Antonella Casola mostly deals with Molecular biology, Immunology, Inflammation, Chemokine and Virus. The various areas that Antonella Casola examines in her Molecular biology study include Gene expression, Interferon, Reactive oxygen species, Transcription factor and Regulation of gene expression. Her Immunology research focuses on Immune system in particular.
Her Inflammation research focuses on subjects like Lung, which are linked to Lower respiratory tract infection, Pathogenesis and Butylated hydroxyanisole. Antonella Casola interconnects Chemotaxis, Basophil and Monocyte in the investigation of issues within Chemokine. Her study on Virus is covered under Virology.
Her primary scientific interests are in Immunology, Virology, Virus, Human metapneumovirus and Chemokine. Her study in Lung extends to Immunology with its themes. Her work on Interferon as part of general Virology research is frequently linked to Mononegavirales, thereby connecting diverse disciplines of science.
Antonella Casola works mostly in the field of Virus, limiting it down to concerns involving Cytokine and, occasionally, Tumor necrosis factor alpha and A549 cell. As part of one scientific family, Antonella Casola deals mainly with the area of Human metapneumovirus, narrowing it down to issues related to the Microbiology, and often Downregulation and upregulation and Chemokine secretion. Her Chemokine research is multidisciplinary, incorporating elements of Regulation of gene expression, Signal transduction, G protein and Interleukin 8.
Her scientific interests lie mostly in Immunology, Virus, Microbiology, Human metapneumovirus and Virology. Many of her studies on Immunology apply to Lung as well. Her Virus study combines topics from a wide range of disciplines, such as Microvesicles, Respiratory tract infections, Respiratory system and RNA.
Her Microbiology research incorporates elements of A549 cell, Lactate dehydrogenase A, Cytokine and Innate immune system. The Human metapneumovirus study combines topics in areas such as TLR7, Signal transduction, Interferon and Citric acid cycle. The concepts of her Proinflammatory cytokine study are interwoven with issues in Chemokine and Lung injury.
Virus, Immunology, Inflammation, Oxidative stress and Reactive oxygen species are her primary areas of study. Her Virus research is within the category of Virology. In her study, Cell signaling is strongly linked to Lung, which falls under the umbrella field of Immunology.
Her research investigates the connection between Oxidative stress and topics such as Glutathione that intersect with issues in Cell biology. Her research in Reactive oxygen species intersects with topics in Thioredoxin and Signal transduction. Her research in RNA focuses on subjects like Exosome, which are connected to Chemokine, A549 cell, Microbiology and Cytokine.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Retinoic Acid-Inducible Gene I Mediates Early Antiviral Response and Toll-Like Receptor 3 Expression in Respiratory Syncytial Virus-Infected Airway Epithelial Cells
Ping Liu;Mohammad Jamaluddin;Kui Li;Roberto P. Garofalo.
Journal of Virology (2007)
Nuclear factor-κB-dependent induction of interleukin-8 gene expression by tumor necrosis factor α : Evidence for an antioxidant sensitive activating pathway distinct from nuclear translocation
Spiros Vlahopoulos;Istvan Boldogh;Antonella Casola;Allan R. Brasier.
Blood (1999)
Respiratory Syncytial Virus–Induced Activation of Nuclear Factor–κB in the Lung Involves Alveolar Macrophages and Toll-Like Receptor 4–Dependent Pathways
Helene A. Haeberle;Ryuta Takizawa;Antonella Casola;Allan R. Brasier.
The Journal of Infectious Diseases (2002)
Transcriptional Activation of the Interleukin-8 Gene by Respiratory Syncytial Virus Infection in Alveolar Epithelial Cells: Nuclear Translocation of the RelA Transcription Factor as a Mechanism Producing Airway Mucosal Inflammation
Roberto Garofalo;Mona Sabry;Mohammad Jamaluddin;Robert K. Yu.
Journal of Virology (1996)
Cell-Specific Expression of RANTES, MCP-1, and MIP-1α by Lower Airway Epithelial Cells and Eosinophils Infected with Respiratory Syncytial Virus
Barbara Olszewska-Pazdrak;Antonella Casola;Tadahito Saito;Rafeul Alam.
Journal of Virology (1998)
Expression of Respiratory Syncytial Virus-Induced Chemokine Gene Networks in Lower Airway Epithelial Cells Revealed by cDNA Microarrays
Yuhong Zhang;Bruce A. Luxon;Antonella Casola;Roberto P. Garofalo.
Journal of Virology (2001)
A Promoter Recruitment Mechanism for Tumor Necrosis Factor-α-induced Interleukin-8 Transcription in Type II Pulmonary Epithelial Cells DEPENDENCE ON NUCLEAR ABUNDANCE OF Rel A, NF-κB1, AND c-Rel TRANSCRIPTION FACTORS
Allan R. Brasier;Mohammad Jamaluddin;Antonella Casola;Weili Duan.
Journal of Biological Chemistry (1998)
Inducible Expression of Inflammatory Chemokines in Respiratory Syncytial Virus-Infected Mice: Role of MIP-1α in Lung Pathology
Helene A. Haeberle;William A. Kuziel;Hans-Juergen Dieterich;Antonella Casola.
Journal of Virology (2001)
Respiratory Syncytial Virus Induces Selective Production of the Chemokine RANTES by Upper Airway Epithelial Cells
Tadahito Saito;Ronald W. Deskin;Antonella Casola;Helene Häeberle.
The Journal of Infectious Diseases (1997)
Differential response of dendritic cells to human metapneumovirus and respiratory syncytial virus
Antonieta Guerrero-Plata;Antonella Casola;Giovanni Suarez;Xiang Yu.
American Journal of Respiratory Cell and Molecular Biology (2006)
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