D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 49 Citations 7,571 202 World Ranking 13747 National Ranking 157

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Apoptosis

Do Sik Min mainly investigates Molecular biology, Cell biology, Phospholipase D, Biochemistry and Signal transduction. His study in Molecular biology is interdisciplinary in nature, drawing from both Transfection, Proto-Oncogene Proteins p21, Intracellular, Kinase and Resveratrol. His Cell biology study incorporates themes from Mutation, Apoptosis, Ubiquitin ligase and Carcinogenesis.

Do Sik Min studies Phospholipase D, focusing on Phospholipase D1 in particular. He has researched Signal transduction in several fields, including PLD2, Cancer research and Growth factor. His Cancer research research includes elements of Cell, Cancer stem cell, Metastasis and Colorectal cancer, Adenomatous polyposis coli.

His most cited work include:

  • Molecular mechanisms of curcumin-induced cytotoxicity: induction of apoptosis through generation of reactive oxygen species, down-regulation of Bcl-XL and IAP, the release of cytochrome c and inhibition of Akt (348 citations)
  • Phospholipase C isozymes selectively couple to specific neurotransmitter receptors (256 citations)
  • Resveratrol inhibits phorbol myristate acetate-induced matrix metalloproteinase-9 expression by inhibiting JNK and PKC δ signal transduction (183 citations)

What are the main themes of his work throughout his whole career to date?

Do Sik Min mainly investigates Cell biology, Phospholipase D, Molecular biology, Cancer research and Phospholipase D1. His Cell biology research integrates issues from Endocrinology, Biochemistry and Cellular differentiation. His Phospholipase D study combines topics from a wide range of disciplines, such as Cell growth, PLD2, Phosphatidic acid, Internal medicine and Isozyme.

He interconnects Phospholipase, Transfection, Phospholipase C, G protein and Kinase in the investigation of issues within Molecular biology. The study incorporates disciplines such as Carcinogenesis, Cancer, Colorectal cancer, Wnt signaling pathway and Downregulation and upregulation in addition to Cancer research. His work in Phospholipase D1 tackles topics such as Western blot which are related to areas like Immunohistochemistry and Blot.

He most often published in these fields:

  • Cell biology (42.98%)
  • Phospholipase D (34.47%)
  • Molecular biology (31.06%)

What were the highlights of his more recent work (between 2014-2021)?

  • Cancer research (37.02%)
  • Cell biology (42.98%)
  • Wnt signaling pathway (20.00%)

In recent papers he was focusing on the following fields of study:

Do Sik Min mainly focuses on Cancer research, Cell biology, Wnt signaling pathway, Catenin and Signal transduction. His Cancer research research is multidisciplinary, relying on both Carcinogenesis, Cancer, Colorectal cancer, Downregulation and upregulation and Phospholipase D1. His Cell biology research incorporates themes from Biochemistry, Cellular differentiation and Osteoblast.

Many of his research projects under Biochemistry are closely connected to Translocator protein and Ro5-4864 with Translocator protein and Ro5-4864, tying the diverse disciplines of science together. His work investigates the relationship between Signal transduction and topics such as Phosphorylation that intersect with problems in Oncolytic virus. His studies in Beta-catenin integrate themes in fields like HEK 293 cells and Molecular biology.

Between 2014 and 2021, his most popular works were:

  • Small-molecule binding of the axin RGS domain promotes β-catenin and Ras degradation (51 citations)
  • Small-molecule binding of the axin RGS domain promotes β-catenin and Ras degradation (51 citations)
  • Targeting phospholipase D1 attenuates intestinal tumorigenesis by controlling β-catenin signaling in cancer-initiating cells (33 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Apoptosis

His main research concerns Cell biology, Wnt signaling pathway, Catenin, Cancer research and Beta-catenin. His biological study spans a wide range of topics, including Inflammasome, Biochemistry and Cellular differentiation. Do Sik Min combines subjects such as Molecular biology, Signal transduction, Proto-Oncogene Proteins p21 and Axin Protein with his study of Cancer research.

Do Sik Min has researched Molecular biology in several fields, including Carcinogenesis, Azoxymethane, microRNA, Small molecule binding and Anti-apoptotic Ras signalling cascade. His research investigates the link between Beta-catenin and topics such as HEK 293 cells that cross with problems in Phospholipase D1. His Phospholipase D1 research is under the purview of Phospholipase D.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Molecular mechanisms of curcumin-induced cytotoxicity: induction of apoptosis through generation of reactive oxygen species, down-regulation of Bcl-XL and IAP, the release of cytochrome c and inhibition of Akt

Ju Hyung Woo;Young Ho Kim;Yun Jung Choi;Dae Gon Kim.
Carcinogenesis (2003)

581 Citations

Phospholipase C isozymes selectively couple to specific neurotransmitter receptors

Daesoo Kim;Ki Sun Jun;Seong Beom Lee;Nae Gyu Kang.
Nature (1997)

352 Citations

Resveratrol inhibits phorbol myristate acetate-induced matrix metalloproteinase-9 expression by inhibiting JNK and PKC δ signal transduction

Ju Hyung Woo;Jun Hee Lim;Young Ho Kim;Seong Il Suh.
Oncogene (2004)

253 Citations

Involvement of tyrosine phosphorylation and protein kinase C in the activation of phospholipase D by H2O2 in Swiss 3T3 fibroblasts.

Do Sik Min;Eung Gook Kim;John H. Exton.
Journal of Biological Chemistry (1998)

224 Citations

Protective effects of epicatechin against the toxic effects of streptozotocin on rat pancreatic islets: In vivo and in vitro

Myung Jun Kim;Gyeong Ryul Ryu;Ji Sung Chung;Sang Soo Sim.
Pancreas (2003)

183 Citations

Role of Oncogenic K-Ras in Cancer Stem Cell Activation by Aberrant Wnt/β-Catenin Signaling

Byoung San Moon;Woo Jeong Jeong;Jieun Park;Tae Il Kim.
Journal of the National Cancer Institute (2014)

172 Citations

α-Synuclein Interacts with Phospholipase D Isozymes and Inhibits Pervanadate-induced Phospholipase D Activation in Human Embryonic Kidney-293 Cells

Bong-Hyun Ahn;Hyangshuk Rhim;Shi Yeon Kim;Young-Mo Sung.
Journal of Biological Chemistry (2002)

172 Citations

Bcl-2 overexpression attenuates resveratrol-induced apoptosis in U937 cells by inhibition of caspase-3 activity

Jong Wook Park;Yun Jung Choi;Seong Il Suh;Won Ki Baek.
Carcinogenesis (2001)

169 Citations

Overexpression of phospholipase D enhances matrix metalloproteinase-2 expression and glioma cell invasion via protein kinase C and protein kinase A/NF-κB/Sp1-mediated signaling pathways

Mi Hee Park;Bong Hyun Ahn;Yong Kil Hong;Do Sik Min.
Carcinogenesis (2009)

139 Citations

Transmodulation between phospholipase D and c-Src enhances cell proliferation.

Bong Hyun Ahn;Shi Yeon Kim;Eun Hee Kim;Kyeong Sook Choi.
Molecular and Cellular Biology (2003)

135 Citations

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