D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 56 Citations 14,687 94 World Ranking 2532 National Ranking 1032

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Virus
  • RNA

Kui Li mainly focuses on Virology, Interferon, Innate immune system, Hepatitis C virus and NS3. His Virology research includes themes of TLR3, Toll-like receptor, ISG15 and IRF3. His Interferon research incorporates elements of RNA, Molecular biology, Signal transduction and Viral replication.

His work on Mitochondrial antiviral-signaling protein and Pattern recognition receptor as part of his general Innate immune system study is frequently connected to Coronavirus, thereby bridging the divide between different branches of science. His NS3 study contributes to a more complete understanding of Virus. The NS2-3 protease study combines topics in areas such as Signal transducing adaptor protein and Interferon regulatory factors.

His most cited work include:

  • Immune evasion by hepatitis C virus NS3/4A protease-mediated cleavage of the Toll-like receptor 3 adaptor protein TRIF (937 citations)
  • Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by hepatitis C virus core protein (793 citations)
  • Regulating Intracellular Antiviral Defense and Permissiveness to Hepatitis C Virus RNA Replication through a Cellular RNA Helicase, RIG-I (777 citations)

What are the main themes of his work throughout his whole career to date?

Kui Li mainly investigates Virology, Interferon, Innate immune system, Virus and Hepatitis C virus. His biological study spans a wide range of topics, including Toll-like receptor and IRF3. His work deals with themes such as Molecular biology, Signal transduction, TLR3 and Interferon regulatory factors, which intersect with Interferon.

When carried out as part of a general Innate immune system research project, his work on Pattern recognition receptor is frequently linked to work in Coronavirus, therefore connecting diverse disciplines of study. His study in the fields of Antibody-dependent enhancement under the domain of Virus overlaps with other disciplines such as Pestivirus. His Hepatitis C virus research is multidisciplinary, incorporating elements of Flavivirus and In situ hybridization, Digoxigenin.

He most often published in these fields:

  • Virology (67.71%)
  • Interferon (51.04%)
  • Innate immune system (35.42%)

What were the highlights of his more recent work (between 2013-2021)?

  • Virology (67.71%)
  • Innate immune system (35.42%)
  • Interferon (51.04%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Virology, Innate immune system, Interferon, Viral replication and Virus. His Virology research is multidisciplinary, incorporating perspectives in TLR3 and Picornavirus. Kui Li combines subjects such as Porcine reproductive and respiratory syndrome virus and microRNA with his study of Innate immune system.

His Interferon study incorporates themes from Pegylated interferon, Signal transduction, Immune system and MDA5. His research integrates issues of Ectopic expression, Immunity and Cell biology in his study of Viral replication. His work investigates the relationship between Virus and topics such as NF-κB that intersect with problems in Pattern recognition receptor, Viral pathogenesis and Innate lymphoid cell.

Between 2013 and 2021, his most popular works were:

  • Toll-like receptors in antiviral innate immunity (309 citations)
  • Porcine Epidemic Diarrhea Virus 3C-Like Protease Regulates Its Interferon Antagonism by Cleaving NEMO (73 citations)
  • Hepatitis A Virus 3C Protease Cleaves NEMO To Impair Induction of Beta Interferon (54 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Immune evasion by hepatitis C virus NS3/4A protease-mediated cleavage of the Toll-like receptor 3 adaptor protein TRIF

Kui Li;Eileen Foy;Josephine C. Ferreon;Mitsuyasu Nakamura.
Proceedings of the National Academy of Sciences of the United States of America (2005)

1345 Citations

Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by hepatitis C virus core protein

Michiari Okuda;Kui Li;Michael R. Beard;Lori A. Showalter.
Gastroenterology (2002)

1094 Citations

Regulation of Interferon Regulatory Factor-3 by the Hepatitis C Virus Serine Protease

Eileen Foy;Kui Li;Chunfu Wang;Rhea Sumpter.
Science (2003)

1067 Citations

Regulating Intracellular Antiviral Defense and Permissiveness to Hepatitis C Virus RNA Replication through a Cellular RNA Helicase, RIG-I

Rhea Sumpter;Yueh Ming Loo;Eileen Foy;Kui Li.
Journal of Virology (2005)

1055 Citations

Control of antiviral defenses through hepatitis C virus disruption of retinoic acid-inducible gene-I signaling.

Eileen Foy;Kui Li;Rhea Sumpter;Yueh Ming Loo.
Proceedings of the National Academy of Sciences of the United States of America (2005)

690 Citations

Selectable Subgenomic and Genome-Length Dicistronic RNAs Derived from an Infectious Molecular Clone of the HCV-N Strain of Hepatitis C Virus Replicate Efficiently in Cultured Huh7 Cells

Masanori Ikeda;Min Kyung Yi;Kui Li;Stanley M. Lemon.
Journal of Virology (2002)

637 Citations

Toll-like receptors in antiviral innate immunity

Sandra N. Lester;Kui Li.
Journal of Molecular Biology (2014)

581 Citations

Viral and therapeutic control of IFN-β promoter stimulator 1 during hepatitis C virus infection

Yueh Ming Loo;David M. Owen;Kui Li;Andrea K. Erickson.
Proceedings of the National Academy of Sciences of the United States of America (2006)

497 Citations

Distinct Poly(I-C) and Virus-activated Signaling Pathways Leading to Interferon-β Production in Hepatocytes

Kui Li;Zihong Chen;Nobuyuki Kato;Michael Gale.
Journal of Biological Chemistry (2005)

493 Citations

Regulation of IRF-3-dependent Innate Immunity by the Papain-like Protease Domain of the Severe Acute Respiratory Syndrome Coronavirus *

Santhana G. Devaraj;Nan Wang;Zhongbin Chen;Zihong Chen.
Journal of Biological Chemistry (2007)

396 Citations

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