Markus M. Heimesaat spends much of his time researching Immunology, Microbiology, Ileitis, Inflammation and Immune system. In most of his Immunology studies, his work intersects topics such as Inflammatory bowel disease. His Microbiology research incorporates elements of Microbiome, Organism, Gnotobiotic animal, Campylobacter jejuni and Prevotella.
His Campylobacter jejuni research includes themes of Colonisation resistance, Campylobacter and Enterocolitis. The Inflammation study combines topics in areas such as Matrix metalloproteinase, Viral cardiomyopathy and Cytokine. When carried out as part of a general Immune system research project, his work on Toll-like receptor, Gut–brain axis and Host immunity is frequently linked to work in Scientific field, therefore connecting diverse disciplines of study.
His primary areas of investigation include Microbiology, Immunology, Immune system, Campylobacter jejuni and Inflammation. His work carried out in the field of Microbiology brings together such families of science as Host–pathogen interaction, Virulence and Escherichia coli. Markus M. Heimesaat performs integrative study on Immunology and Ileitis.
Immune system connects with themes related to Tumor necrosis factor alpha in his study. His studies in Campylobacter jejuni integrate themes in fields like Apoptosis, Campylobacter and Enterocolitis. His Toll-like receptor research is multidisciplinary, relying on both Lipopolysaccharide and TLR4.
The scientist’s investigation covers issues in Microbiology, Immune system, Campylobacter jejuni, Campylobacteriosis and Antibiotics. His Microbiology research includes elements of Host–pathogen interaction, Virulence, Otitis and Staphylococcus aureus. His study explores the link between Immune system and topics such as Campylobacter coli that cross with problems in Gastrointestinal tract and Interleukin 10.
The study incorporates disciplines such as Secretion, Enterocolitis, Immunology and Resveratrol in addition to Campylobacter jejuni. His study in Gut flora and Inflammation falls within the category of Immunology. His biological study spans a wide range of topics, including Pathogen, Colonisation resistance, Toll-like receptor and Cytokine secretion.
His main research concerns Campylobacter jejuni, Microbiology, Immune system, Campylobacteriosis and Enterocolitis. His study ties his expertise on Immunology together with the subject of Campylobacter jejuni. Markus M. Heimesaat combines subjects such as Host–pathogen interaction and Virulence with his study of Microbiology.
His research on Immune system frequently connects to adjacent areas such as Campylobacter. As a member of one scientific family, Markus M. Heimesaat mostly works in the field of Campylobacteriosis, focusing on Toll-like receptor and, on occasion, Enteritis, Gastrointestinal tract, Interleukin 10 and Immunopathology. His studies in Campylobacter Jejuni Infection integrate themes in fields like Innate immune system, Colonisation resistance and Gut flora.
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A guide to histomorphological evaluation of intestinal inflammation in mouse models
Ulrike Erben;Christoph Loddenkemper;Katja Doerfel;Simone Spieckermann.
International Journal of Clinical and Experimental Pathology (2014)
Gram-Negative Bacteria Aggravate Murine Small Intestinal Th1-Type Immunopathology following Oral Infection with Toxoplasma gondii
Markus M. Heimesaat;Stefan Bereswill;André Fischer;David Fuchs.
Journal of Immunology (2006)
Ly6Chi Monocytes Provide a Link between Antibiotic-Induced Changes in Gut Microbiota and Adult Hippocampal Neurogenesis
Luisa Möhle;Daniele Mattei;Markus M. Heimesaat;Stefan Bereswill.
Cell Reports (2016)
Anti-inflammatory effects of resveratrol, curcumin and simvastatin in acute small intestinal inflammation
Stefan Bereswill;Melba Muñoz;André Fischer;Rita Plickert.
PLOS ONE (2010)
Interleukin (IL)-23 mediates Toxoplasma gondii-induced immunopathology in the gut via matrixmetalloproteinase-2 and IL-22 but independent of IL-17.
Melba Muñoz;Markus M. Heimesaat;Kerstin Danker;Daniela Struck.
Journal of Experimental Medicine (2009)
The NLRP3 inflammasome is differentially activated by pneumolysin variants and contributes to host defense in pneumococcal pneumonia.
Martin Witzenrath;Florence Pache;Daniel Lorenz;Uwe Koppe.
Journal of Immunology (2011)
Shift towards pro-inflammatory intestinal bacteria aggravates acute murine colitis via Toll-like receptors 2 and 4.
Markus M. Heimesaat;André Fischer;Britta Siegmund;Andreas Kupz.
PLOS ONE (2007)
Novel Murine Infection Models Provide Deep Insights into the “Ménage à Trois” of Campylobacter jejuni, Microbiota and Host Innate Immunity
Stefan Bereswill;André Fischer;Rita Plickert;Lea-Maxie Haag.
PLOS ONE (2011)
MyD88/TLR9 mediated immunopathology and gut microbiota dynamics in a novel murine model of intestinal graft-versus-host disease
Markus M Heimesaat;Axel Nogai;Stefan Bereswill;Rita Plickert.
Toll-like receptor and IL-12 signaling control susceptibility to contact hypersensitivity
Stefan F. Martin;Jan C. Dudda;Eva Bachtanian;Annalisa Lembo.
Journal of Experimental Medicine (2008)
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