What is he best known for?
The fields of study he is best known for:
The scientist’s investigation covers issues in Cell biology, Molecular biology, p38 mitogen-activated protein kinases, Protein kinase A and Phosphorylation.
His studies in Cell biology integrate themes in fields like Heat shock protein and Receptor.
His studies deal with areas such as MAP2K7, ASK1, Mitogen-activated protein kinase kinase and MAP kinase kinase kinase as well as Molecular biology.
Matthias Gaestel interconnects Tumor necrosis factor alpha, Tristetraprolin, Cancer research and Messenger RNA in the investigation of issues within p38 mitogen-activated protein kinases.
His Protein kinase A study integrates concerns from other disciplines, such as Hsp27 and Cell migration.
His Phosphorylation research is multidisciplinary, incorporating elements of Gene expression and Cytokine.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Small heat shock proteins are molecular chaperones (1229 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of study are Cell biology, Protein kinase A, p38 mitogen-activated protein kinases, Kinase and Molecular biology.
His study in Phosphorylation, Signal transduction, MAP kinase kinase kinase, Mitogen-activated protein kinase kinase and ASK1 are all subfields of Cell biology.
His research in Protein kinase A intersects with topics in Endocrinology, Hsp27, Internal medicine, Regulation of gene expression and MAPK/ERK pathway.
His p38 mitogen-activated protein kinases research incorporates elements of Tumor necrosis factor alpha, Cancer research, Cytokine, Mitogen-activated protein kinase and Tristetraprolin.
His biological study spans a wide range of topics, including Inflammation and Receptor.
His study in Molecular biology is interdisciplinary in nature, drawing from both Messenger RNA and Gene expression.
He most often published in these fields:
- Cell biology (49.52%)
- Protein kinase A (24.76%)
- p38 mitogen-activated protein kinases (24.13%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (49.52%)
- Kinase (22.22%)
- Protein kinase A (24.76%)
In recent papers he was focusing on the following fields of study:
His primary scientific interests are in Cell biology, Kinase, Protein kinase A, p38 mitogen-activated protein kinases and Signal transduction.
His work deals with themes such as Tumor necrosis factor alpha, Inflammation and Cytokine, which intersect with Cell biology.
The study incorporates disciplines such as Programmed cell death and Necroptosis in addition to Tumor necrosis factor alpha.
The various areas that Matthias Gaestel examines in his Kinase study include Autophagy, T cell, Receptor, Knockout mouse and Acute-phase protein.
His Protein kinase A research is multidisciplinary, relying on both Mitogen-activated protein kinase, Internal medicine, Endocrinology and Exon.
Matthias Gaestel combines subjects such as Cancer research, Serine, Mitogen-activated protein kinase kinase, Interleukin 33 and Fibroblast with his study of p38 mitogen-activated protein kinases.
Between 2013 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- TNF and increased intracellular iron alter macrophage polarization to a detrimental M1 phenotype in the injured spinal cord. (296 citations)
- p38(MAPK)/MK2-dependent phosphorylation controls cytotoxic RIPK1 signalling in inflammation and infection (105 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Cell biology, Protein kinase A, p38 mitogen-activated protein kinases, Kinase and Tumor necrosis factor alpha.
His work on Cell biology deals in particular with Phosphorylation and Signal transduction.
His Protein kinase A research includes themes of Mitogen-activated protein kinase, Internal medicine, Endocrinology and Insulin resistance.
The p38 mitogen-activated protein kinases study combines topics in areas such as Interleukin 33 and Pemphigus vulgaris.
The concepts of his Kinase study are interwoven with issues in Autophagy, Inflammation, Immunology and Programmed cell death.
His research integrates issues of MAP1LC3B, Chaperone-mediated autophagy, Computational biology and Autolysosome in his study of Programmed cell death.
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