What is he best known for?
The fields of study he is best known for:
- Immune system
- Cytokine
- Inflammation
Foo Y. Liew mainly investigates Immunology, Cytokine, Interleukin 33, Cell biology and Immune system.
His research in Immunology intersects with topics in Receptor and Cytotoxic T cell.
His work carried out in the field of Cytokine brings together such families of science as Interferon and Innate lymphoid cell.
The study incorporates disciplines such as Immunoglobulin E, Adoptive cell transfer, Cell migration and Macrophage in addition to Interleukin 33.
His research integrates issues of T cell, Cell, Toll-like receptor and CD40 in his study of Cell biology.
His Leishmania major research extends to Immune system, which is thematically connected.
His most cited work include:
- Negative regulation of Toll-like receptor-mediated immune responses (1312 citations)
- Altered immune responses in mice lacking inducible nitric oxide synthase (1168 citations)
- Disease-associated functions of IL-33: the new kid in the IL-1 family. (761 citations)
What are the main themes of his work throughout his whole career to date?
Foo Y. Liew spends much of his time researching Immunology, Cytokine, Inflammation, Immune system and Interleukin 33.
His studies deal with areas such as Nitric oxide and Cell biology as well as Immunology.
His Cytokine research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha, Nitric oxide synthase and Innate lymphoid cell.
As part of one scientific family, Foo Y. Liew deals mainly with the area of Inflammation, narrowing it down to issues related to the Interleukin 18, and often Endocrinology.
His work is dedicated to discovering how Immune system, Antigen are connected with Antibody and other disciplines.
Foo Y. Liew interconnects Mast cell, Adoptive cell transfer, Interleukin 17 and Degranulation in the investigation of issues within Interleukin 33.
He most often published in these fields:
- Immunology (75.25%)
- Cytokine (31.02%)
- Inflammation (31.35%)
What were the highlights of his more recent work (between 2014-2021)?
- Immunology (75.25%)
- Interleukin 33 (26.07%)
- Inflammation (31.35%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Immunology, Interleukin 33, Inflammation, Disease and Sepsis.
His research in Proinflammatory cytokine, Cytokine, Immune system, Arthritis and Rheumatoid arthritis are components of Immunology.
His Cytokine research is multidisciplinary, incorporating elements of Dengue virus, Dengue fever and Receptor antagonist.
Foo Y. Liew has researched Interleukin 33 in several fields, including Innate immune system, Innate lymphoid cell, Interleukin 12, ZAP70 and Inflammatory response.
Foo Y. Liew combines subjects such as Receptor, Cancer research, Tendinopathy and Effector with his study of Inflammation.
His Sepsis research is multidisciplinary, relying on both CCR2 and Chemokine receptor.
Between 2014 and 2021, his most popular works were:
- Interleukin-33 in health and disease (412 citations)
- IL-33 ameliorates Alzheimer’s disease-like pathology and cognitive decline (148 citations)
- IL-33 ameliorates Alzheimer’s disease-like pathology and cognitive decline (148 citations)
In his most recent research, the most cited papers focused on:
- Immune system
- Cytokine
- Inflammation
The scientist’s investigation covers issues in Immunology, Interleukin 33, Innate lymphoid cell, Microglia and Inflammation.
His works in Methotrexate, IL-2 receptor, Arthritis, Autoimmune disease and Rheumatoid arthritis are all subjects of inquiry into Immunology.
His biological study spans a wide range of topics, including Interleukin 3, Antigen-presenting cell, Natural killer T cell, Neuroscience and Peripheral blood.
His Innate lymphoid cell study integrates concerns from other disciplines, such as Regulatory T cell, Cell, Immunosuppression and Sepsis.
The Microglia study combines topics in areas such as Synaptic plasticity, Proinflammatory cytokine, Alzheimer's disease and Innate immune system.
His study in Inflammation is interdisciplinary in nature, drawing from both Tendinopathy, Tendon and Signal transduction, Cell biology.
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