World's Best Scientists 2026 revealed!

D-Index & Metrics

Immunology

D-Index
51
Citations
10873
World Ranking
4134
National Ranking
1886

David D. Brand publication distribution in Immunology in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Immunology in 2026. The highlighted bar marks where David D. Brand sits on this spectrum.

62–71 publications: 9 scientists 72–81 publications: 23 scientists 82–91 publications: 40 scientists 92–101 publications: 72 scientists 102–111 publications: 86 scientists 112–121 publications: 108 scientists 122–131 publications: 150 scientists 132–141 publications: 160 scientists 142–151 publications: 159 scientists 152–161 publications: 189 scientists 162–171 publications: 166 scientists 172–181 publications: 181 scientists 182–191 publications: 188 scientists 192–201 publications: 187 scientists 202–211 publications: 178 scientists 212–221 publications: 158 scientists 222–231 publications: 168 scientists 232–241 publications: 159 scientists 242–251 publications: 160 scientists 252–261 publications: 148 scientists 262–271 publications: 116 scientists 272–281 publications: 125 scientists 282–291 publications: 115 scientists 292–301 publications: 111 scientists 302–311 publications: 95 scientists 312–321 publications: 97 scientists 322–331 publications: 97 scientists 332–341 publications: 99 scientists 342–351 publications: 96 scientists 352–361 publications: 69 scientists 362–371 publications: 76 scientists 372–381 publications: 71 scientists 382–391 publications: 71 scientists 392–401 publications: 62 scientists 402–411 publications: 54 scientists 412–421 publications: 41 scientists 422–431 publications: 63 scientists 432–441 publications: 53 scientists 442–451 publications: 31 scientists 452–461 publications: 42 scientists 462–471 publications: 40 scientists 472–481 publications: 29 scientists 482–491 publications: 34 scientists 492–501 publications: 30 scientists 502–511 publications: 23 scientists 512–521 publications: 41 scientists 522–531 publications: 29 scientists 532–541 publications: 28 scientists 542–551 publications: 16 scientists 552–561 publications: 18 scientists 562–571 publications: 18 scientists 572–581 publications: 17 scientists 582–591 publications: 17 scientists 592–601 publications: 17 scientists 602–611 publications: 19 scientists 612–621 publications: 13 scientists 622–631 publications: 12 scientists 632–641 publications: 11 scientists 642–651 publications: 16 scientists 652–661 publications: 9 scientists 662–671 publications: 5 scientists 672–681 publications: 13 scientists 682–691 publications: 12 scientists 692–701 publications: 6 scientists 702–711 publications: 6 scientists 712–721 publications: 9 scientists 722–731 publications: 9 scientists 732–741 publications: 6 scientists 742–751 publications: 11 scientists 752–761 publications: 10 scientists 762–771 publications: 7 scientists 772–781 publications: 12 scientists 782–791 publications: 7 scientists 792–801 publications: 5 scientists 802–806 publications: 1 scientists 807+ publications: 100 scientists
62 publications 807+

This scientist: 138 publications — 12th percentile

12% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 807 publications or more.

David D. Brand D-index placement in Immunology in 2026

The chart shows the D-index (discipline H-index) distribution of Immunology scientists ranked by Research.com in 2026. The highlighted bar marks where David D. Brand sits on this spectrum.

