D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 44 Citations 6,858 152 World Ranking 3691 National Ranking 143

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Cytokine
  • Cancer

Akihiro Matsukawa focuses on Immunology, Chemokine, Cytokine, Inflammation and Tumor necrosis factor alpha. The Immunology study combines topics in areas such as Bronchoalveolar lavage, Peritonitis and Receptor antagonist. As a part of the same scientific family, Akihiro Matsukawa mostly works in the field of Receptor antagonist, focusing on Arthritis and, on occasion, Endocrinology, Lipopolysaccharide and Neutrophil elastase.

Akihiro Matsukawa has researched Chemokine in several fields, including Peritoneum and Pathology. His Cytokine study frequently draws connections between adjacent fields such as Monoclonal antibody. His study looks at the intersection of Inflammation and topics like STAT3 with Adoptive cell transfer.

His most cited work include:

  • Galectin-9 suppresses the generation of Th17, promotes the induction of regulatory T cells, and regulates experimental autoimmune arthritis (336 citations)
  • Innate immune response in Th1- and Th2-dominant mouse strains. (284 citations)
  • Endogenous monocyte chemoattractant protein-1 (MCP-1) protects mice in a model of acute septic peritonitis: cross-talk between MCP-1 and leukotriene B4. (218 citations)

What are the main themes of his work throughout his whole career to date?

Akihiro Matsukawa mainly focuses on Immunology, Cytokine, Inflammation, Chemokine and Tumor necrosis factor alpha. His Immunology research is multidisciplinary, relying on both Molecular biology and Peritonitis. He combines subjects such as Arthritis, Virus, Influenza A virus, Monoclonal antibody and Receptor antagonist with his study of Cytokine.

The concepts of his Inflammation study are interwoven with issues in Cancer research, Sepsis and MAPK/ERK pathway. As part of one scientific family, Akihiro Matsukawa deals mainly with the area of Chemokine, narrowing it down to issues related to the Pathology, and often Lung. His research in Tumor necrosis factor alpha intersects with topics in Lipopolysaccharide and Interleukin 8.

He most often published in these fields:

  • Immunology (43.23%)
  • Cytokine (24.48%)
  • Inflammation (23.96%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cancer research (15.10%)
  • Cancer (4.17%)
  • Lung (9.37%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Cancer research, Cancer, Lung, Pathology and Immunohistochemistry. The study incorporates disciplines such as Carcinogenesis, Cancer stem cell, Carcinoma and Osteoblast in addition to Cancer research. His work deals with themes such as Allograft rejection, Tracheal transplantation, Early phase and Th1 response, which intersect with Pathology.

His study looks at the relationship between Lung transplantation and topics such as MAPK/ERK pathway, which overlap with Inflammation, Internal medicine, Cytotoxic T cell and Interferon. He works mostly in the field of Interleukin, limiting it down to concerns involving Lipopolysaccharide and, occasionally, Tumor necrosis factor alpha. His Tumor necrosis factor alpha study integrates concerns from other disciplines, such as Necrosis and Molecular biology.

Between 2017 and 2021, his most popular works were:

  • miRNA-133a-5p Inhibits the Expression of Osteoblast Differentiation-Associated Markers by Targeting the 3' UTR of RUNX2. (18 citations)
  • Collagen-Binding Hepatocyte Growth Factor (HGF) alone or with a Gelatin- furfurylamine Hydrogel Enhances Functional Recovery in Mice after Spinal Cord Injury. (17 citations)
  • Combined effect of anti-high-mobility group box-1 monoclonal antibody and peramivir against influenza A virus-induced pneumonia in mice. (11 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Cancer
  • Cytokine

His primary areas of study are Cancer research, Cancer, Internal medicine, Inflammation and Endocrinology. His Cancer research research incorporates themes from RUNX2 and Osteoblast. In the subject of general Cancer, his work in Targeted therapy, Combination cancer therapy and Cancer stem cell is often linked to Homeobox protein NANOG, thereby combining diverse domains of study.

His work on Tumor necrosis factor alpha, Adipose tissue, White adipose tissue and Stromal vascular fraction as part of general Internal medicine research is frequently linked to Metabolic syndrome, thereby connecting diverse disciplines of science. His Inflammation study incorporates themes from Adipocyte, Hepatocyte, Necrosis, Interleukin and MAPK/ERK pathway. His study in Endocrinology focuses on Liver injury and Lipopolysaccharide.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Innate immune response in Th1- and Th2-dominant mouse strains.

Hiroyuki Watanabe;Kousuke Numata;Takaaki Ito;Katsumasa Takagi.
Shock (2004)

499 Citations

Galectin-9 suppresses the generation of Th17, promotes the induction of regulatory T cells, and regulates experimental autoimmune arthritis

Masako Seki;Souichi Oomizu;Ken mei Sakata;Atsuko Sakata.
Clinical Immunology (2008)

477 Citations

Early Recruitment of Neutrophils Determines Subsequent T1/T2 Host Responses in a Murine Model of Legionella pneumophila Pneumonia

Kazuhiro Tateda;Thomas A. Moore;Jane C. Deng;Michael W. Newstead.
Journal of Immunology (2001)

308 Citations

Endogenous monocyte chemoattractant protein-1 (MCP-1) protects mice in a model of acute septic peritonitis: cross-talk between MCP-1 and leukotriene B4.

Akihiro Matsukawa;Cory M. Hogaboam;Nickolas W. Lukacs;Pamela M. Lincoln.
Journal of Immunology (1999)

281 Citations

Local overexpression of monocyte chemoattractant protein-1 at vessel wall induces infiltration of macrophages and formation of atherosclerotic lesion: synergism with hypercholesterolemia

Masayuki Namiki;Seinosuke Kawashima;Tomoya Yamashita;Masanori Ozaki.
Arteriosclerosis, Thrombosis, and Vascular Biology (2002)

195 Citations

Exaggerated hepatic injury due to acetaminophen challenge in mice lacking C-C chemokine receptor 2

Cory M. Hogaboam;Cynthia L. Bone-Larson;Matthew L. Steinhauser;Akihiro Matsukawa.
American Journal of Pathology (2000)

171 Citations

Expression and Contribution of Endogenous IL-13 in an Experimental Model of Sepsis

Akihiro Matsukawa;Cory M. Hogaboam;Nickolas W. Lukacs;Pamela M. Lincoln.
Journal of Immunology (2000)

169 Citations

Aberrant inflammation and lethality to septic peritonitis in mice lacking STAT3 in macrophages and neutrophils.

Akihiro Matsukawa;Kiyoshi Takeda;Shinji Kudo;Takako Maeda.
Journal of Immunology (2003)

157 Citations

Pivotal role of signal transducer and activator of transcription (Stat)4 and Stat6 in the innate immune response during sepsis.

Akihiro Matsukawa;Mark H. Kaplan;Cory M. Hogaboam;Nicholas W. Lukacs.
Journal of Experimental Medicine (2001)

152 Citations

Pivotal Role of the CC Chemokine, Macrophage-Derived Chemokine, in the Innate Immune Response

Akihiro Matsukawa;Cory M. Hogaboam;Nickolas W. Lukacs;Pamela M. Lincoln.
Journal of Immunology (2000)

141 Citations

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