His primary scientific interests are in Immunology, Eosinophilia, Eotaxin, Interleukin 5 and Eosinophil. His Immunology research includes elements of Eosinophilic esophagitis, Lung and Pathology. His Eotaxin study combines topics in areas such as CCR3, Cytokine, Pathogenesis, Cytotoxic T cell and Immunotherapy.
His work in Interleukin 5 addresses issues such as T cell, which are connected to fields such as BALB/c and Respiratory epithelium. His Eosinophil cationic protein, Eosinophil-derived neurotoxin and Eosinophil Granule Proteins study, which is part of a larger body of work in Eosinophil, is frequently linked to Mucus, bridging the gap between disciplines. His Inflammation study incorporates themes from Immunoglobulin E, Aeroallergen, Ovalbumin and Sensitization.
His primary scientific interests are in Immunology, Eosinophil, Inflammation, Eotaxin and Eosinophilia. His work is connected to Interleukin 5, Anaphylaxis, Immunoglobulin E, Cytokine and Immune system, as a part of Immunology. His study looks at the relationship between Eosinophil and topics such as Chemokine, which overlap with Chemotaxis.
His Inflammation research incorporates elements of Intestinal mucosa, Colitis and Pathology. His Eotaxin study combines topics from a wide range of disciplines, such as CCR3 and Interleukin 13. The study incorporates disciplines such as Pathogenesis and Aeroallergen in addition to Eosinophilia.
His primary areas of study are Immunology, Cell biology, Inflammation, Immune system and Mast cell. His study on Immunology is mostly dedicated to connecting different topics, such as Inflammatory bowel disease. His work on Function is typically connected to STARD7 as part of general Cell biology study, connecting several disciplines of science.
His research in Inflammation tackles topics such as Innate immune system which are related to areas like CCL11, T helper cell, Pathogenesis, Eosinophil and Calprotectin. His research in Immune system intersects with topics in Gastrointestinal tract and Effector. Simon P. Hogan combines subjects such as Food allergy, Immunoglobulin E and ABL with his study of Mast cell.
His primary areas of investigation include Immunology, Immune system, Immunoglobulin E, Mast cell and Inflammation. His studies deal with areas such as Intestinal mucosa, Inflammatory bowel disease and Ulcerative colitis as well as Immunology. His Immune system course of study focuses on Antigen and Food allergy, Interleukin 13 and Gut flora.
In his study, Lipopolysaccharide, ABL, Receptor and Extravasation is strongly linked to Anaphylaxis, which falls under the umbrella field of Immunoglobulin E. Simon P. Hogan has researched Inflammation in several fields, including Superior mesenteric artery, Barrier function and Tight junction. The Allergic inflammation study which covers Eosinophil that intersects with Cytokine.
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Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model.
P S Foster;S P Hogan;A J Ramsay;K I Matthaei.
Journal of Experimental Medicine (1996)
Intestinal barrier function: molecular regulation and disease pathogenesis.
Katherine R. Groschwitz;Simon P. Hogan.
The Journal of Allergy and Clinical Immunology (2009)
Eosinophils: Biological Properties and Role in Health and Disease
Simon P. Hogan;Helene F. Rosenberg;Redwan Moqbel;Simon Phipps.
Clinical & Experimental Allergy (2008)
Eotaxin-3 and a uniquely conserved gene-expression profile in eosinophilic esophagitis
Carine Blanchard;Ning Wang;Keith F. Stringer;Anil Mishra.
Journal of Clinical Investigation (2006)
An etiological role for aeroallergens and eosinophils in experimental esophagitis
Anil Mishra;Simon P. Hogan;Eric B. Brandt;Marc E. Rothenberg.
Journal of Clinical Investigation (2001)
An improved murine model of asthma: selective airway inflammation, epithelial lesions and increased methacholine responsiveness following chronic exposure to aerosolised allergen
Jason Temelkovski;Simon P Hogan;Darren P Shepherd;Paul S Foster.
Thorax (1998)
Fundamental signals that regulate eosinophil homing to the gastrointestinal tract
Anil Mishra;Simon P. Hogan;James J. Lee;Paul S. Foster.
Journal of Clinical Investigation (1999)
Transgenic expression of bean alpha-amylase inhibitor in peas results in altered structure and immunogenicity.
Vanessa E. Prescott;Peter M. Campbell;Andrew Moore;Joerg Mattes.
Journal of Agricultural and Food Chemistry (2005)
IL-5 Promotes Eosinophil Trafficking to the Esophagus
Anil Mishra;Simon P. Hogan;Eric B. Brandt;Marc E. Rothenberg.
Journal of Immunology (2002)
Intrinsic defect in T cell production of interleukin (IL)-13 in the absence of both IL-5 and eotaxin precludes the development of eosinophilia and airways hyperreactivity in experimental asthma.
Joerg Mattes;Ming Yang;Surendran Mahalingam;Joachim Kuehr.
Journal of Experimental Medicine (2002)
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