D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 45 Citations 10,416 78 World Ranking 2661 National Ranking 96

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Enzyme

His primary areas of investigation include Immunology, Interleukin 5, Eotaxin, Eosinophilia and Inflammation. His study involves Eosinophil, Interleukin 13, Interleukin, Cytokine and T cell, a branch of Immunology. Klaus I. Matthaei combines subjects such as Peritoneal cavity and Lung with his study of Interleukin 5.

His work in Eotaxin addresses issues such as Bone marrow, which are connected to fields such as Pulmonary Eosinophilia, Macrophage inflammatory protein and Endocrinology. His Eosinophilia research is multidisciplinary, incorporating perspectives in Cellular differentiation, Ovalbumin, CD5, Antibody and Stem cell. The various areas that Klaus I. Matthaei examines in his Inflammation study include Sensitization, Eosinophil differentiation, Respiratory epithelium and Asthma.

His most cited work include:

  • Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model. (1264 citations)
  • IL-5-Deficient Mice Have a Developmental Defect in CD5+ B-1 Cells and Lack Eosinophilia but Have Normal Antibody and Cytotoxic T Cell Responses (509 citations)
  • The role of interleukin-6 in mucosal IgA antibody responses in vivo (419 citations)

What are the main themes of his work throughout his whole career to date?

The scientist’s investigation covers issues in Immunology, Interleukin 5, Eosinophil, Internal medicine and Endocrinology. His is involved in several facets of Immunology study, as is seen by his studies on Eosinophilia, Inflammation, Eotaxin, Interleukin 13 and Interleukin. His Eosinophilia research integrates issues from Immunoglobulin E, Aeroallergen and Ovalbumin.

His Interleukin 5 research entails a greater understanding of Cytokine. His work is dedicated to discovering how Eosinophil, Pathogenesis are connected with Asthma and other disciplines. His Endocrinology research includes elements of Receptor and Matrigel.

He most often published in these fields:

  • Immunology (47.88%)
  • Interleukin 5 (25.45%)
  • Eosinophil (21.21%)

What were the highlights of his more recent work (between 2008-2015)?

  • Internal medicine (20.61%)
  • Endocrinology (19.39%)
  • Immunology (47.88%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Internal medicine, Endocrinology, Immunology, Cell biology and Gene. The study incorporates disciplines such as Growth factor and Matrigel in addition to Endocrinology. His study in Immunology concentrates on Eosinophil, Interleukin 5, Nippostrongylus, Nippostrongylus brasiliensis and Secondary infection.

His work deals with themes such as Allergy and Allergic inflammation, which intersect with Eosinophil. Klaus I. Matthaei has researched Interleukin 5 in several fields, including Peritoneal cavity, Eotaxin, Interferon gamma, Cytokine receptor and BALB/c. As part of the same scientific family, he usually focuses on Wound healing, concentrating on Inflammation and intersecting with Cytokine, Eosinophilia, Infiltration, Cell migration and Cell signaling.

Between 2008 and 2015, his most popular works were:

  • Expression of distinct RNAs from 3′ untranslated regions (160 citations)
  • Toll/IL-1 Signaling Is Critical for House Dust Mite–specific Th1 and Th2 Responses (144 citations)
  • Plasmacytoid Dendritic Cells Promote Host Defense against Acute Pneumovirus Infection via the TLR7–MyD88-Dependent Signaling Pathway (74 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Enzyme

His primary scientific interests are in Immunology, Endocrinology, Internal medicine, Eosinophil and Interleukin 5. The study of Immunology is intertwined with the study of Signal transduction in a number of ways. His research in Endocrinology intersects with topics in Receptor, Epithelium, Interleukin 13 and Caco-2.

His Internal medicine research includes themes of Chloride channel, Cell biology and Cystic fibrosis transmembrane conductance regulator. His studies in Eosinophil integrate themes in fields like Secondary infection, House dust mite, Allergy, Allergic inflammation and Innate immune system. His Interleukin 5 research is multidisciplinary, relying on both STAT6, Eotaxin, Cellular immunity and Nippostrongylus brasiliensis, Nippostrongylus.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model.

P S Foster;S P Hogan;A J Ramsay;K I Matthaei.
Journal of Experimental Medicine (1996)

2047 Citations

The role of interleukin-6 in mucosal IgA antibody responses in vivo

Alistair J. Ramsay;Alan J. Husband;Ian A. Ramshaw;Shisan Bao.
Science (1994)

644 Citations

IL-5-Deficient Mice Have a Developmental Defect in CD5+ B-1 Cells and Lack Eosinophilia but Have Normal Antibody and Cytotoxic T Cell Responses

Manfred Kopf;Frank Brombacher;Philip D Hodgkin;Alistair J Ramsay.
Immunity (1996)

641 Citations

Relationship between interleukin-5 and eotaxin in regulating blood and tissue eosinophilia in mice.

A W Mould;K I Matthaei;I G Young;P S Foster.
Journal of Clinical Investigation (1997)

474 Citations

Histone variant H2A.Z is required for early mammalian development.

Renate Faast;Varaporn Thonglairoam;Thomas C Schulz;Jacquie Beall.
Current Biology (2001)

434 Citations

A Novel T Cell-Regulated Mechanism Modulating Allergen-Induced Airways Hyperreactivity in BALB/c Mice Independently of IL-4 and IL-5

Simon P. Hogan;Klaus I. Matthaei;Janine M. Young;Aulikki Koskinen.
Journal of Immunology (1998)

408 Citations

IL-13 induces eosinophil recruitment into the lung by an IL-5- and eotaxin-dependent mechanism.

Samuel M. Pope;Eric B. Brandt;Anil Mishra;Simon P. Hogan.
The Journal of Allergy and Clinical Immunology (2001)

314 Citations

IL-9– and mast cell–mediated intestinal permeability predisposes to oral antigen hypersensitivity

Elizabeth E. Forbes;Katherine Groschwitz;J. Pablo Abonia;Eric B. Brandt.
Journal of Experimental Medicine (2008)

310 Citations

Interleukin-5-producing CD4+ T cells play a pivotal role in aeroallergen-induced eosinophilia, bronchial hyperreactivity, and lung damage in mice

Simon P. Hogan;Aulikki Koskinen;Klaus I. Matthaei;Ian G. Young.
American Journal of Respiratory and Critical Care Medicine (1998)

286 Citations

Elemental signals regulating eosinophil accumulation in the lung.

Paul S. Foster;Arne W. Mould;Ming Yang;Jason Mackenzie.
Immunological Reviews (2001)

261 Citations

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