Gerald Berry;Andrew N. J. McKenzie

  • TIM-4 is the ligand for TIM-1, and the TIM-1–TIM-4 interaction regulates T cell proliferation

    Jennifer Hartt Meyers;Sumone Chakravarti;David Schlesinger;Zsolt Illes

  • TIM-1 induces T cell activation and inhibits the development of peripheral tolerance

    Sarah E Umetsu;Wan-Ling Lee;Jennifer J McIntire;Laura Downey

  • Corticosteroids Inhibit Il-12 Production In Human Monocytes And Enhance Their Capacity To Induce Il-4 Synthesis In Cd4+ Lymphocytes

    M H Blotta;R H DeKruyff;D T Umetsu

  • Induction of T helper type 1–like regulatory cells that express Foxp3 and protect against airway hyper-reactivity

    Philippe Stock;Omid Akbari;Omid Akbari;Gerald Berry;Gordon J Freeman

  • TIM-family Proteins Promote Infection of Multiple Enveloped Viruses through Virion-associated Phosphatidylserine

    Stephanie Jemielity;Jinyize J. Wang;Ying Kai Chan;Asim Aminsharif Ahmed

  • T Cell/Transmembrane, Ig, and Mucin-3 Allelic Variants Differentially Recognize Phosphatidylserine and Mediate Phagocytosis of Apoptotic Cells

    Rosemarie H. DeKruyff;Xia Bu;Angela Ballesteros;César Santiago

  • Th1 and Th2 CD4+ cells in human allergic diseases

    Dale T. Umetsu;Rosemarie H. DeKruyff

Frequent Co-Authors

Dale T. Umetsu
Dale T. Umetsu Stanford University
Gordon J. Freeman
Gordon J. Freeman Harvard University
Omid Akbari
Omid Akbari University of Southern California
Harvey Cantor
Harvey Cantor Harvard University
Paul B. Savage
Paul B. Savage Brigham Young University
Carol Clayberger
Carol Clayberger Northwestern University
Gerald J. Berry
Gerald J. Berry Stanford University
Vijay K. Kuchroo
Vijay K. Kuchroo Harvard University
Shoshana Levy
Shoshana Levy Stanford University
Arlene H. Sharpe
Arlene H. Sharpe Harvard University

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