D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 50 Citations 6,633 193 World Ranking 2749 National Ranking 6

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Internal medicine
  • Cytokine

Her main research concerns Immunology, Arthritis, Synovial fluid, Internal medicine and FOXP3. Many of her studies on Immunology apply to Systemic lupus erythematosus as well. Mi-La Cho combines subjects such as Cancer research, Cellular differentiation, Interleukin 17, Cytokine and RANKL with her study of Arthritis.

Her work focuses on many connections between Interleukin 17 and other disciplines, such as Molecular biology, that overlap with her field of interest in Protein kinase B. Within one scientific family, Mi-La Cho focuses on topics pertaining to Endocrinology under Internal medicine, and may sometimes address concerns connected to Angiogenesis, Interleukin 8 and Receptor. The various areas that Mi-La Cho examines in her FOXP3 study include Macrophage, Cytotoxic T cell, IL-2 receptor and Inflammatory arthritis.

Her most cited work include:

  • The catabolic pathway mediated by Toll-like receptors in human osteoarthritic chondrocytes. (195 citations)
  • Up-regulation of IL-23p19 expression in rheumatoid arthritis synovial fibroblasts by IL-17 through PI3-kinase-, NF-κB- and p38 MAPK-dependent signalling pathways (146 citations)
  • Interleukin-22 promotes osteoclastogenesis in rheumatoid arthritis through induction of RANKL in human synovial fibroblasts (135 citations)

What are the main themes of her work throughout her whole career to date?

Mi-La Cho spends much of her time researching Immunology, Arthritis, Proinflammatory cytokine, Inflammation and Cancer research. Her Immune system, Interleukin 17, FOXP3, Interleukin and T cell investigations are all subjects of Immunology research. The study incorporates disciplines such as Endocrinology, Rheumatoid arthritis and Cytokine in addition to Arthritis.

Her Proinflammatory cytokine research incorporates elements of Tumor necrosis factor alpha and Inflammatory bowel disease. Her work in Inflammation covers topics such as Pharmacology which are related to areas like Metformin and Cartilage. The various areas that she examines in her Cancer research study include Cellular differentiation, Osteoclast, Downregulation and upregulation, STAT3 and PI3K/AKT/mTOR pathway.

She most often published in these fields:

  • Immunology (53.29%)
  • Arthritis (34.54%)
  • Proinflammatory cytokine (22.70%)

What were the highlights of her more recent work (between 2017-2021)?

  • Inflammation (22.37%)
  • Cancer research (21.71%)
  • Arthritis (34.54%)

In recent papers she was focusing on the following fields of study:

Inflammation, Cancer research, Arthritis, Immunology and Pharmacology are her primary areas of study. She has included themes like Interleukin, Endocrinology and Cartilage in her Inflammation study. Her work deals with themes such as Tumor necrosis factor alpha, Germinal center, B cell and Interleukin 17, which intersect with Interleukin.

Her studies deal with areas such as T cell, STAT3, Cell therapy and FOXP3 as well as Cancer research. Her Arthritis study combines topics in areas such as Osteoclast, Rheumatoid arthritis and Cellular differentiation. Mi-La Cho focuses mostly in the field of Immunology, narrowing it down to topics relating to Cell and, in certain cases, Flow cytometry.

Between 2017 and 2021, her most popular works were:

  • Immunological pathogenesis of inflammatory bowel disease. (117 citations)
  • IL-17-mediated mitochondrial dysfunction impairs apoptosis in rheumatoid arthritis synovial fibroblasts through activation of autophagy. (69 citations)
  • Interleukin-10 produced by myeloid-derived suppressor cells is critical for the induction of Tregs and attenuation of rheumatoid inflammation in mice. (67 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The catabolic pathway mediated by Toll-like receptors in human osteoarthritic chondrocytes.

Hyun Ah Kim;Mi-La Cho;Hye Young Choi;Chang Sik Yoon.
Arthritis & Rheumatism (2006)

284 Citations

Up-regulation of IL-23p19 expression in rheumatoid arthritis synovial fibroblasts by IL-17 through PI3-kinase-, NF-κB- and p38 MAPK-dependent signalling pathways

H.-R. Kim;M.-L. Cho;K.-W. Kim;J.-Y. Juhn.
Rheumatology (2007)

219 Citations

Interleukin-22 promotes osteoclastogenesis in rheumatoid arthritis through induction of RANKL in human synovial fibroblasts

Kyoung-Woon Kim;Hae-Rim Kim;Jin-Young Park;Jin-Sil Park.
Arthritis & Rheumatism (2012)

207 Citations

Metformin Attenuates Experimental Autoimmune Arthritis through Reciprocal Regulation of Th17/Treg Balance and Osteoclastogenesis

Hye-Jin Son;Jennifer Lee;Seon-Yeong Lee;Eun-Kyung Kim.
Mediators of Inflammation (2014)

183 Citations

Metformin Ameliorates Inflammatory Bowel Disease by Suppression of the STAT3 Signaling Pathway and Regulation of the between Th17/Treg Balance.

Seon-Yeong Lee;Seung Hoon Lee;Eun-Ji Yang;Eun-Kyung Kim.
PLOS ONE (2015)

176 Citations

Immunological pathogenesis of inflammatory bowel disease.

Seung Hoon Lee;Jeong eun Kwon;Mi-La Cho.
Intestinal Research (2018)

174 Citations

Up-regulation of stromal cell–derived factor 1 (CXCL12) production in rheumatoid synovial fibroblasts through interactions with T lymphocytes: Role of interleukin-17 and CD40L–CD40 interaction

Kyoung-Woon Kim;Mi-La Cho;Hae-Rim Kim;Ji-Hyeon Ju.
Arthritis & Rheumatism (2007)

166 Citations

IL-10 suppresses Th17 cells and promotes regulatory T cells in the CD4+ T cell population of rheumatoid arthritis patients

Yu-Jung Heo;Young-Bin Joo;Hye-Jwa Oh;Mi-Kyung Park.
Immunology Letters (2010)

166 Citations

Increased interleukin-17 production via a phosphoinositide 3-kinase/Akt and nuclear factor κB-dependent pathway in patients with rheumatoid arthritis

Kyoung-Woon Kim;Mi-La Cho;Mi-Kyung Park;Chong-Hyeon Yoon.
Arthritis Research & Therapy (2004)

161 Citations

Enhanced T cell proliferative response to type II collagen and synthetic peptide CII (255-274) in patients with rheumatoid arthritis.

Ho-Youn Kim;Wan-Uk Kim;Mi-La Cho;Suk Kyeong Lee.
Arthritis & Rheumatism (1999)

145 Citations

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