Bernhard Ryffel mainly investigates Immunology, Inflammation, Tumor necrosis factor alpha, Immune system and Innate immune system. Immunology is represented through his Cytokine, Proinflammatory cytokine, Interleukin 22, Interleukin and Toll-like receptor research. His Inflammation study combines topics in areas such as Fibrosis, Interleukin 33, Adoptive cell transfer and Macrophage.
His Tumor necrosis factor alpha research is multidisciplinary, incorporating elements of Bronchoconstriction, Immunity, Interferon gamma and CD40. Bernhard Ryffel has included themes like Cytotoxic T cell, Gut flora and Myeloid-derived Suppressor Cell in his Immune system study. His study focuses on the intersection of Innate immune system and fields such as Acquired immune system with connections in the field of Mycobacterium tuberculosis.
His scientific interests lie mostly in Immunology, Inflammation, Immune system, Tumor necrosis factor alpha and Receptor. His work deals with themes such as Tuberculosis and Mycobacterium tuberculosis, which intersect with Immunology. Bernhard Ryffel interconnects Fibrosis, Interleukin 33 and Lung in the investigation of issues within Inflammation.
Bernhard Ryffel combines subjects such as Cancer research, Microbiology and Virology with his study of Immune system. His Tumor necrosis factor alpha study integrates concerns from other disciplines, such as Mycobacterium bovis and Antibody. His Inflammasome study incorporates themes from Autophagy and Endocrinology.
Bernhard Ryffel focuses on Inflammation, Immunology, Immune system, Lung and Inflammasome. In general Inflammation, his work in Proinflammatory cytokine is often linked to Sting linking many areas of study. His research is interdisciplinary, bridging the disciplines of Mycobacterium tuberculosis and Immunology.
His research investigates the connection between Immune system and topics such as Microbiology that intersect with issues in Probiotic. His Lung research includes themes of COPD, Cancer research and Pathology. His Inflammasome research is multidisciplinary, relying on both Autophagy and Pharmacology.
His primary scientific interests are in Inflammation, Immunology, Inflammasome, Cell biology and Immune system. The concepts of his Inflammation study are interwoven with issues in Tumor necrosis factor alpha, Innate immune system, Downregulation and upregulation and Autophagy. His Immunology study frequently draws connections to other fields, such as Signal transduction.
Bernhard Ryffel has researched Inflammasome in several fields, including Regulator and Endocrinology. He combines subjects such as Caspase-Independent Apoptosis, Orphan receptor, Antigen and Escherichia coli with his study of Cell biology. His studies deal with areas such as Pancreas, Regulatory macrophages and Microbiology as well as Immune system.
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Gut microbiome influences efficacy of PD-1–based immunotherapy against epithelial tumors
Bertrand Routy;Bertrand Routy;Bertrand Routy;Lisa Derosa;Lisa Derosa;Lisa Derosa;Connie P. M. Duong;Connie P. M. Duong.
Activation of the NLRP3 inflammasome in dendritic cells induces IL-1β–dependent adaptive immunity against tumors
François Ghiringhelli;Lionel Apetoh;Antoine Tesniere;Antoine Tesniere;Antoine Tesniere;Laetitia Aymeric;Laetitia Aymeric;Laetitia Aymeric.
Nature Medicine (2009)
The intestinal microbiota modulates the anticancer immune effects of cyclophosphamide
Sophie Viaud;Sophie Viaud;Fabiana Saccheri;Grégoire Mignot;Takahiro Yamazaki.
Interleukin-17 is a negative regulator of established allergic asthma
Silvia Schnyder-Candrian;Dieudonnée Togbe;Isabelle Couillin;Isabelle Mercier.
Journal of Experimental Medicine (2006)
Membrane-associated Hsp72 from tumor-derived exosomes mediates STAT3-dependent immunosuppressive function of mouse and human myeloid-derived suppressor cells
Fanny Chalmin;Sylvain Ladoire;Grégoire Mignot;Julie Vincent.
Journal of Clinical Investigation (2010)
Alternating electric fields arrest cell proliferation in animal tumor models and human brain tumors
Eilon D. Kirson;Vladimír Dbalý;František Tovaryš;Josef Vymazal.
Proceedings of the National Academy of Sciences of the United States of America (2007)
Chemotherapy-triggered cathepsin B release in myeloid-derived suppressor cells activates the Nlrp3 inflammasome and promotes tumor growth
Mélanie Bruchard;Grégoire Mignot;Grégoire Mignot;Valentin Derangère;Valentin Derangère;Fanny Chalmin;Fanny Chalmin.
Nature Medicine (2013)
Toll-Like Receptor 4 Expression Is Required to Control Chronic Mycobacterium tuberculosis Infection in Mice
Brian Abel;Nathalie Thieblemont;Nathalie Thieblemont;Valerie J. F. Quesniaux;Najmeeyah Brown.
Journal of Immunology (2002)
NOD2-mediated dysbiosis predisposes mice to transmissible colitis and colorectal cancer
Aurélie Couturier-Maillard;Thomas Secher;Ateequr Rehman;Sylvain Normand.
Journal of Clinical Investigation (2013)
CARD9 impacts colitis by altering gut microbiota metabolism of tryptophan into aryl hydrocarbon receptor ligands
Bruno Lamas;Mathias L Richard;Valentin Leducq;Hang-Phuong Pham.
Nature Medicine (2016)
Profile was last updated on December 6th, 2021.
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