D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 40 Citations 5,891 119 World Ranking 3655 National Ranking 99

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Internal medicine

Martin R. Stämpfli spends much of his time researching Immunology, Inflammation, Immune system, Neutrophilia and Bronchoalveolar lavage. His COPD research extends to Immunology, which is thematically connected. His Inflammation research includes elements of Tumor necrosis factor alpha, Exacerbation, Influenza A virus and Lung.

His Lung research incorporates themes from Matrix metalloproteinase, Transforming growth factor, Extracellular matrix, Cell biology and Fibrosis. His Immune system study frequently links to adjacent areas such as Cytotoxic T cell. His research integrates issues of Chemokine and Dose–response relationship in his study of Bronchoalveolar lavage.

His most cited work include:

  • How cigarette smoke skews immune responses to promote infection, lung disease and cancer (439 citations)
  • Smad3 Null Mice Develop Airspace Enlargement and Are Resistant to TGF-β-Mediated Pulmonary Fibrosis (316 citations)
  • GM-CSF transgene expression in the airway allows aerosolized ovalbumin to induce allergic sensitization in mice. (251 citations)

What are the main themes of his work throughout his whole career to date?

Immunology, Inflammation, Lung, Immune system and COPD are his primary areas of study. His Antibody, Immunoglobulin E, Eosinophilia, Antigen and Ovalbumin study are his primary interests in Immunology. His Inflammation research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha, Pneumonia, Allergy, Neutrophilia and Bronchoalveolar lavage.

Martin R. Stämpfli works mostly in the field of Lung, limiting it down to concerns involving Pathology and, occasionally, Perfusion, Breathing and Peripheral blood mononuclear cell. His work deals with themes such as House dust mite and Sensitization, which intersect with Immune system. His COPD research includes themes of Exacerbation, Sputum, Haemophilus influenzae and Pathogenesis.

He most often published in these fields:

  • Immunology (79.26%)
  • Inflammation (29.26%)
  • Lung (26.06%)

What were the highlights of his more recent work (between 2017-2021)?

  • Immunology (79.26%)
  • COPD (23.94%)
  • Sputum (8.51%)

In recent papers he was focusing on the following fields of study:

His scientific interests lie mostly in Immunology, COPD, Sputum, Lung and Pulmonary disease. His work is connected to Immune system and Inflammation, as a part of Immunology. The concepts of his COPD study are interwoven with issues in Respiratory disease, Transcriptome, Tissue homeostasis and Gene.

His Sputum research also works with subjects such as

  • Antigen, Thyroid peroxidase, Interstitial lung disease and Cohort most often made with reference to Autoantibody,
  • Microbiome and related Moraxella catarrhalis, Haemophilus influenzae and Metagenomics. His studies deal with areas such as Asthma and Multidrug resistance-associated protein 2 as well as Lung. The various areas that Martin R. Stämpfli examines in his Pulmonary disease study include Macrophage, Respiratory system and Cardiology.

Between 2017 and 2021, his most popular works were:

  • Optimising experimental research in respiratory diseases: an ERS statement. (42 citations)
  • Female sex and gender in lung/sleep health and disease: Increased understanding of basic biological, pathophysiological, and behavioral mechanisms leading to better health for female patients with lung disease (31 citations)
  • Transcriptomic and barrier responses of human airway epithelial cells exposed to cannabis smoke (16 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

Martin R. Stämpfli mostly deals with Immunology, Transcriptome, COPD, Lung and Oxidative stress. His study in Immune system and T cell falls within the category of Immunology. Martin R. Stämpfli has included themes like Epithelium, Interleukin 8, Cytokine, CXCL10 and Pharmacology in his Transcriptome study.

His work carried out in the field of COPD brings together such families of science as Gene signature, Gene expression, Gene and Pathogenesis. His Lymphangioleiomyomatosis study in the realm of Lung connects with subjects such as Behavioural sciences. His Oxidative stress study combines topics in areas such as Barrier function, Respiratory health, Human airway and Cannabis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

How cigarette smoke skews immune responses to promote infection, lung disease and cancer

Martin R. Stämpfli;Gary P. Anderson.
Nature Reviews Immunology (2009)

611 Citations

Smad3 Null Mice Develop Airspace Enlargement and Are Resistant to TGF-β-Mediated Pulmonary Fibrosis

Philippe Bonniaud;Martin Kolb;Martin Kolb;Tom Galt;Jennifer Robertson.
Journal of Immunology (2004)

417 Citations

GM-CSF transgene expression in the airway allows aerosolized ovalbumin to induce allergic sensitization in mice.

M. R. Stampfli;R. E. Wiley;G. S. Neigh;B. U. Gajewska.
Journal of Clinical Investigation (1998)

298 Citations

Cytokine and eosinophil responses in the lung, peripheral blood, and bone marrow compartments in a murine model of allergen-induced airways inflammation.

Yuichi Ohkawara;Xue-Feng Lei;Martin R. Stampfli;Jean S. Marshall.
American Journal of Respiratory Cell and Molecular Biology (1997)

271 Citations

Cigarette smoke decreases pulmonary dendritic cells and impacts antiviral immune responsiveness.

Clinton S. Robbins;David E. Dawe;Susanna I. Goncharova;Mahmoud A. Pouladi.
American Journal of Respiratory Cell and Molecular Biology (2004)

243 Citations

Impact of Cigarette Smoke on Clearance and Inflammation after Pseudomonas aeruginosa Infection

Anna G. Drannik;Mahmoud A. Pouladi;Clinton S. Robbins;Susanna I. Goncharova.
American Journal of Respiratory and Critical Care Medicine (2004)

205 Citations

Interleukin-10 Gene Transfer to the Airway Regulates Allergic Mucosal Sensitization in Mice

Martin R. Stämpfli;Monika Cwiartka;Beata U. Gajewska;David Alvarez.
American Journal of Respiratory Cell and Molecular Biology (1999)

186 Citations

IL-1α/IL-1R1 Expression in Chronic Obstructive Pulmonary Disease and Mechanistic Relevance to Smoke-Induced Neutrophilia in Mice

Fernando M. Botelho;Carla M. T. Bauer;Donna Finch;Jake K. Nikota.
PLOS ONE (2011)

137 Citations

Impairment of human NK cell cytotoxic activity and cytokine release by cigarette smoke.

M. Firoz Mian;Nicole M. Lauzon;Martin R. Stämpfli;Karen L. Mossman.
Journal of Leukocyte Biology (2008)

126 Citations

Cigarette smoke exposure attenuates cytokine production by mouse alveolar macrophages.

Gordon J. Gaschler;Caleb C. J. Zavitz;Carla M. T. Bauer;Marko Skrtic.
American Journal of Respiratory Cell and Molecular Biology (2008)

124 Citations

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