His primary areas of study are Microbiology, Immunology, Helicobacter pylori, Ileitis and Mutant. His Microbiology research incorporates themes from Repressor, Prevotella, Regulation of gene expression, Superoxide and Virulence. His biological study deals with issues like Inflammatory bowel disease, which deal with fields such as Colitis and Cecum.
The study incorporates disciplines such as NFKB1, Secretion, Gene cluster, Gastric mucosa and Urease in addition to Helicobacter pylori. His Mutant research focuses on subjects like Bacteria, which are linked to Ferritin. Stefan Bereswill works mostly in the field of Gut flora, limiting it down to concerns involving Immunopathology and, occasionally, Transplantation, Graft-versus-host disease, Colonisation resistance and Campylobacter jejuni.
Stefan Bereswill mostly deals with Microbiology, Immunology, Immune system, Campylobacter jejuni and Helicobacter pylori. His study explores the link between Microbiology and topics such as Mutant that cross with problems in Bacteria. His work in the fields of Inflammation, Innate immune system, Immunopathology and Gut flora overlaps with other areas such as Ileitis.
His Immune system research includes themes of Lamina propria, Spleen and Arcobacter butzleri. His Campylobacter jejuni research integrates issues from Interleukin 22, Apoptosis, Interleukin 18 and Campylobacter. Secretion is closely connected to Virulence in his research, which is encompassed under the umbrella topic of Helicobacter pylori.
Stefan Bereswill mainly investigates Microbiology, Immune system, Campylobacter jejuni, Immunology and Campylobacter Jejuni Infection. His studies in Microbiology integrate themes in fields like Tumor necrosis factor alpha, Lamina propria, Human microbiome, Pseudomonas aeruginosa and Spleen. The Immune system study combines topics in areas such as Intestinal mucosa, Cytokine and Ileum.
His research in Campylobacter jejuni intersects with topics in Apoptosis, Tight junction and NOD2. His Immunology research is multidisciplinary, relying on both Probiotic and Antibiotics. His Campylobacter Jejuni Infection study also includes
His main research concerns Immunology, Microbiology, Antibiotics, Immune system and Tumor necrosis factor alpha. His work in the fields of Immunology, such as FOXP3, Dust mites and Allergy, overlaps with other areas such as Mechanism and Regulatory T cell. His Microbiology study combines topics in areas such as Probiotic, CD8, Spleen, Cytokine secretion and Ileum.
His research investigates the connection between Antibiotics and topics such as Gut flora that intersect with issues in Antibody, Neurogenesis, Knockout mouse and Adoptive cell transfer. Stefan Bereswill merges Immune system with Ileitis in his study. His Tumor necrosis factor alpha research is multidisciplinary, incorporating perspectives in Inflammation, Apoptosis, Intestinal epithelium, Campylobacter jejuni and Tight junction.
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Gram-Negative Bacteria Aggravate Murine Small Intestinal Th1-Type Immunopathology following Oral Infection with Toxoplasma gondii
Markus M. Heimesaat;Stefan Bereswill;André Fischer;David Fuchs.
Journal of Immunology (2006)
Anti-inflammatory effects of resveratrol, curcumin and simvastatin in acute small intestinal inflammation
Stefan Bereswill;Melba Muñoz;André Fischer;Rita Plickert.
PLOS ONE (2010)
Ly6Chi Monocytes Provide a Link between Antibiotic-Induced Changes in Gut Microbiota and Adult Hippocampal Neurogenesis
Luisa Möhle;Daniele Mattei;Markus M. Heimesaat;Stefan Bereswill.
Cell Reports (2016)
Interleukin (IL)-23 mediates Toxoplasma gondii-induced immunopathology in the gut via matrixmetalloproteinase-2 and IL-22 but independent of IL-17.
Melba Muñoz;Markus M. Heimesaat;Kerstin Danker;Daniela Struck.
Journal of Experimental Medicine (2009)
Preventive antibacterial therapy in acute ischemic stroke: a randomized controlled trial.
Hendrik Harms;Konstantin Prass;Christian Meisel;Juliane Klehmet.
PLOS ONE (2008)
The NLRP3 inflammasome is differentially activated by pneumolysin variants and contributes to host defense in pneumococcal pneumonia.
Martin Witzenrath;Florence Pache;Daniel Lorenz;Uwe Koppe.
Journal of Immunology (2011)
Shift towards pro-inflammatory intestinal bacteria aggravates acute murine colitis via Toll-like receptors 2 and 4.
Markus M. Heimesaat;André Fischer;Britta Siegmund;Andreas Kupz.
PLOS ONE (2007)
Novel Murine Infection Models Provide Deep Insights into the “Ménage à Trois” of Campylobacter jejuni, Microbiota and Host Innate Immunity
Stefan Bereswill;André Fischer;Rita Plickert;Lea-Maxie Haag.
PLOS ONE (2011)
MyD88/TLR9 mediated immunopathology and gut microbiota dynamics in a novel murine model of intestinal graft-versus-host disease
Markus M Heimesaat;Axel Nogai;Stefan Bereswill;Rita Plickert.
Proteins encoded by the cag pathogenicity island of Helicobacter pylori are required for NF-kappaB activation.
Erik Glocker;Christina Lange;Antonello Covacci;Stefan Bereswill.
Infection and Immunity (1998)
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