2009 - Fellow of the American Academy of Arts and Sciences
2008 - Fellow of the American Association for the Advancement of Science (AAAS)
1998 - Member of the National Academy of Medicine (NAM)
Member of the Association of American Physicians
Her scientific interests lie mostly in Internal medicine, Cardiology, Cancer research, Cell biology and In vivo. Her Internal medicine study frequently draws connections to adjacent fields such as Endocrinology. Her work is connected to Coronary artery disease, Coronary arteries and Vasodilation, as a part of Cardiology.
Her Cancer research study integrates concerns from other disciplines, such as Cell growth, Progerin, LMNA, Anatomy and Progeria. Her In vivo research includes elements of Gene transfer, Liposome, Recombinant DNA, DNA and Pharmacology. The Recombinant DNA study combines topics in areas such as Molecular biology, Genetic transfer, Transfection and Pathology.
Her primary areas of investigation include Internal medicine, Cardiology, Cell biology, In vivo and Cancer research. Her Internal medicine study frequently draws connections between related disciplines such as Endocrinology. The various areas that Elizabeth G. Nabel examines in her Cardiology study include Impact factor and Asymptomatic, Surgery.
Her In vivo study incorporates themes from Gene transfer and Gene. Her Cancer research research is multidisciplinary, relying on both Cancer, Cell growth, Restenosis, Cyclin-dependent kinase and Immunology. Elizabeth G. Nabel has researched Vasodilation in several fields, including Vasoconstriction and Coronary arteries.
Elizabeth G. Nabel mainly focuses on Internal medicine, Pathology, Vascular disease, Lung and Gerontology. Her study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology, Oncology and Cardiology. The study incorporates disciplines such as Cancer research, Protein-Arginine N-Methyltransferases and Kinase in addition to Endocrinology.
Her studies in Pathology integrate themes in fields like Progerin, Umbilical vein, Progeria and Cellular senescence. She works mostly in the field of Vascular disease, limiting it down to concerns involving Myocardial infarction and, occasionally, MEDLINE, Coronary artery disease, Improved survival, Arteriolosclerosis and Stroke. Her Lung research incorporates themes from Progenitor cell, Progenitor, Reprogramming, Immunology and Intensive care medicine.
Her primary areas of study are Internal medicine, Myocardial infarction, Vascular disease, Cell aging and Pathology. Elizabeth G. Nabel combines subjects such as Tyrosine phosphorylation, Methylation and Cardiology with her study of Internal medicine. Her Cardiology study combines topics in areas such as Dementia, MEDLINE and Improved survival.
Her research in Vascular disease intersects with topics in Pulse wave velocity, Pulse pressure and Disease. Her research investigates the connection between Pathology and topics such as Plaque instability that intersect with issues in Mesenchymal stem cell. Her research integrates issues of Progerin, Progeria and Senescence in her study of Arteriolosclerosis.
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Coronary vasomotor response to acetylcholine relates to risk factors for coronary artery disease.
J A Vita;C B Treasure;E G Nabel;J M McLenachan.
Direct gene transfer with DNA-liposome complexes in melanoma: expression, biologic activity, and lack of toxicity in humans
Gary J. Nabel;Elizabeth G. Nabel;Zhi Yong Yang;Bernard A. Fox.
Proceedings of the National Academy of Sciences of the United States of America (1993)
A Tale of Coronary Artery Disease and Myocardial Infarction
Elizabeth G. Nabel;Eugene Braunwald.
The New England Journal of Medicine (2012)
Site-specific gene expression in vivo by direct gene transfer into the arterial wall.
Elizabeth G. Nabel;Gregory Plautz;Gary J. Nabel.
Treatment of diseases by site-specific instillation of cells or site-specific transformation of cells and kits therefor
Elizabeth G. Nabel;Gary J. Nabel.
Grand challenges in chronic non-communicable diseases
Abdallah S. Daar;Peter A. Singer;Deepa Leah Persad;Stig K. Pramming.
Heme oxygenase-1 protects against vascular constriction and proliferation.
Henricus J. Duckers;Manfred Boehm;Andrea L. True;Shaw Fang Yet.
Nature Medicine (2001)
Dilation of normal and constriction of atherosclerotic coronary arteries caused by the cold pressor test.
E G Nabel;P Ganz;J B Gordon;R W Alexander.
Circadian variation of transient myocardial ischemia in patients with coronary artery disease.
M B Rocco;J Barry;S Campbell;E Nabel.
Phenotype and Course of Hutchinson–Gilford Progeria Syndrome
Melissa A. Merideth;Leslie B. Gordon;Sarah Clauss;Vandana Sachdev.
The New England Journal of Medicine (2008)
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