His primary scientific interests are in Natural killer T cell, CD1D, Biochemistry, Immunology and Ceragenin. His work deals with themes such as Natural killer cell, Receptor and Antigen-presenting cell, which intersect with Natural killer T cell. His CD1D research is multidisciplinary, incorporating perspectives in CD1, Glycolipid, Antigen presentation and Cell biology.
Paul B. Savage has included themes like Gram-negative bacteria and Cholic acid in his Ceragenin study. His research in Bacteria focuses on subjects like Antibiotics, which are connected to Cationic polymerization. His Antigen study combines topics in areas such as Innate immune system and Antibody.
Paul B. Savage mainly focuses on Antimicrobial, Microbiology, Ceragenin, Natural killer T cell and Immunology. His study looks at the relationship between Antimicrobial and fields such as Bacteria, as well as how they intersect with chemical problems. His biological study spans a wide range of topics, including Pseudomonas aeruginosa and Biofilm.
His Ceragenin research also works with subjects such as
Paul B. Savage mainly investigates Antimicrobial, Microbiology, Ceragenin, Antimicrobial peptides and Biofilm. His study in Antimicrobial is interdisciplinary in nature, drawing from both Antibacterial activity, Pharmacology, Acinetobacter baumannii and Staphylococcus aureus. Paul B. Savage works mostly in the field of Antibacterial activity, limiting it down to concerns involving Endotracheal tube and, occasionally, Immunology.
His Microbiology research includes elements of Pseudomonas aeruginosa and Bacteria. His Ceragenin research incorporates elements of Cancer cell, Apoptosis, Bone Infection, Intensive care medicine and Bacillus subtilis. His study on Cancer cell also encompasses disciplines like
The scientist’s investigation covers issues in Microbiology, Antimicrobial, Ceragenin, Antimicrobial peptides and Biofilm. His Microbiology study frequently draws connections to adjacent fields such as Bacteria. His studies in Ceragenin integrate themes in fields like Ex vivo, Cancer cell, Reactive oxygen species, Pseudomonas aeruginosa and DNA fragmentation.
His research integrates issues of Cancer research, Carcinogenesis, Apoptosis, Colistin and Pharmacology in his study of Antimicrobial peptides. The Biofilm study combines topics in areas such as Cystic fibrosis and Lung. His Innate immune system study is concerned with Immunology in general.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The biology of NKT cells.
Albert Bendelac;Paul B. Savage;Luc Teyton.
Annual Review of Immunology (2007)
Exogenous and endogenous glycolipid antigens activate NKT cells during microbial infections
Jochen Mattner;Kristin L. DeBord;Nahed Ismail;Randal D. Goff.
Nature (2005)
Lysosomal Glycosphingolipid Recognition by NKT Cells
Dapeng Zhou;Jochen Mattner;Carlos Cantu;Nicolas Schrantz.
Science (2004)
Identification of an IL-17–producing NK1.1neg iNKT cell population involved in airway neutrophilia
Marie-Laure Michel;Alexandre Castro Keller;Christophe Paget;Christophe Paget;Masakazu Fujio.
Journal of Experimental Medicine (2007)
Bacterial lipid composition and the antimicrobial efficacy of cationic steroid compounds (Ceragenins).
Raquel F. Epand;Paul B. Savage;Richard M. Epand.
Biochimica et Biophysica Acta (2007)
Editing of CD1d-bound lipid antigens by endosomal lipid transfer proteins.
Dapeng Zhou;Carlos Cantu;Yuval Sagiv;Nicolas Schrantz.
Science (2004)
Structure and function of a potent agonist for the semi-invariant natural killer T cell receptor.
Dirk M Zajonc;Carlos Cantu;Jochen Mattner;Dapeng Zhou.
Nature Immunology (2005)
An Effective Fluorescent Chemosensor for Mercury Ions
Luca Prodi;Carlotta Bargossi;Marco Montalti;Nelsi Zaccheroni.
Journal of the American Chemical Society (2000)
Ozone exposure in a mouse model induces airway hyperreactivity that requires the presence of natural killer T cells and IL-17
Muriel Pichavant;Sho Goya;Everett H Meyer;Everett H Meyer;Richard A. Johnston.
Journal of Experimental Medicine (2008)
Innate lymphoid cells responding to IL-33 mediate airway hyperreactivity independently of adaptive immunity
Hye Young Kim;Ya Jen Chang;Srividya Subramanian;Hyun Hee Lee.
The Journal of Allergy and Clinical Immunology (2012)
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