What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Cytokine
William A. Kuziel mainly investigates CCR2, Immunology, CC chemokine receptors, Chemokine and Immune system.
The study incorporates disciplines such as Pathology, Endocrinology and Monocyte in addition to CCR2.
His research on Immunology often connects related topics like Lung.
William A. Kuziel has included themes like CCL21, CCL5, C-C chemokine receptor type 6, CXCL16 and T helper cell in his CC chemokine receptors study.
His work carried out in the field of Chemokine brings together such families of science as Influenza A virus, Neuroglia, Glial fibrillary acidic protein and Leukocyte migration.
The Chemokine receptor study combines topics in areas such as Leukocyte chemotaxis and Lymphocyte.
His most cited work include:
- TNF/iNOS-producing dendritic cells mediate innate immune defense against bacterial infection. (964 citations)
- Severe reduction in leukocyte adhesion and monocyte extravasation in mice deficient in CC chemokine receptor 2 (615 citations)
- An animal model of age-related macular degeneration in senescent Ccl-2- or Ccr-2-deficient mice. (584 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Immunology, CCR2, Chemokine, CC chemokine receptors and Chemokine receptor.
His research on Immunology frequently links to adjacent areas such as Lung.
His CCR2 research incorporates themes from CCL2, Endocrinology and Monocyte.
His Monocyte research incorporates elements of Tumor necrosis factor alpha, Microbiology, Proinflammatory cytokine, Bronchoalveolar lavage and Lung injury.
William A. Kuziel has researched Chemokine in several fields, including Pathogenesis, Pathology, Cytokine and Cell biology.
His research in CC chemokine receptors intersects with topics in CX3CR1, CXCL2, CCL13, C-C chemokine receptor type 6 and Signal transduction.
He most often published in these fields:
- Immunology (71.17%)
- CCR2 (63.96%)
- Chemokine (47.75%)
What were the highlights of his more recent work (between 2006-2014)?
- Immunology (71.17%)
- CCR2 (63.96%)
- Chemokine (47.75%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Immunology, CCR2, Chemokine, Endocrinology and Internal medicine.
His biological study spans a wide range of topics, including Receptor and Central nervous system.
CCR2 is a subfield of Chemokine receptor that William A. Kuziel explores.
His Chemokine receptor research is multidisciplinary, incorporating elements of CD8 and Virology.
His research integrates issues of Phenotype and Intracellular parasite in his study of Chemokine.
William A. Kuziel interconnects Inflammation and CC chemokine receptors in the investigation of issues within Endocrinology.
Between 2006 and 2014, his most popular works were:
- Lung epithelial apoptosis in influenza virus pneumonia: the role of macrophage-expressed TNF-related apoptosis-inducing ligand (260 citations)
- Role of CC Chemokine Receptor 2 in Bone Marrow Cells in the Recruitment of Macrophages into Obese Adipose Tissue (117 citations)
- Role of inflammation in the development of renal damage and dysfunction in Angiotensin II-induced hypertension (115 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Cytokine
His main research concerns CCR2, Immunology, CC chemokine receptors, Receptor and Internal medicine.
His study ties his expertise on Adipose tissue together with the subject of CCR2.
His Tumor necrosis factor alpha, Chemokine receptor, C-C chemokine receptor type 6, CXCL10 and CCR1 investigations are all subjects of Immunology research.
His study in CC chemokine receptors is interdisciplinary in nature, drawing from both Proinflammatory cytokine, CCL13, CXCL2 and Monocyte.
His Receptor study combines topics in areas such as Exudate, Orthomyxoviridae, Apoptosis, Virus and Lung.
His study focuses on the intersection of Internal medicine and fields such as Endocrinology with connections in the field of Inflammation, Bone marrow and Mannose receptor.
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