40–41 D-Index: 28 scientists 42–43 D-Index: 91 scientists 44–45 D-Index: 157 scientists 46–47 D-Index: 192 scientists 48–49 D-Index: 175 scientists 50–51 D-Index: 188 scientists 52–53 D-Index: 177 scientists 54–55 D-Index: 155 scientists 56–57 D-Index: 194 scientists 58–59 D-Index: 201 scientists 60–61 D-Index: 197 scientists 62–63 D-Index: 201 scientists 64–65 D-Index: 173 scientists 66–67 D-Index: 167 scientists 68–69 D-Index: 172 scientists 70–71 D-Index: 199 scientists 72–73 D-Index: 184 scientists 74–75 D-Index: 133 scientists 76–77 D-Index: 163 scientists 78–79 D-Index: 127 scientists 80–81 D-Index: 121 scientists 82–83 D-Index: 125 scientists 84–85 D-Index: 121 scientists 86–87 D-Index: 102 scientists 88–89 D-Index: 96 scientists 90–91 D-Index: 75 scientists 92–93 D-Index: 72 scientists 94–95 D-Index: 74 scientists 96–97 D-Index: 66 scientists 98–99 D-Index: 68 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 60 scientists 104–105 D-Index: 54 scientists 106–107 D-Index: 36 scientists 108–109 D-Index: 23 scientists 110–111 D-Index: 52 scientists 112–113 D-Index: 41 scientists 114–115 D-Index: 34 scientists 116–117 D-Index: 25 scientists 118–119 D-Index: 28 scientists 120–121 D-Index: 11 scientists 122–123 D-Index: 20 scientists 124–125 D-Index: 27 scientists 126–127 D-Index: 19 scientists 128–129 D-Index: 20 scientists 130–131 D-Index: 16 scientists 132–133 D-Index: 17 scientists 134–135 D-Index: 14 scientists 136–137 D-Index: 15 scientists 138–139 D-Index: 9 scientists 140–141 D-Index: 11 scientists 142–143 D-Index: 9 scientists 144–145 D-Index: 9 scientists 146–147 D-Index: 7 scientists 148–149 D-Index: 7 scientists 150–151 D-Index: 7 scientists 152–153 D-Index: 5 scientists 154–155 D-Index: 8 scientists 156 D-Index: 6 scientists 157+ D-Index: 96 scientists
40 D-Index 157+

This scientist: 51 D-Index — 17th percentile

17% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 157 D-Index or more.

Overview

David D. Brand is affiliated with the University of Tennessee Health Science Center in the United States. Their research primarily falls within the broad field of Medicine, with a focus on specialized subfields including Rheumatology, Surgery, Immunology, Molecular Biology, and Immunology and Allergy.

The scientist's work covers several main topics related to disease mechanisms and therapeutic approaches. These include:

  • Osteoarthritis Treatment and Mechanisms
  • Cell Adhesion Molecules Research
  • Monoclonal and Polyclonal Antibodies Research
  • Rheumatoid Arthritis Research and Therapies
  • Knee injuries and reconstruction techniques
  • Cancer-related molecular mechanisms research
  • Inflammasome and immune disorders

The publication record features contributions to various journals, with multiple papers appearing in the following venues:

  • Journal of Biological Chemistry
  • Frontiers in Immunology
  • International Journal of Molecular Sciences
  • bioRxiv (Cold Spring Harbor Laboratory)
  • PLoS ONE

Selected recent papers include:

  • "Heterogeneous ferroptosis susceptibility of macrophages caused by focal iron overload exacerbates rheumatoid arthritis," 2023, Redox Biology
  • "Leukocyte-associated immunoglobulin-like receptor 1 inhibits T-cell signaling by decreasing protein phosphorylation in the T-cell signaling pathway," 2020, Journal of Biological Chemistry
  • "Development of Tri-Layered Biomimetic Atelocollagen Scaffolds with Interfaces for Osteochondral Tissue Engineering," 2022, Advanced Healthcare Materials
  • "Infusion of GMSCs relieves autoimmune arthritis by suppressing the externalization of neutrophil extracellular traps via PGE2-PKA-ERK axis," 2023, Journal of Advanced Research
  • "Comparison of Phenotypic Characteristics and Prognosis Between Black and White Patients in a Tertiary ALS Clinic," 2020, Neurology

David D. Brand has collaborated frequently with several co-authors, reflecting interdisciplinary and collaborative research engagements. Frequent co-authors include:

  • Linda K. Myers
  • Edward F. Rosloniec
  • Karen A. Hasty
  • Song Guo Zheng
  • John Stuart

Best Publications

  • Collagen-induced arthritis

    David D Brand;David D Brand;Kary A Latham;Kary A Latham;Edward F Rosloniec;Edward F Rosloniec

  • Role of Vitamin A in the Immune System

    Zhiyi Huang;Yu Liu;Guangying Qi;David Brand

  • IL-17–dependent cellular immunity to collagen type V predisposes to obliterative bronchiolitis in human lung transplants

    William J. Burlingham;Robert B. Love;Robert B. Love;Ewa Jankowska-Gan;Lynn D. Haynes

  • Role of TNF-TNF Receptor 2 Signal in Regulatory T Cells and Its Therapeutic Implications

    Sujuan Yang;Julie Wang;David Douglass Brand;Song Guo Zheng

  • Immunopathogenesis of collagen arthritis.

