D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 51 Citations 9,082 108 World Ranking 2679 National Ranking 1285

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Antibody

His main research concerns Immunology, Arthritis, Type II collagen, Antibody and Immune system. His work in the fields of Immunology, such as Rheumatoid arthritis, Antigen, Cellular immunity and Cell mediated immunity, overlaps with other areas such as Peripheral blood mononuclear cell. His biological study spans a wide range of topics, including Type I collagen, Immunization, Molecular biology, Epitope and Cartilage.

His Type II collagen research includes elements of T cell, Polyarthritis, B cell and Pathology. In general Antibody study, his work on Autoimmunity, Immunoglobulin G and Autoantibody often relates to the realm of Sensorineural hearing loss, thereby connecting several areas of interest. His work carried out in the field of Autoimmunity brings together such families of science as Systemic lupus erythematosus, Molecular mimicry and Virology.

His most cited work include:

  • Serum transfer of collagen-induced arthritis in mice. (373 citations)
  • Induction of arthritis with monoclonal antibodies to collagen. (367 citations)
  • Type II collagen-induced arthritis in rats. Passive transfer with serum and evidence that IgG anticollagen antibodies can cause arthritis. (310 citations)

What are the main themes of his work throughout his whole career to date?

John M. Stuart spends much of his time researching Arthritis, Immunology, Type II collagen, T cell and Molecular biology. His Arthritis study improves the overall literature in Internal medicine. John M. Stuart has researched Type II collagen in several fields, including Epitope, Antibody, Immune system and Peptide.

His Antibody course of study focuses on Inflammatory arthritis and Cancer research. John M. Stuart interconnects Signal transduction, Cell biology, Recombinant DNA and Cytokine secretion in the investigation of issues within T cell. His research in Molecular biology intersects with topics in Cell, Gene expression, Mutant, Immunogenicity and Phosphorylation.

He most often published in these fields:

  • Arthritis (93.88%)
  • Immunology (60.54%)
  • Type II collagen (60.54%)

What were the highlights of his more recent work (between 2010-2020)?

  • Arthritis (93.88%)
  • Quantitative trait locus (22.45%)
  • Molecular biology (38.10%)

In recent papers he was focusing on the following fields of study:

John M. Stuart mainly focuses on Arthritis, Quantitative trait locus, Molecular biology, T cell and Genetics. Arthritis is a primary field of his research addressed under Immunology. While the research belongs to areas of Quantitative trait locus, John M. Stuart spends his time largely on the problem of Congenic, intersecting his research to questions surrounding Genome, Phenotype and Chromosome.

The Molecular biology study combines topics in areas such as Cell, Biochemistry, Enzyme, Vitamin C and Gene expression. The concepts of his T cell study are interwoven with issues in Internal medicine, Transgene and Cell biology. In his research on the topic of Internal medicine, Type II collagen and Immune tolerance is strongly related with Endocrinology.

Between 2010 and 2020, his most popular works were:

  • Detection of early cartilage damage using targeted nanosomes in a post-traumatic osteoarthritis mouse model. (28 citations)
  • Theranostic immunoliposomes for osteoarthritis (26 citations)
  • Dual-specificity phosphatases 2: surprising positive effect at the molecular level and a potential biomarker of diseases. (24 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Enzyme

His primary scientific interests are in Arthritis, Immunology, T-cell receptor, HLA-DR1 and Molecular biology. Within one scientific family, he focuses on topics pertaining to Inflammation under Arthritis, and may sometimes address concerns connected to Transgene, Rheumatology and Matrix metalloproteinase. His Immunology study frequently links to adjacent areas such as Internal medicine.

His research in Internal medicine is mostly concerned with Type II collagen. The various areas that John M. Stuart examines in his Type II collagen study include Cancer research and Antibody. His Molecular biology study incorporates themes from Genetics, Gene and Locus.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Serum transfer of collagen-induced arthritis in mice.

John M. Stuart;Frank J. Dixon.
Journal of Experimental Medicine (1983)

566 Citations

Induction of arthritis with monoclonal antibodies to collagen.

K Terato;K A Hasty;R A Reife;M A Cremer.
Journal of Immunology (1992)

564 Citations

Type II collagen-induced arthritis in rats. Passive transfer with serum and evidence that IgG anticollagen antibodies can cause arthritis.

J M Stuart;M A Cremer;A S Townes;A H Kang.
Journal of Experimental Medicine (1982)

474 Citations

Autoimmunity due to molecular mimicry as a cause of neurological disease

Michael C. Levin;Michael C. Levin;Sang Min Lee;Franck Kalume;Yvette Morcos.
Nature Medicine (2002)

309 Citations

An HLA-DR1 Transgene Confers Susceptibility to Collagen-induced Arthritis Elicited with Human Type II Collagen

Edward F. Rosloniec;David D. Brand;Linda K. Myers;Karen B. Whittington.
Journal of Experimental Medicine (1997)

286 Citations

The role of stromelysin in the cartilage destruction that accompanies inflammatory arthritis.

Karen A. Hasty;Robert A. Reife;Andrew H. Kang;John M. Stuart.
Arthritis & Rheumatism (1990)

267 Citations

Collagen-induced arthritis in rats

John M Stuart;Michael A Cremer;Andrew H Kang;Alexander S Townes.
Arthritis & Rheumatism (1979)

256 Citations

Nature and specificity of the immune response to collagen in type II collagen-induced arthritis in mice.

John M. Stuart;Alexander S. Townes;Andrew H. Kang.
Journal of Clinical Investigation (1982)

253 Citations

Collagen Autoimmune Arthritis

John M. Stuart;Alexander S. Townes;Andrew H. Kang.
Annual Review of Immunology (1984)

246 Citations

Incidence and specificity of antibodies to types I, II, III, IV, and V collagen in rheumatoid arthritis and other rheumatic diseases as measured by 125i‐radioimmunoassay

John M. Stuart;Eugene H. Huffstutter;Alexander S. Townes;Alexander S. Townes;Andrew H. Kang.
Arthritis & Rheumatism (1983)

214 Citations

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