R. Stokes Peebles mainly investigates Immunology, Virus, Virology, Respiratory system and Lung. His studies link Bronchoalveolar lavage with Immunology. The Virology study combines topics in areas such as Immunoglobulin E, Cytokine and Pathogenesis.
His research in Respiratory system intersects with topics in Ovalbumin, Mucus and Allergy. His work investigates the relationship between Lung and topics such as STAT1 that intersect with problems in Secretion, Interleukin 10, Epitope and Eotaxin. He interconnects Endocrinology, Inflammation, Interleukin 33, Signal transduction and Bone marrow in the investigation of issues within Interleukin 13.
The scientist’s investigation covers issues in Immunology, Immune system, Virus, Inflammation and Virology. His work on Immunology is being expanded to include thematically relevant topics such as Lung. R. Stokes Peebles has researched Immune system in several fields, including Cellular differentiation and Cytokine.
His Virus study combines topics in areas such as Respiratory tract and Respiratory system. His work carried out in the field of Inflammation brings together such families of science as Interleukin, Receptor, Innate immune system and Endocrinology. R. Stokes Peebles studied Asthma and Respiratory disease that intersect with Eosinophilia.
R. Stokes Peebles focuses on Immunology, Innate lymphoid cell, Immune system, Virus and Inflammation. Immunology is closely attributed to Lung in his work. His research on Innate lymphoid cell also deals with topics like
His Immune system research is multidisciplinary, incorporating elements of Absorption, Multiplex, Immunoassay and Metagenomics. His work deals with themes such as Interferon, Respiratory tract infections, Respiratory system and Disease, which intersect with Virus. His Inflammation research integrates issues from Eosinophil, Innate immune system, Pharmacology and House dust mite.
R. Stokes Peebles mostly deals with Immunology, Virus, Immune system, Innate lymphoid cell and Interleukin 33. Immunology is often connected to Lung in his work. His studies examine the connections between Virus and genetics, as well as such issues in Respiratory system, with regards to Microbiome.
The concepts of his Immune system study are interwoven with issues in Pulmonary fibrosis, Idiopathic pulmonary fibrosis, Immunopathology and STAT1. His Innate lymphoid cell study incorporates themes from Thymic stromal lymphopoietin, Cytokine and Signal transduction. The various areas that R. Stokes Peebles examines in his Interleukin 33 study include Receptor and Interleukin 13.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Th17-mediated inflammation in asthma
Dawn C Newcomb;R Stokes Peebles.
Current Opinion in Immunology (2013)
The Role of IFN in Respiratory Syncytial Virus Pathogenesis
Joan E. Durbin;Joan E. Durbin;Teresa R. Johnson;Russell K. Durbin;Russell K. Durbin;Sara E. Mertz;Sara E. Mertz.
Journal of Immunology (2002)
Differential Type I Interferon Induction by Respiratory Syncytial Virus and Influenza A Virus In Vivo
Nancy A. Jewell;Negin Vaghefi;Sara E. Mertz;Parvis Akter.
Journal of Virology (2007)
Early infection with respiratory syncytial virus impairs regulatory T cell function and increases susceptibility to allergic asthma
Nandini Krishnamoorthy;Anupriya Khare;Timothy B Oriss;Mahesh Raundhal.
Nature Medicine (2012)
Prostaglandin I2 Analogs Inhibit Proinflammatory Cytokine Production and T Cell Stimulatory Function of Dendritic Cells
Weisong Zhou;Koichi Hashimoto;Kasia Goleniewska;Jamye F. O’Neal.
Journal of Immunology (2007)
Human Metapneumovirus Infection Plays an Etiologic Role in Acute Asthma Exacerbations Requiring Hospitalization in Adults
John V. Williams;James E. Crowe;Rachel Enriquez;Patricia Minton.
The Journal of Infectious Diseases (2005)
Epidemiology of asthma: the year in review
Tina V. Hartert;R. Stokes Peebles.
Current Opinion in Pulmonary Medicine (2000)
Differential immune responses and pulmonary pathophysiology are induced by two different strains of respiratory syncytial virus.
Nicholas W. Lukacs;Martin L. Moore;Brian D. Rudd;Aaron A. Berlin.
American Journal of Pathology (2006)
Differential Pathogenesis of Respiratory Syncytial Virus Clinical Isolates in BALB/c Mice
Kate L. Stokes;Michael H. Chi;Kaori Sakamoto;Dawn C. Newcomb.
Journal of Virology (2011)
Immune-mediated disease pathogenesis in respiratory syncytial virus infection.
Barney S. Graham;Teresa R. Johnson;R.Stokes Peebles.
Immunopharmacology (2000)
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