    David D. Brand;Andrew H. Kang;Andrew H. Kang;Edward F. Rosloniec;Edward F. Rosloniec

  • An HLA-DR1 Transgene Confers Susceptibility to Collagen-induced Arthritis Elicited with Human Type II Collagen

    Edward F. Rosloniec;David D. Brand;Linda K. Myers;Karen B. Whittington

  • Cutting Edge: All-Trans Retinoic Acid Sustains the Stability and Function of Natural Regulatory T Cells in an Inflammatory Milieu

    Xiaohui Zhou;Ning Kong;Ning Kong;Julie Wang;Huiming Fan

  • Adoptive Transfer of Human Gingiva‐Derived Mesenchymal Stem Cells Ameliorates Collagen‐Induced Arthritis via Suppression of Th1 and Th17 Cells and Enhancement of Regulatory T Cell Differentiation

    Maogen Chen;Wenru Su;Xiaohong Lin;Zhiyong Guo

  • Critical role of all-trans retinoic acid in stabilizing human natural regulatory T cells under inflammatory conditions

    Ling Lu;Ling Lu;Qin Lan;Qin Lan;Zhiyuan Li;Xiaohui Zhou;Xiaohui Zhou

  • Cleavage of denatured natural collagen type II by neutrophil gelatinase B reveals enzyme specificity, post-translational modifications in the substrate, and the formation of remnant epitopes in rheumatoid arthritis

    Philippe Van den Steen;Paul Proost;Bernard Grillet;David D Brand

  • Role of SMAD and Non-SMAD Signals in the Development of Th17 and Regulatory T Cells

    Ling Lu;Julie Wang;Feng Zhang;Yang Chai

  • Anti-type V collagen lymphocytes that express IL-17 and IL-23 induce rejection pathology in fresh and well-healed lung transplants.

    S. Yoshida;A. Haque;T. Mizobuchi;T. Iwata

  • Collagen-induced arthritis.

    Edward F. Rosloniec;Michael Cremer;Andrew H. Kang;Linda K. Myers

  • Advances in distinguishing natural from induced Foxp3+ regulatory T cells

    Xiaohong Lin;Maogen Chen;Maogen Chen;Ya Liu;Zhiyong Guo

  • Induction of Autoimmune Arthritis in HLA-DR4 (DRB1*0401) Transgenic Mice by Immunization with Human and Bovine Type II Collagen

    Edward F. Rosloniec;David D. Brand;Linda K. Myers;Yukio Esaki

  • Antigen-specific transforming growth factor β–induced Treg cells, but not natural Treg cells, ameliorate autoimmune arthritis in mice by shifting the Th17/Treg cell balance from Th17 predominance to Treg cell predominance

    Ning Kong;Qin Lan;Qin Lan;Maogen Chen;Julie Wang

  • Targeting IL-2: an unexpected effect in treating immunological diseases.

    Congxiu Ye;David Brand;Song Guo Zheng;Song Guo Zheng

  • Porphyromonas gingivalis oral infection exacerbates the development and severity of collagen-induced arthritis

    Julie Teresa Marchesan;Elizabeth Ann Gerow;Riley Schaff;Andrei Dan Taut

  • All-trans retinoic acid promotes TGF-β-induced Tregs via histone modification but not DNA demethylation on Foxp3 gene locus.

    Ling Lu;Jilin Ma;Zhiyuan Li;Qin Lan;Qin Lan

  • Relevance of Posttranslational Modifications for the Arthritogenicity of Type II Collagen

    Linda K. Myers;Johanna Myllyharju;Minna Nokelainen;David D. Brand;David D. Brand

  • Role of Smad and non-Smad Signals in the Development of Th17 and Regulatory T Cells

    Song Guo Zheng;Ling Lu;Julie Wang;Feng Zhang

Frequent Co-Authors

Song Guo Zheng
Song Guo Zheng Shanghai Jiao Tong University
John M. Stuart
John M. Stuart University of Tennessee Health Science Center
Andrew H. Kang
Andrew H. Kang University of Tennessee Health Science Center
David A. Horwitz
David A. Horwitz University of Southern California
Bernhard Ryffel
Bernhard Ryffel University of Orléans
Wei Shi
Wei Shi University of Southern California
Karen A. Hasty
Karen A. Hasty University of Tennessee Health Science Center
Oscar W. Cummings
Oscar W. Cummings Indiana University
Bin Li
Bin Li Shanghai Jiao Tong University
Arnold E. Postlethwaite
Arnold E. Postlethwaite University of Tennessee Health Science Center

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Exploring these diverse educational pathways broadens career options and strengthens expertise relevant to Immunology, ensuring a solid footing in the healthcare field.

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