What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include FOXP3, Cell biology, Transcription factor, Cancer research and Immunology.
His FOXP3 study is concerned with the larger field of Immune system.
His study in Cell biology is interdisciplinary in nature, drawing from both Molecular biology, Histone, Acetylation and Transcriptional regulation.
His Molecular biology study incorporates themes from Spindle apparatus, Cellular differentiation, Histone code, Histone H2A and Histone H1.
He interconnects Fertilizer, Nitrate and Arabidopsis in the investigation of issues within Transcription factor.
His Cancer research study combines topics in areas such as T cell, miR-214, Signal transduction, Regulation of gene expression and Effector.
His most cited work include:
- Deacetylase inhibition promotes the generation and function of regulatory T cells (696 citations)
- FOXP3 interactions with histone acetyltransferase and class II histone deacetylases are required for repression (337 citations)
- PAMAM nanoparticles promote acute lung injury by inducing autophagic cell death through the Akt-TSC2-mTOR signaling pathway. (206 citations)
What are the main themes of his work throughout his whole career to date?
Bin Li mainly investigates Cell biology, FOXP3, Cancer research, Immune system and Immunology.
The various areas that Bin Li examines in his Cell biology study include HEK 293 cells, Molecular biology, Ubiquitin and Cellular differentiation.
His biological study spans a wide range of topics, including Regulatory T cell, IL-2 receptor, Transcription factor, Function and Autoimmunity.
His research in Cancer research intersects with topics in Gene knockdown, Cell growth, Carcinogenesis, Downregulation and upregulation and Immunotherapy.
Immune tolerance and T cell are among the areas of Immune system where Bin Li concentrates his study.
His Cancer research extends to Immunology, which is thematically connected.
He most often published in these fields:
- Cell biology (23.33%)
- FOXP3 (21.34%)
- Cancer research (19.85%)
What were the highlights of his more recent work (between 2019-2021)?
- Cancer research (19.85%)
- Immune system (16.13%)
- FOXP3 (21.34%)
In recent papers he was focusing on the following fields of study:
Bin Li focuses on Cancer research, Immune system, FOXP3, Cell biology and Immunology.
His Cancer research research includes themes of Gene knockdown, Cell growth, Carcinogenesis, Downregulation and upregulation and In vivo.
His Regulatory T cell study in the realm of Immune system connects with subjects such as Schistosoma Japonicum Infection.
His Regulatory T cell study integrates concerns from other disciplines, such as Cytokine and Immune tolerance.
His studies in FOXP3 integrate themes in fields like Inflammation, Transcription factor and Function.
The study of Cell biology is intertwined with the study of T-cell receptor in a number of ways.
Between 2019 and 2021, his most popular works were:
- Nonstructural protein 6 of porcine epidemic diarrhea virus induces autophagy to promote viral replication via the PI3K/Akt/mTOR axis (7 citations)
- RNA-binding protein Musashi2 stabilizing androgen receptor drives prostate cancer progression (7 citations)
- Biallelic mutations in CDC20 cause female infertility characterized by abnormalities in oocyte maturation and early embryonic development (6 citations)
In his most recent research, the most cited papers focused on:
Cancer research, Cell biology, Mutation, FOXP3 and Immune system are his primary areas of study.
Bin Li combines subjects such as Transcriptional activity, Messenger RNA, Cell growth and Gene knockdown with his study of Cancer research.
His Cell biology research incorporates elements of Ubiquitin and Transcription factor.
His study looks at the relationship between Transcription factor and topics such as Deubiquitination, which overlap with Inflammation.
FOXP3 and Regulatory T cell are frequently intertwined in his study.
His work deals with themes such as Autophagy and Survival rate, which intersect with Immune system.
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