His scientific interests lie mostly in Immunology, Sepsis, Chemotaxis, Organ dysfunction and Inflammation. All of his Immunology and Interleukin 33, Neutrophil extracellular traps, Chemokine, Immune system and Tumor necrosis factor alpha investigations are sub-components of the entire Immunology study. The concepts of his Sepsis study are interwoven with issues in Proinflammatory cytokine, Lipopolysaccharide, Signal transduction and CXC chemokine receptors.
His Chemotaxis research includes themes of Chemokine receptor and Pharmacology. His Organ dysfunction research incorporates elements of Paralysis and NEUTROPHIL MIGRATION. His specific area of interest is Inflammation, where he studies CXCL1.
His primary scientific interests are in Immunology, Sepsis, Inflammation, Internal medicine and Receptor. His research on Immunology often connects related topics like Chemotaxis. His Chemotaxis research includes elements of CCR2 and CXC chemokine receptors.
His Sepsis study combines topics in areas such as Peritoneal cavity, Lipopolysaccharide, Bacteremia, Proinflammatory cytokine and Systemic inflammation. José C. Alves-Filho combines subjects such as Arthritis, Pathophysiology, Cytokine and Pharmacology with his study of Inflammation. His research investigates the connection between Receptor and topics such as Nitric oxide that intersect with issues in Biochemistry.
José C. Alves-Filho mostly deals with Immunology, Inflammation, Pathophysiology, Cytokine and Cancer research. His biological study spans a wide range of topics, including Apoptosis, Peripheral blood mononuclear cell and Aryl hydrocarbon receptor. His Inflammation study incorporates themes from Macrophage, Sepsis and Cell biology.
His biological study deals with issues like Lipopolysaccharide, which deal with fields such as Pharmacology and Tumor necrosis factor alpha. His Pathophysiology research is multidisciplinary, incorporating perspectives in Epithelium, Lung, A549 cell and Neutrophil extracellular traps. The various areas that José C. Alves-Filho examines in his Cytokine study include Endocrinology, Joint pain and Immune system.
His primary areas of study are Immunology, Pathophysiology, Inflammation, Peripheral blood mononuclear cell and Inflammasome. His studies in Immunology integrate themes in fields like Sciatic nerve and Peripheral nerve injury. His study in Pathophysiology is interdisciplinary in nature, drawing from both Epithelium, Lung, A549 cell and Neutrophil extracellular traps.
His Neutrophil extracellular traps research includes themes of Organ dysfunction, Sepsis, Pathology, Cytotoxic T cell and Programmed cell death. His research in Inflammation intersects with topics in Glycolysis, Pyruvate kinase and PKM2. His research investigates the link between Inflammasome and topics such as Apoptosis that cross with problems in Severity of illness.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Interleukin-33 attenuates sepsis by enhancing neutrophil influx to the site of infection
Jose C Alves-Filho;Fabiane Sônego;Fabricio O Souto;Andressa Freitas.
Nature Medicine (2010)
Regulation of chemokine receptor by Toll-like receptor 2 is critical to neutrophil migration and resistance to polymicrobial sepsis
Jose C. Alves-Filho;Andressa Freitas;Fabricio O. Souto;Fernando Spiller.
Proceedings of the National Academy of Sciences of the United States of America (2009)
IL-33 induces neutrophil migration in rheumatoid arthritis and is a target of anti-TNF therapy
Waldiceu A Verri;Fabrício O Souto;Silvio M Vieira;Sergio C L Almeida.
Annals of the Rheumatic Diseases (2010)
IL-33 attenuates EAE by suppressing IL-17 and IFN-γ production and inducing alternatively activated macrophages.
Hui-Rong Jiang;Marija Milovanović;Debbie Allan;Wanda Niedbala.
European Journal of Immunology (2012)
THE ROLE OF NEUTROPHILS IN SEVERE SEPSIS
José C. Alves-Filho;Andressa de Freitas;Fernando Spiller;Fabrício O. Souto.
Neutrophil paralysis in sepsis.
José C. Alves-Filho;Fernando Spiller;Fernando Q. Cunha.
Neutrophil Extracellular Traps Induce Organ Damage during Experimental and Clinical Sepsis
Paula Giselle Czaikoski;José Maurício Segundo Correia Mota;Daniele Carvalho Nascimento;Fabiane Sônego.
PLOS ONE (2016)
Heme oxygenase/carbon monoxide‐biliverdin pathway down regulates neutrophil rolling, adhesion and migration in acute inflammation
A Freitas;J C Alves‐Filho;D D Secco;A F Neto.
British Journal of Pharmacology (2006)
Toll-like receptor 4 signaling leads to neutrophil migration impairment in polymicrobial sepsis.
José C. Alves-Filho;Andressa de Freitas;Momtchilo Russo;Fernando Q. Cunha.
Critical Care Medicine (2006)
Down-regulation of CXCR2 on Neutrophils in Severe Sepsis Is Mediated by Inducible Nitric Oxide Synthase–derived Nitric Oxide
Fabrício Rios-Santos;José C. Alves-Filho;Fabrício Oliveira Souto;Fernando Spiller.
American Journal of Respiratory and Critical Care Medicine (2007)
Profile was last updated on December 6th, 2021.
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