His main research concerns Immunology, Monocyte, Chemokine, Cancer research and Cytokine. Barrett J. Rollins combines subjects such as Inflammation, Endocrinology, Chemotaxis and Macrophage with his study of Monocyte. The concepts of his Chemotaxis study are interwoven with issues in Molecular biology and Cell culture.
His Chemokine research is multidisciplinary, incorporating perspectives in Cancer, Transgene and Disease, Pathogenesis, Pathology. The various areas that Barrett J. Rollins examines in his Cancer research study include Mutation, Protein kinase B, Apolipoprotein E and Bone marrow. His work carried out in the field of Cytokine brings together such families of science as Tumor necrosis factor alpha, Growth factor and FGF10.
Barrett J. Rollins focuses on Immunology, Chemokine, Monocyte, Cancer research and Molecular biology. His Immunology study often links to related topics such as Macrophage. His Chemokine study integrates concerns from other disciplines, such as Cancer and Cell biology.
His Monocyte research incorporates elements of Chemoattractant activity, Endocrinology, In vitro and Pathology. His Cancer research study combines topics in areas such as Mutation, Protein kinase B, Gene silencing and MAPK/ERK pathway. His Molecular biology research is multidisciplinary, relying on both Cell culture, Regulation of gene expression, Gene expression, Gene and Chemotaxis.
Barrett J. Rollins mainly focuses on Langerhans cell histiocytosis, Cancer research, Bioinformatics, Histiocytosis and Cancer. His Langerhans cell histiocytosis study is concerned with Pathology in general. His biological study spans a wide range of topics, including Cell of origin and Dendritic cell.
His study in Cancer research is interdisciplinary in nature, drawing from both Mutation, Inflammation, Immunology and MAPK/ERK pathway. Inflammation is a component of his Chemokine, CCL2 and CCR2 studies. His Immunology study frequently links to adjacent areas such as Neovascularization.
His primary areas of study are Histiocytosis, Langerhans cell histiocytosis, Pathology, Protein kinase domain and MAPK/ERK pathway. His Pathology research includes elements of Cell of origin and Phenotype. The Protein kinase domain study combines topics in areas such as Mutation, Cancer research, ARAF and Protein kinase A.
His studies deal with areas such as Molecular biology and Wild type as well as MAPK/ERK pathway. The Disease study which covers Retrospective cohort study that intersects with Bioinformatics. His work in Bioinformatics addresses subjects such as Genotype, which are connected to disciplines such as Cancer.
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The β-Chemokine Receptors CCR3 and CCR5 Facilitate Infection by Primary HIV-1 Isolates
Hyeryun Choe;Michael Farzan;Ying Sun;Nancy Sullivan.
Absence of Monocyte Chemoattractant Protein-1 Reduces Atherosclerosis in Low Density Lipoprotein Receptor–Deficient Mice
Long Gu;Yoshikatsu Okada;Steven K. Clinton;Craig Gerard.
Molecular Cell (1998)
Chemokines and disease.
Craig Gerard;Barrett J. Rollins.
Nature Immunology (2001)
Abnormalities in Monocyte Recruitment and Cytokine Expression in Monocyte Chemoattractant Protein 1–deficient Mice
Bao Lu;Barbara J. Rutledge;Long Gu;Joseph Fiorillo.
Journal of Experimental Medicine (1998)
AACR project genie: Powering precision medicine through an international consortium
S. M. Sweeney;E. Cerami;A. Baras;T. J. Pugh.
Cancer Discovery (2017)
Recurrent BRAF mutations in Langerhans cell histiocytosis
Gayane Badalian-Very;Gayane Badalian-Very;Jo-Anne Vergilio;Jo-Anne Vergilio;Barbara A. Degar;Barbara A. Degar;Laura E. MacConaill.
Mice deficient in tumor necrosis factor-alpha are resistant to skin carcinogenesis.
Robert J. Moore;David M. Owens;Gordon Stamp;Caroline Arnott.
Nature Medicine (1999)
MCP-1 deficiency reduces susceptibility to atherosclerosis in mice that overexpress human apolipoprotein B
Jennifa Gosling;Sarah Slaymaker;Long Gu;Susan Tseng.
Journal of Clinical Investigation (1999)
Control of TH2 polarization by the chemokine monocyte chemoattractant protein-1.
Long Gu;Susan Tseng;Renée M. Horner;Carmen Tam.
Absence of Monocyte Chemoattractant Protein 1 in Mice Leads to Decreased Local Macrophage Recruitment and Antigen-Specific T Helper Cell Type 1 Immune Response in Experimental Autoimmune Encephalomyelitis
DeRen Huang;Jintang Wang;Pia Kivisakk;Barrett J. Rollins.
Journal of Experimental Medicine (2001)
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