His primary scientific interests are in Immunology, Cytokine, Immune system, Chemokine and Inflammation. His Immunology research is multidisciplinary, incorporating perspectives in Respiratory disease, Lung and Respiratory tract, Respiratory system. His work on Interleukin, Interferon gamma and Interleukin 10 as part of his general Cytokine study is frequently connected to Breast feeding, thereby bridging the divide between different branches of science.
His Immune system research incorporates themes from Anti-inflammatory and Virus, Viral replication. His Chemokine study incorporates themes from Transcription factor, Chemotaxis and Monocyte. As a part of the same scientific study, Roberto P. Garofalo usually deals with the Inflammation, concentrating on NFKB1 and frequently concerns with Molecular biology, Gene isoform, Gene expression profiling and Gene expression.
His primary areas of investigation include Immunology, Chemokine, Virology, Virus and Immune system. He interconnects Lung and Respiratory system in the investigation of issues within Immunology. Roberto P. Garofalo has researched Chemokine in several fields, including Proinflammatory cytokine, Interferon, Chemotaxis and Interleukin 8.
His work carried out in the field of Virology brings together such families of science as Human metapneumovirus, Respiratory tract infections and Dendritic cell. His research investigates the link between Immune system and topics such as Cell biology that cross with problems in Receptor. His biological study spans a wide range of topics, including Respiratory disease and Respiratory tract.
His primary scientific interests are in Immunology, Cell biology, Chemokine, Inflammation and Bronchiolitis. His Immunology study integrates concerns from other disciplines, such as Respiratory tract infections, Respiratory system and Lung. His Cell biology research is multidisciplinary, relying on both Interferon, CXCL1, Receptor, CXC chemokine receptors and Gene silencing.
His Chemokine study combines topics from a wide range of disciplines, such as Proinflammatory cytokine, Peripheral blood mononuclear cell, Calcium-sensing receptor and Bone resorption. His research integrates issues of Calcium metabolism, Calcium and Oxidative stress, Endocrinology in his study of Inflammation. His Bronchiolitis research incorporates elements of Tumor necrosis factor alpha and Microbiology.
His main research concerns Immunology, Virus, Chemokine, Immune system and Inflammation. The various areas that Roberto P. Garofalo examines in his Immunology study include Oxidative stress, Lung and Cell biology. His Chemokine research includes elements of Calcium metabolism, Calcium, Interferon, Bone resorption and Peripheral blood mononuclear cell.
His study in Immune system is interdisciplinary in nature, drawing from both Respiratory tract, Secretory protein and Respiratory tract infections. His Inflammation study combines topics in areas such as Reactive oxygen species, Proteasome, KEAP1, GPX1 and Pharmacology. His Viral replication research integrates issues from Bronchiolitis and Lung injury.
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Severe Human Lower Respiratory Tract Illness Caused by Respiratory Syncytial Virus and Influenza Virus Is Characterized by the Absence of Pulmonary Cytotoxic Lymphocyte Responses
Timothy P. Welliver;Roberto P. Garofalo;Yashoda Hosakote;Karen H. Hintz.
The Journal of Infectious Diseases (2007)
Metapneumovirus and acute wheezing in children
Tuomas Jartti;Bernadette Van Den Hoogen;Roberto P. Garofalo;Albert D. M. E. Osterhaus.
The Lancet (2002)
Retinoic Acid-Inducible Gene I Mediates Early Antiviral Response and Toll-Like Receptor 3 Expression in Respiratory Syncytial Virus-Infected Airway Epithelial Cells
Ping Liu;Mohammad Jamaluddin;Kui Li;Roberto P. Garofalo.
Journal of Virology (2007)
Eosinophil degranulation in the respiratory tract during naturally acquired respiratory syncytial virus infection.
Roberto Garofalo;Jan L.L. Kimpen;Jan L.L. Kimpen;Robert C. Welliver;Robert C. Welliver;Pearay L. Ogra.
The Journal of Pediatrics (1992)
Respiratory Syncytial Virus–Induced Activation of Nuclear Factor–κB in the Lung Involves Alveolar Macrophages and Toll-Like Receptor 4–Dependent Pathways
Helene A. Haeberle;Ryuta Takizawa;Antonella Casola;Allan R. Brasier.
The Journal of Infectious Diseases (2002)
Transcriptional Activation of the Interleukin-8 Gene by Respiratory Syncytial Virus Infection in Alveolar Epithelial Cells: Nuclear Translocation of the RelA Transcription Factor as a Mechanism Producing Airway Mucosal Inflammation
Roberto Garofalo;Mona Sabry;Mohammad Jamaluddin;Robert K. Yu.
Journal of Virology (1996)
Cell-Specific Expression of RANTES, MCP-1, and MIP-1α by Lower Airway Epithelial Cells and Eosinophils Infected with Respiratory Syncytial Virus
Barbara Olszewska-Pazdrak;Antonella Casola;Tadahito Saito;Rafeul Alam.
Journal of Virology (1998)
Macrophage Inflammatory Protein-1α (Not T Helper Type 2 Cytokines) Is Associated with Severe Forms of Respiratory Syncytial Virus Bronchiolitis
Roberto P. Garofalo;John Patti;Karen A. Hintz;Vanessa Hill.
The Journal of Infectious Diseases (2001)
Expression of Respiratory Syncytial Virus-Induced Chemokine Gene Networks in Lower Airway Epithelial Cells Revealed by cDNA Microarrays
Yuhong Zhang;Bruce A. Luxon;Antonella Casola;Roberto P. Garofalo.
Journal of Virology (2001)
Cytokines in Breast Milk from Allergic and Nonallergic Mothers
Malin Fagerås Böttcher;Maria C. Jenmalm;Roberto Garofalo;Bengt Björkstén.
Pediatric Research (2000)
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