D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 62 Citations 17,067 187 World Ranking 2135 National Ranking 1036

Overview

What is he best known for?

The fields of study he is best known for:

  • Immune system
  • Gene
  • Internal medicine

His primary scientific interests are in Immunology, Eosinophilia, Eotaxin, Interleukin 5 and Eosinophil. His Immunology research includes elements of Eosinophilic esophagitis, Lung and Pathology. His Eotaxin study combines topics in areas such as CCR3, Cytokine, Pathogenesis, Cytotoxic T cell and Immunotherapy.

His work in Interleukin 5 addresses issues such as T cell, which are connected to fields such as BALB/c and Respiratory epithelium. His Eosinophil cationic protein, Eosinophil-derived neurotoxin and Eosinophil Granule Proteins study, which is part of a larger body of work in Eosinophil, is frequently linked to Mucus, bridging the gap between disciplines. His Inflammation study incorporates themes from Immunoglobulin E, Aeroallergen, Ovalbumin and Sensitization.

His most cited work include:

  • Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model. (1264 citations)
  • Intestinal barrier function: molecular regulation and disease pathogenesis. (823 citations)
  • Eotaxin-3 and a uniquely conserved gene-expression profile in eosinophilic esophagitis (670 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Immunology, Eosinophil, Inflammation, Eotaxin and Eosinophilia. His work is connected to Interleukin 5, Anaphylaxis, Immunoglobulin E, Cytokine and Immune system, as a part of Immunology. His study looks at the relationship between Eosinophil and topics such as Chemokine, which overlap with Chemotaxis.

His Inflammation research incorporates elements of Intestinal mucosa, Colitis and Pathology. His Eotaxin study combines topics from a wide range of disciplines, such as CCR3 and Interleukin 13. The study incorporates disciplines such as Pathogenesis and Aeroallergen in addition to Eosinophilia.

He most often published in these fields:

  • Immunology (71.84%)
  • Eosinophil (21.84%)
  • Inflammation (21.84%)

What were the highlights of his more recent work (between 2014-2020)?

  • Immunology (71.84%)
  • Cell biology (12.64%)
  • Inflammation (21.84%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Immunology, Cell biology, Inflammation, Immune system and Mast cell. His study on Immunology is mostly dedicated to connecting different topics, such as Inflammatory bowel disease. His work on Function is typically connected to STARD7 as part of general Cell biology study, connecting several disciplines of science.

His research in Inflammation tackles topics such as Innate immune system which are related to areas like CCL11, T helper cell, Pathogenesis, Eosinophil and Calprotectin. His research in Immune system intersects with topics in Gastrointestinal tract and Effector. Simon P. Hogan combines subjects such as Food allergy, Immunoglobulin E and ABL with his study of Mast cell.

Between 2014 and 2020, his most popular works were:

  • IL-25 and CD4+ TH2 cells enhance type 2 innate lymphoid cell–derived IL-13 production, which promotes IgE-mediated experimental food allergy (78 citations)
  • Microbial antigen encounter during a preweaning interval is critical for tolerance to gut bacteria (65 citations)
  • Modeling TH 2 responses and airway inflammation to understand fundamental mechanisms regulating the pathogenesis of asthma. (55 citations)

In his most recent research, the most cited papers focused on:

  • Immune system
  • Gene
  • Internal medicine

His primary areas of investigation include Immunology, Immune system, Immunoglobulin E, Mast cell and Inflammation. His studies deal with areas such as Intestinal mucosa, Inflammatory bowel disease and Ulcerative colitis as well as Immunology. His Immune system course of study focuses on Antigen and Food allergy, Interleukin 13 and Gut flora.

In his study, Lipopolysaccharide, ABL, Receptor and Extravasation is strongly linked to Anaphylaxis, which falls under the umbrella field of Immunoglobulin E. Simon P. Hogan has researched Inflammation in several fields, including Superior mesenteric artery, Barrier function and Tight junction. The Allergic inflammation study which covers Eosinophil that intersects with Cytokine.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model.

P S Foster;S P Hogan;A J Ramsay;K I Matthaei.
Journal of Experimental Medicine (1996)

1669 Citations

Intestinal barrier function: molecular regulation and disease pathogenesis.

Katherine R. Groschwitz;Simon P. Hogan.
The Journal of Allergy and Clinical Immunology (2009)

1449 Citations

Eosinophils: Biological Properties and Role in Health and Disease

Simon P. Hogan;Helene F. Rosenberg;Redwan Moqbel;Simon Phipps.
Clinical & Experimental Allergy (2008)

943 Citations

Eotaxin-3 and a uniquely conserved gene-expression profile in eosinophilic esophagitis

Carine Blanchard;Ning Wang;Keith F. Stringer;Anil Mishra.
Journal of Clinical Investigation (2006)

892 Citations

An etiological role for aeroallergens and eosinophils in experimental esophagitis

Anil Mishra;Simon P. Hogan;Eric B. Brandt;Marc E. Rothenberg.
Journal of Clinical Investigation (2001)

674 Citations

An improved murine model of asthma: selective airway inflammation, epithelial lesions and increased methacholine responsiveness following chronic exposure to aerosolised allergen

Jason Temelkovski;Simon P Hogan;Darren P Shepherd;Paul S Foster.
Thorax (1998)

491 Citations

Fundamental signals that regulate eosinophil homing to the gastrointestinal tract

Anil Mishra;Simon P. Hogan;James J. Lee;Paul S. Foster.
Journal of Clinical Investigation (1999)

428 Citations

Transgenic expression of bean alpha-amylase inhibitor in peas results in altered structure and immunogenicity.

Vanessa E. Prescott;Peter M. Campbell;Andrew Moore;Joerg Mattes.
Journal of Agricultural and Food Chemistry (2005)

378 Citations

IL-5 Promotes Eosinophil Trafficking to the Esophagus

Anil Mishra;Simon P. Hogan;Eric B. Brandt;Marc E. Rothenberg.
Journal of Immunology (2002)

373 Citations

Intrinsic defect in T cell production of interleukin (IL)-13 in the absence of both IL-5 and eotaxin precludes the development of eosinophilia and airways hyperreactivity in experimental asthma.

Joerg Mattes;Ming Yang;Surendran Mahalingam;Joachim Kuehr.
Journal of Experimental Medicine (2002)

360 Citations

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