D-Index & Metrics Best Publications
Jose-Carlos Gutierrez-Ramos

Jose-Carlos Gutierrez-Ramos

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 55 Citations 17,185 80 World Ranking 2217 National Ranking 1093

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

Jose-Carlos Gutierrez-Ramos focuses on Immunology, Chemokine, T cell, Eosinophil and Eotaxin. His Immunology research focuses on Inflammation, Monocyte, Adoptive cell transfer, Eosinophilia and Immune system. His studies deal with areas such as Homing, Chemotaxis, Neutrophil clearance and Cell biology as well as Chemokine.

The various areas that Jose-Carlos Gutierrez-Ramos examines in his T cell study include Interleukin 2 and Antigen. His biological study spans a wide range of topics, including Monocyte Chemoattractant Proteins, Allergic inflammation and Macrophage inflammatory protein. His Eotaxin research integrates issues from CCR3 and Beta Chemokine.

His most cited work include:

  • A major role for VCAM-1, but not ICAM-1, in early atherosclerosis (973 citations)
  • Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils. (683 citations)
  • Intercellular adhesion molecule-1-deficient mice are protected against ischemic renal injury. (637 citations)

What are the main themes of his work throughout his whole career to date?

Jose-Carlos Gutierrez-Ramos mainly investigates Immunology, Cell biology, Chemokine, Inflammation and T cell. His is doing research in Immune system, Eotaxin, Sensitization, CD28 and Antigen, both of which are found in Immunology. His study in Cell biology is interdisciplinary in nature, drawing from both Cytokine and In vivo.

As part of one scientific family, Jose-Carlos Gutierrez-Ramos deals mainly with the area of Chemokine, narrowing it down to issues related to the Chemotaxis, and often Monocyte and Extravasation. His Inflammation study combines topics from a wide range of disciplines, such as Immunoglobulin E, House dust mite, Immunopathology, Pathology and Lung. The T cell study combines topics in areas such as T lymphocyte, CD154, Cyclosporin a, Interleukin 2 and Cytotoxic T cell.

He most often published in these fields:

  • Immunology (69.16%)
  • Cell biology (28.04%)
  • Chemokine (21.50%)

What were the highlights of his more recent work (between 2002-2012)?

  • Immunology (69.16%)
  • Cell biology (28.04%)
  • Immune system (14.02%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Immunology, Cell biology, Immune system, Inflammation and Chemokine. All of his Immunology and T cell, CC chemokine receptors, CCL20, CCL5 and Ovalbumin investigations are sub-components of the entire Immunology study. His Cell biology research incorporates elements of CXC chemokine receptors, Cytokine, In vivo and Bone marrow.

Many of his research projects under Immune system are closely connected to Context with Context, tying the diverse disciplines of science together. As part of the same scientific family, Jose-Carlos Gutierrez-Ramos usually focuses on Inflammation, concentrating on House dust mite and intersecting with Pathology and Aeroallergen. Jose-Carlos Gutierrez-Ramos interconnects Homing and Neutrophil clearance in the investigation of issues within Chemokine.

Between 2002 and 2012, his most popular works were:

  • Tim-3 inhibits T helper type 1-mediated auto- and alloimmune responses and promotes immunological tolerance (549 citations)
  • Chemokines Acting via CXCR2 and CXCR4 Control the Release of Neutrophils from the Bone Marrow and Their Return following Senescence (486 citations)
  • The proteasome of Mycobacterium tuberculosis is required for resistance to nitric oxide. (421 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Cytokine

His main research concerns Immunology, Immune system, Inflammation, T cell and House dust mite. Immunology is often connected to Cell biology in his work. His work in the fields of Immune system, such as IL-2 receptor, intersects with other areas such as Context.

The various areas that he examines in his Inflammation study include Aeroallergen, Pathology, Antigen, Allergy and Nasal administration. His T cell study combines topics from a wide range of disciplines, such as Interleukin 2, Virus genetics and Transplantation. Much of his study explores House dust mite relationship to Ovalbumin.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

A major role for VCAM-1, but not ICAM-1, in early atherosclerosis

Myron I. Cybulsky;Kaeko Iiyama;Hongmei Li;Suning Zhu.
Journal of Clinical Investigation (2001)

1530 Citations

Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils.

Paul D. Ponath;Shixin Qin;Douglas J. Ringler;Ian Clark-Lewis.
Journal of Clinical Investigation (1996)

863 Citations

T1/ST2 is preferentially expressed on murine Th2 cells, independent of interleukin 4, interleukin 5, and interleukin 10, and important for Th2 effector function

Max Löhning;Arne Stroehmann;Anthony J. Coyle;Jane L. Grogan.
Proceedings of the National Academy of Sciences of the United States of America (1998)

820 Citations

Intercellular adhesion molecule-1-deficient mice are protected against ischemic renal injury.

K. J. Kelly;Winfred W. Williams;Robert B. Colvin;Shane M. Meehan.
Journal of Clinical Investigation (1996)

808 Citations

Tim-3 inhibits T helper type 1-mediated auto- and alloimmune responses and promotes immunological tolerance

Alberto Sánchez-Fueyo;Jane Tian;Dominic Picarella;Christoph Domenig.
Nature Immunology (2003)

793 Citations

RANTES and monocyte chemoattractant protein-1 (MCP-1) play an important role in the inflammatory phase of crescentic nephritis, but only MCP-1 is involved in crescent formation and interstitial fibrosis.

Clare M. Lloyd;Andrew W. Minto;Martin E. Dorf;Amanda Proudfoot.
Journal of Experimental Medicine (1997)

739 Citations

Neurotactin, a membrane-anchored chemokine upregulated in brain inflammation

Yang Pan;Clare Lloyd;Hong Zhou;Sylvia Dolich.
Nature (1997)

723 Citations

The expanding B7 superfamily: Increasing complexity in costimulatory signals regulating T cell function

Anthony J. Coyle;Jose-Carlos Gutierrez-Ramos.
Nature Immunology (2001)

698 Citations

The Coordinated Action of CC Chemokines in the Lung Orchestrates Allergic Inflammation and Airway Hyperresponsiveness

Jose-Angel Gonzalo;Clare M. Lloyd;Danyi Wen;Juan P. Albar.
Journal of Experimental Medicine (1998)

654 Citations

Chemokines Acting via CXCR2 and CXCR4 Control the Release of Neutrophils from the Bone Marrow and Their Return following Senescence

Coralie Martin;Peter C E Burdon;Gary Bridger;Jose Carlos Gutierrez-Ramos.
Immunity (2003)

633 Citations

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